Interferon and TLR genes, but not endogenous bornavirus-like elements, limit BoDV1 replication after intracerebral infection.

Interferon and TLR genes, but not endogenous bornavirus-like elements, limit BoDV1 replication after intracerebral infection.

Borna disease virus 1 (BoDV1) is a disease-causing agent in some livestock and, as has recently been shown, in humans. What constitutes a protective immune response to BoDV1 is unclear. Previous studies found that endogenous bornavirus-like nucleoprotein elements (EBLNs) present in mammalian genomes...

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Main Authors: Rie Koide, Takaya Abe, Taichi Harimoto, Anselmo Jiro Kamada, Yuka Saito, Matteo Guerrini, Asami Fujii, Erica Parrish, Masayuki Horie, Hiroshi Kiyonari, Kazuhiko Yamamoto, Keizo Tomonaga, Nicholas F Parrish
Format: Article
Language:English
Published: Public Library of Science (PLoS) 2025-05-01
Series:PLoS Pathogens
Online Access:https://doi.org/10.1371/journal.ppat.1013165
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https://doi.org/10.1371/journal.ppat.1013165

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