Fisetin limits Chikungunya virus-induced apoptosis hallmarks in hepatocellular carcinoma cells

Abstract The study investigated the protective effects of fisetin on chikungunya virus (CHIKV)-induced apoptosis hallmarks in Huh7 cells. Fisetin significantly reduced CHIKV RNA levels and viral infectivity, outperforming Z-VAD-FMK and cisplatin. At 30 µM, fisetin markedly decreased (by > 90%) th...

Full description

Saved in:
Bibliographic Details
Main Authors: Rafidah Lani, Pouya Hassandarvish, Sazaly AbuBakar
Format: Article
Language:English
Published: Nature Portfolio 2025-07-01
Series:Scientific Reports
Subjects:
Online Access:https://doi.org/10.1038/s41598-025-09213-6
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1849234798600519680
author Rafidah Lani
Pouya Hassandarvish
Sazaly AbuBakar
author_facet Rafidah Lani
Pouya Hassandarvish
Sazaly AbuBakar
author_sort Rafidah Lani
collection DOAJ
description Abstract The study investigated the protective effects of fisetin on chikungunya virus (CHIKV)-induced apoptosis hallmarks in Huh7 cells. Fisetin significantly reduced CHIKV RNA levels and viral infectivity, outperforming Z-VAD-FMK and cisplatin. At 30 µM, fisetin markedly decreased (by > 90%) the number of infectious viral particles at 24 and 48 h post-infection (hpi). Fisetin also hindered CHIKV-induced DNA fragmentation, with the lowest levels observed in CHIKV-infected cells treated with fisetin compared to other treatments. Immunoblot analysis revealed that fisetin inhibited caspase-mediated PARP cleavage and significantly reduced cleaved PARP levels, indicating decreased apoptosis. Additionally, fisetin diminished the expression of cleaved caspase-3 and HSP-27 proteins, while restoring HIF-1α protein levels, suggesting a protective role against CHIKV-induced apoptosis. The study highlights the potential of fisetin as an effective antiviral agent against CHIKV through the modulation of apoptosis and oxidative stress pathways. These findings underscore the therapeutic promise of fisetin for treating CHIKV-induced apoptosis and warrant further investigation to explore its clinical applications and optimize its use in antiviral therapy.
format Article
id doaj-art-bceb9fccf80f43419305f0951e01f870
institution Kabale University
issn 2045-2322
language English
publishDate 2025-07-01
publisher Nature Portfolio
record_format Article
series Scientific Reports
spelling doaj-art-bceb9fccf80f43419305f0951e01f8702025-08-20T04:03:01ZengNature PortfolioScientific Reports2045-23222025-07-0115111010.1038/s41598-025-09213-6Fisetin limits Chikungunya virus-induced apoptosis hallmarks in hepatocellular carcinoma cellsRafidah Lani0Pouya Hassandarvish1Sazaly AbuBakar2Department of Medical Microbiology, Faculty of Medicine, Universiti MalayaTropical Infectious Diseases Research and Education Centre (TIDREC), Universiti MalayaTropical Infectious Diseases Research and Education Centre (TIDREC), Universiti MalayaAbstract The study investigated the protective effects of fisetin on chikungunya virus (CHIKV)-induced apoptosis hallmarks in Huh7 cells. Fisetin significantly reduced CHIKV RNA levels and viral infectivity, outperforming Z-VAD-FMK and cisplatin. At 30 µM, fisetin markedly decreased (by > 90%) the number of infectious viral particles at 24 and 48 h post-infection (hpi). Fisetin also hindered CHIKV-induced DNA fragmentation, with the lowest levels observed in CHIKV-infected cells treated with fisetin compared to other treatments. Immunoblot analysis revealed that fisetin inhibited caspase-mediated PARP cleavage and significantly reduced cleaved PARP levels, indicating decreased apoptosis. Additionally, fisetin diminished the expression of cleaved caspase-3 and HSP-27 proteins, while restoring HIF-1α protein levels, suggesting a protective role against CHIKV-induced apoptosis. The study highlights the potential of fisetin as an effective antiviral agent against CHIKV through the modulation of apoptosis and oxidative stress pathways. These findings underscore the therapeutic promise of fisetin for treating CHIKV-induced apoptosis and warrant further investigation to explore its clinical applications and optimize its use in antiviral therapy.https://doi.org/10.1038/s41598-025-09213-6ApoptosisChikungunyaFlavonoidsFisetinAntiviralCytoprotective
spellingShingle Rafidah Lani
Pouya Hassandarvish
Sazaly AbuBakar
Fisetin limits Chikungunya virus-induced apoptosis hallmarks in hepatocellular carcinoma cells
Scientific Reports
Apoptosis
Chikungunya
Flavonoids
Fisetin
Antiviral
Cytoprotective
title Fisetin limits Chikungunya virus-induced apoptosis hallmarks in hepatocellular carcinoma cells
title_full Fisetin limits Chikungunya virus-induced apoptosis hallmarks in hepatocellular carcinoma cells
title_fullStr Fisetin limits Chikungunya virus-induced apoptosis hallmarks in hepatocellular carcinoma cells
title_full_unstemmed Fisetin limits Chikungunya virus-induced apoptosis hallmarks in hepatocellular carcinoma cells
title_short Fisetin limits Chikungunya virus-induced apoptosis hallmarks in hepatocellular carcinoma cells
title_sort fisetin limits chikungunya virus induced apoptosis hallmarks in hepatocellular carcinoma cells
topic Apoptosis
Chikungunya
Flavonoids
Fisetin
Antiviral
Cytoprotective
url https://doi.org/10.1038/s41598-025-09213-6
work_keys_str_mv AT rafidahlani fisetinlimitschikungunyavirusinducedapoptosishallmarksinhepatocellularcarcinomacells
AT pouyahassandarvish fisetinlimitschikungunyavirusinducedapoptosishallmarksinhepatocellularcarcinomacells
AT sazalyabubakar fisetinlimitschikungunyavirusinducedapoptosishallmarksinhepatocellularcarcinomacells