Structure-Activity Relationship Study of Majusculamide D: Overcoming Metabolic Instability and Severe Toxicity with a Fluoro Analogue

Majusculamide D, isolated from the marine cyanobacterium <i>Moorea producens</i>, is an anticancer lipopentapeptide consisting of fatty acid, tripeptide, and pyrrolyl proline moieties. In this work, by utilizing a convergent synthetic approach, late-stage modification, and bioisostere st...

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Main Authors: Xiuhe Zhao, Xiaonan Xi, Mingxiao Zhang, Mengxue Lv, Xiang Zhang, Yaxin Lu, Liang Wang, Yue Chen
Format: Article
Language:English
Published: MDPI AG 2024-11-01
Series:Marine Drugs
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Online Access:https://www.mdpi.com/1660-3397/22/12/537
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Summary:Majusculamide D, isolated from the marine cyanobacterium <i>Moorea producens</i>, is an anticancer lipopentapeptide consisting of fatty acid, tripeptide, and pyrrolyl proline moieties. In this work, by utilizing a convergent synthetic approach, late-stage modification, and bioisostere strategy, 26 majusculamide D analogues were synthesized, and two (<b>1i</b> and <b>1j</b>) demonstrated IC<sub>50</sub> values < 1 nM against PANC-1 cancer cells. The results summarized a preliminary structure-activity relationship mainly at the C23, C4, C34, and C10 sites. A series of in vitro assays, including wound healing, transwell, clone formation, EdU, and western blot, confirmed that majusculamide D inhibited the migration, invasion, and proliferation of pancreatic cancer cells. The optimized fluorinated analogue <b>1n</b> demonstrated a notable enhancement in stability during the mouse plasma assay (>50% left after 24 h), exhibited tumor-suppressive effects (51.5% at a dosage of 5 mg/kg), and successfully mitigated the severe toxicity (no mouse dead) observed in the group treated with majusculamide D (3 mice dead) in a xenografted mouse model.
ISSN:1660-3397