Alternative splicing of the Snap23 microexon is regulated by MBNL, QKI, and RBFOX2 in a tissue-specific manner and is altered in striated muscle diseases

Alternative splicing of the Snap23 microexon is regulated by MBNL, QKI, and RBFOX2 in a tissue-specific manner and is altered in striated muscle diseases

The reprogramming of alternative splicing networks during development is a hallmark of tissue maturation and identity. Alternative splicing of microexons (small, genomic regions ≤ 51 nucleotides) functionally regulate protein-protein interactions in the brain and is altered in several neuronal disea...

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Bibliographic Details
Main Authors: Gabrielle M. Gentile, R. Eric Blue, Grant A. Goda, Bryan B. Guzman, Rachel A. Szymanski, Eunice Y. Lee, Nichlas M. Engels, Emma R. Hinkle, Hannah J. Wiedner, Aubriana N. Bishop, Jonathan T. Harrison, Hua Zhang, Xander H.T. Wehrens, Daniel Dominguez, Jimena Giudice
Format: Article
Language:English
Published: Taylor & Francis Group 2025-12-01
Series:RNA Biology
Subjects:
Microexon
alternative splicing
striated muscle
Snap23
RNA-binding proteins
Online Access:https://www.tandfonline.com/doi/10.1080/15476286.2025.2491160
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https://www.tandfonline.com/doi/10.1080/15476286.2025.2491160

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