Effect of continuous Ia fibre activity suppression on hyperreflexia-related spasticity and maladaptive synaptic connections in the spinal cord after injury

Abstract Spasticity is defined as the velocity-dependent hyperexcitability of the stretch reflex that develops after a central nervous system injury. Spasticity is caused by plastic neuronal changes following injury. Current treatments that block spastic muscle contractions do not promote recovery f...

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Main Authors: Takuto Hanasaki, Keita Hanaki, Yukito Sako, Yasushi Uchiyama, Sachiko Lee-Hotta
Format: Article
Language:English
Published: Nature Portfolio 2025-07-01
Series:Scientific Reports
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Online Access:https://doi.org/10.1038/s41598-025-09397-x
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author Takuto Hanasaki
Keita Hanaki
Yukito Sako
Yasushi Uchiyama
Sachiko Lee-Hotta
author_facet Takuto Hanasaki
Keita Hanaki
Yukito Sako
Yasushi Uchiyama
Sachiko Lee-Hotta
author_sort Takuto Hanasaki
collection DOAJ
description Abstract Spasticity is defined as the velocity-dependent hyperexcitability of the stretch reflex that develops after a central nervous system injury. Spasticity is caused by plastic neuronal changes following injury. Current treatments that block spastic muscle contractions do not promote recovery from motor dysfunction. We aimed to confirm that Ia fibre activity suppression, comprising the stretch reflex, reduces spasticity-related hyperreflexia and improves pathological neuronal plastic changes and motor dysfunction. In this study, we created a hemi-transected spinal cord injury mouse model and continued Ia fibre suppression for 2 weeks. The effects of Ia fibre suppression were evaluated electrophysiologically and histologically. In electrophysiology, spasticity-related rate-dependent depression of Hoffman’s reflex improved from 0.6 to 0.2 in terms of the rate of amplitude change with reference to 0.1 Hz electrical stimulation. Histologically, the number of synapse buttons of Ia fibres per an α motor neuron reduced from 4.2 to 2.6. However, the α motor neuron activity was still higher than that in the sham mice, possibly due to other residual pathological mechanisms of spasticity. Additionally, motor dysfunction was observed in grid walk and single-reach tasks in vehicle- and drug-administered groups. This study confirmed that continuous Ia fibre suppression partly improved the maladaptive synaptic connections in the spinal cord and relieved spasticity-related hyperreflexia.
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spelling doaj-art-d94cf76ab5254ac78528140ccfc757122025-08-20T03:38:15ZengNature PortfolioScientific Reports2045-23222025-07-0115111410.1038/s41598-025-09397-xEffect of continuous Ia fibre activity suppression on hyperreflexia-related spasticity and maladaptive synaptic connections in the spinal cord after injuryTakuto Hanasaki0Keita Hanaki1Yukito Sako2Yasushi Uchiyama3Sachiko Lee-Hotta4Department of Integrated Health Sciences, Graduate School of Medicine, Nagoya UniversityDepartment of Integrated Health Sciences, Graduate School of Medicine, Nagoya UniversityDepartment of Integrated Health Sciences, Graduate School of Medicine, Nagoya UniversityDivision of Creative Physical Therapy, Department of Integrated Health Sciences, Graduate School of Medicine, Nagoya UniversityDivision of Creative Physical Therapy, Department of Integrated Health Sciences, Graduate School of Medicine, Nagoya UniversityAbstract Spasticity is defined as the velocity-dependent hyperexcitability of the stretch reflex that develops after a central nervous system injury. Spasticity is caused by plastic neuronal changes following injury. Current treatments that block spastic muscle contractions do not promote recovery from motor dysfunction. We aimed to confirm that Ia fibre activity suppression, comprising the stretch reflex, reduces spasticity-related hyperreflexia and improves pathological neuronal plastic changes and motor dysfunction. In this study, we created a hemi-transected spinal cord injury mouse model and continued Ia fibre suppression for 2 weeks. The effects of Ia fibre suppression were evaluated electrophysiologically and histologically. In electrophysiology, spasticity-related rate-dependent depression of Hoffman’s reflex improved from 0.6 to 0.2 in terms of the rate of amplitude change with reference to 0.1 Hz electrical stimulation. Histologically, the number of synapse buttons of Ia fibres per an α motor neuron reduced from 4.2 to 2.6. However, the α motor neuron activity was still higher than that in the sham mice, possibly due to other residual pathological mechanisms of spasticity. Additionally, motor dysfunction was observed in grid walk and single-reach tasks in vehicle- and drug-administered groups. This study confirmed that continuous Ia fibre suppression partly improved the maladaptive synaptic connections in the spinal cord and relieved spasticity-related hyperreflexia.https://doi.org/10.1038/s41598-025-09397-xSpasticityIa fibrePLD-mGLuRSpinal cord injury
spellingShingle Takuto Hanasaki
Keita Hanaki
Yukito Sako
Yasushi Uchiyama
Sachiko Lee-Hotta
Effect of continuous Ia fibre activity suppression on hyperreflexia-related spasticity and maladaptive synaptic connections in the spinal cord after injury
Scientific Reports
Spasticity
Ia fibre
PLD-mGLuR
Spinal cord injury
title Effect of continuous Ia fibre activity suppression on hyperreflexia-related spasticity and maladaptive synaptic connections in the spinal cord after injury
title_full Effect of continuous Ia fibre activity suppression on hyperreflexia-related spasticity and maladaptive synaptic connections in the spinal cord after injury
title_fullStr Effect of continuous Ia fibre activity suppression on hyperreflexia-related spasticity and maladaptive synaptic connections in the spinal cord after injury
title_full_unstemmed Effect of continuous Ia fibre activity suppression on hyperreflexia-related spasticity and maladaptive synaptic connections in the spinal cord after injury
title_short Effect of continuous Ia fibre activity suppression on hyperreflexia-related spasticity and maladaptive synaptic connections in the spinal cord after injury
title_sort effect of continuous ia fibre activity suppression on hyperreflexia related spasticity and maladaptive synaptic connections in the spinal cord after injury
topic Spasticity
Ia fibre
PLD-mGLuR
Spinal cord injury
url https://doi.org/10.1038/s41598-025-09397-x
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