SARS-CoV-2 nsp1 mediates broad inhibition of translation in mammals

Summary: Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) non-structural protein 1 (nsp1) promotes innate immune evasion by inhibiting host translation in human cells. However, the role of nsp1 in other host species remains elusive, especially in bats—natural reservoirs of sarbecoviruses...

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Main Authors: Risako Gen, Amin Addetia, Daniel Asarnow, Young-Jun Park, Joel Quispe, Matthew C. Chan, Jack T. Brown, Jimin Lee, Melody G. Campbell, Christopher P. Lapointe, David Veesler
Format: Article
Language:English
Published: Elsevier 2025-05-01
Series:Cell Reports
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Online Access:http://www.sciencedirect.com/science/article/pii/S221112472500467X
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author Risako Gen
Amin Addetia
Daniel Asarnow
Young-Jun Park
Joel Quispe
Matthew C. Chan
Jack T. Brown
Jimin Lee
Melody G. Campbell
Christopher P. Lapointe
David Veesler
author_facet Risako Gen
Amin Addetia
Daniel Asarnow
Young-Jun Park
Joel Quispe
Matthew C. Chan
Jack T. Brown
Jimin Lee
Melody G. Campbell
Christopher P. Lapointe
David Veesler
author_sort Risako Gen
collection DOAJ
description Summary: Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) non-structural protein 1 (nsp1) promotes innate immune evasion by inhibiting host translation in human cells. However, the role of nsp1 in other host species remains elusive, especially in bats—natural reservoirs of sarbecoviruses with a markedly different innate immune system than humans. We reveal that nsp1 potently inhibits translation in Rhinolophus lepidus bat cells, which belong to the same genus as known sarbecovirus reservoir hosts. We determined a cryoelectron microscopy structure of nsp1 bound to the R. lepidus 40S ribosomal subunit, showing that it blocks the mRNA entry channel by targeting a highly conserved site among mammals. Accordingly, we found that nsp1 blocked protein translation in mammalian cells from several species, underscoring its broadly inhibitory activity and conserved role in numerous SARS-CoV-2 hosts. Our findings illuminate the arms race between coronaviruses and mammalian host immunity, providing a foundation for understanding the determinants of viral maintenance in bat hosts and spillover.
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issn 2211-1247
language English
publishDate 2025-05-01
publisher Elsevier
record_format Article
series Cell Reports
spelling doaj-art-efbb4d93bd1940908091401b78dd53e62025-08-20T01:49:23ZengElsevierCell Reports2211-12472025-05-0144511569610.1016/j.celrep.2025.115696SARS-CoV-2 nsp1 mediates broad inhibition of translation in mammalsRisako Gen0Amin Addetia1Daniel Asarnow2Young-Jun Park3Joel Quispe4Matthew C. Chan5Jack T. Brown6Jimin Lee7Melody G. Campbell8Christopher P. Lapointe9David Veesler10Department of Biochemistry, University of Washington, Seattle, WA 98195, USADepartment of Biochemistry, University of Washington, Seattle, WA 98195, USADepartment of Biochemistry, University of Washington, Seattle, WA 98195, USADepartment of Biochemistry, University of Washington, Seattle, WA 98195, USADepartment of Biochemistry, University of Washington, Seattle, WA 98195, USADivision of Basic Sciences, Fred Hutchinson Cancer Center, Seattle, WA, USADepartment of Biochemistry, University of Washington, Seattle, WA 98195, USADepartment of Biochemistry, University of Washington, Seattle, WA 98195, USADivision of Basic Sciences, Fred Hutchinson Cancer Center, Seattle, WA, USADivision of Basic Sciences, Fred Hutchinson Cancer Center, Seattle, WA, USADepartment of Biochemistry, University of Washington, Seattle, WA 98195, USA; Howard Hughes Medical Institute, University of Washington, Seattle, WA 98195, USA; Corresponding authorSummary: Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) non-structural protein 1 (nsp1) promotes innate immune evasion by inhibiting host translation in human cells. However, the role of nsp1 in other host species remains elusive, especially in bats—natural reservoirs of sarbecoviruses with a markedly different innate immune system than humans. We reveal that nsp1 potently inhibits translation in Rhinolophus lepidus bat cells, which belong to the same genus as known sarbecovirus reservoir hosts. We determined a cryoelectron microscopy structure of nsp1 bound to the R. lepidus 40S ribosomal subunit, showing that it blocks the mRNA entry channel by targeting a highly conserved site among mammals. Accordingly, we found that nsp1 blocked protein translation in mammalian cells from several species, underscoring its broadly inhibitory activity and conserved role in numerous SARS-CoV-2 hosts. Our findings illuminate the arms race between coronaviruses and mammalian host immunity, providing a foundation for understanding the determinants of viral maintenance in bat hosts and spillover.http://www.sciencedirect.com/science/article/pii/S221112472500467XCP: Microbiology
spellingShingle Risako Gen
Amin Addetia
Daniel Asarnow
Young-Jun Park
Joel Quispe
Matthew C. Chan
Jack T. Brown
Jimin Lee
Melody G. Campbell
Christopher P. Lapointe
David Veesler
SARS-CoV-2 nsp1 mediates broad inhibition of translation in mammals
Cell Reports
CP: Microbiology
title SARS-CoV-2 nsp1 mediates broad inhibition of translation in mammals
title_full SARS-CoV-2 nsp1 mediates broad inhibition of translation in mammals
title_fullStr SARS-CoV-2 nsp1 mediates broad inhibition of translation in mammals
title_full_unstemmed SARS-CoV-2 nsp1 mediates broad inhibition of translation in mammals
title_short SARS-CoV-2 nsp1 mediates broad inhibition of translation in mammals
title_sort sars cov 2 nsp1 mediates broad inhibition of translation in mammals
topic CP: Microbiology
url http://www.sciencedirect.com/science/article/pii/S221112472500467X
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