Regulation of Senescence in Cancer and Aging

Senescence is regarded as a physiological response of cells to stress, including telomere dysfunction, aberrant oncogenic activation, DNA damage, and oxidative stress. This stress response has an antagonistically pleiotropic effect to organisms: beneficial as a tumor suppressor, but detrimental by c...

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Main Authors: Yahui Kong, Hang Cui, Charusheila Ramkumar, Hong Zhang
Format: Article
Language:English
Published: Wiley 2011-01-01
Series:Journal of Aging Research
Online Access:http://dx.doi.org/10.4061/2011/963172
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author Yahui Kong
Hang Cui
Charusheila Ramkumar
Hong Zhang
author_facet Yahui Kong
Hang Cui
Charusheila Ramkumar
Hong Zhang
author_sort Yahui Kong
collection DOAJ
description Senescence is regarded as a physiological response of cells to stress, including telomere dysfunction, aberrant oncogenic activation, DNA damage, and oxidative stress. This stress response has an antagonistically pleiotropic effect to organisms: beneficial as a tumor suppressor, but detrimental by contributing to aging. The emergence of senescence as an effective tumor suppression mechanism is highlighted by recent demonstration that senescence prevents proliferation of cells at risk of neoplastic transformation. Consequently, induction of senescence is recognized as a potential treatment of cancer. Substantial evidence also suggests that senescence plays an important role in aging, particularly in aging of stem cells. In this paper, we will discuss the molecular regulation of senescence its role in cancer and aging. The potential utility of senescence in cancer therapeutics will also be discussed.
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spelling doaj-art-c69fdbbbcf35435094dbda82bb76fb502025-08-20T02:19:16ZengWileyJournal of Aging Research2090-22122011-01-01201110.4061/2011/963172963172Regulation of Senescence in Cancer and AgingYahui Kong0Hang Cui1Charusheila Ramkumar2Hong Zhang3Department of Cell Biology, University of Massachusetts Medical School, 55 Lake Avenue North, S7-125, Worcester, MA 01655, USADepartment of Cell Biology, University of Massachusetts Medical School, 55 Lake Avenue North, S7-125, Worcester, MA 01655, USADepartment of Cell Biology, University of Massachusetts Medical School, 55 Lake Avenue North, S7-125, Worcester, MA 01655, USADepartment of Cell Biology, University of Massachusetts Medical School, 55 Lake Avenue North, S7-125, Worcester, MA 01655, USASenescence is regarded as a physiological response of cells to stress, including telomere dysfunction, aberrant oncogenic activation, DNA damage, and oxidative stress. This stress response has an antagonistically pleiotropic effect to organisms: beneficial as a tumor suppressor, but detrimental by contributing to aging. The emergence of senescence as an effective tumor suppression mechanism is highlighted by recent demonstration that senescence prevents proliferation of cells at risk of neoplastic transformation. Consequently, induction of senescence is recognized as a potential treatment of cancer. Substantial evidence also suggests that senescence plays an important role in aging, particularly in aging of stem cells. In this paper, we will discuss the molecular regulation of senescence its role in cancer and aging. The potential utility of senescence in cancer therapeutics will also be discussed.http://dx.doi.org/10.4061/2011/963172
spellingShingle Yahui Kong
Hang Cui
Charusheila Ramkumar
Hong Zhang
Regulation of Senescence in Cancer and Aging
Journal of Aging Research
title Regulation of Senescence in Cancer and Aging
title_full Regulation of Senescence in Cancer and Aging
title_fullStr Regulation of Senescence in Cancer and Aging
title_full_unstemmed Regulation of Senescence in Cancer and Aging
title_short Regulation of Senescence in Cancer and Aging
title_sort regulation of senescence in cancer and aging
url http://dx.doi.org/10.4061/2011/963172
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AT hangcui regulationofsenescenceincancerandaging
AT charusheilaramkumar regulationofsenescenceincancerandaging
AT hongzhang regulationofsenescenceincancerandaging