Identification of progression related LncRNAs in colorectal cancer aggressiveness

Abstract Colorectal cancer (CRC) progression involves complex molecular alterations, including the dysregulation of long non-coding RNAs (lncRNAs). In this study, we identified key progression-related lncRNAs in CRC by integrating transcriptomic data from TCGA and single-cell RNA sequencing (scRNA-s...

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Main Authors: Wei Xi, Xinxin Sun, Mingwei Wang, Xizi Wang, Kun Li, Runze Jiang, Xiaodong Jia, Wenxiao Wang
Format: Article
Language:English
Published: Nature Portfolio 2025-05-01
Series:Scientific Reports
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Online Access:https://doi.org/10.1038/s41598-025-02096-7
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author Wei Xi
Xinxin Sun
Mingwei Wang
Xizi Wang
Kun Li
Runze Jiang
Xiaodong Jia
Wenxiao Wang
author_facet Wei Xi
Xinxin Sun
Mingwei Wang
Xizi Wang
Kun Li
Runze Jiang
Xiaodong Jia
Wenxiao Wang
author_sort Wei Xi
collection DOAJ
description Abstract Colorectal cancer (CRC) progression involves complex molecular alterations, including the dysregulation of long non-coding RNAs (lncRNAs). In this study, we identified key progression-related lncRNAs in CRC by integrating transcriptomic data from TCGA and single-cell RNA sequencing (scRNA-seq). Differential expression analysis revealed numerous lncRNAs associated with CRC progression. To systematically prioritize these lncRNAs, we developed a scoring system incorporating multiple progression-related signatures, differential expression, and survival analysis. This approach identified 198 key lncRNAs, including both known (e.g., LINC01615) and novel candidates (e.g., AC007998.3). Experimental validation confirmed that LINC01615 was significantly upregulated in CRC tissues, whereas AC007998.3 was downregulated. Further analyses indicated that these lncRNAs influence CRC progression through cis-, trans-, and post-transcriptional regulation. Patients were classified into distinct molecular subgroups based on lncRNA expression, exhibiting significant differences in prognosis and immune microenvironment composition. The enrichment of progression-related lncRNAs among differentially expressed lncRNAs was statistically significant, reinforcing their functional relevance. Validation across independent datasets demonstrated the robustness of our findings. Our research provides novel insights into the molecular mechanisms underlying CRC progression and highlights the potential of progression-related lncRNAs as prognostic biomarkers and therapeutic targets.
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issn 2045-2322
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spelling doaj-art-9f9e57fb79444154b5bbe30cd2a316202025-08-20T01:53:22ZengNature PortfolioScientific Reports2045-23222025-05-0115111310.1038/s41598-025-02096-7Identification of progression related LncRNAs in colorectal cancer aggressivenessWei Xi0Xinxin Sun1Mingwei Wang2Xizi Wang3Kun Li4Runze Jiang5Xiaodong Jia6Wenxiao Wang7Department of Oncology, Liaocheng People’s HospitalJoint Laboratory for Translational Medicine Research, Liaocheng People’s HospitalTraditional Chinese Medicine Innovation Research Institute, Shandong University of Traditional Chinese MedicineJoint Laboratory for Translational Medicine Research, Liaocheng People’s HospitalJoint Laboratory for Translational Medicine Research, Liaocheng People’s HospitalTraditional Chinese Medicine Innovation Research Institute, Shandong University of Traditional Chinese MedicineJoint Laboratory for Translational Medicine Research, Liaocheng People’s HospitalDepartment of Gastrointestinal Surgery, Liaocheng People’s HospitalAbstract Colorectal cancer (CRC) progression involves complex molecular alterations, including the dysregulation of long non-coding RNAs (lncRNAs). In this study, we identified key progression-related lncRNAs in CRC by integrating transcriptomic data from TCGA and single-cell RNA sequencing (scRNA-seq). Differential expression analysis revealed numerous lncRNAs associated with CRC progression. To systematically prioritize these lncRNAs, we developed a scoring system incorporating multiple progression-related signatures, differential expression, and survival analysis. This approach identified 198 key lncRNAs, including both known (e.g., LINC01615) and novel candidates (e.g., AC007998.3). Experimental validation confirmed that LINC01615 was significantly upregulated in CRC tissues, whereas AC007998.3 was downregulated. Further analyses indicated that these lncRNAs influence CRC progression through cis-, trans-, and post-transcriptional regulation. Patients were classified into distinct molecular subgroups based on lncRNA expression, exhibiting significant differences in prognosis and immune microenvironment composition. The enrichment of progression-related lncRNAs among differentially expressed lncRNAs was statistically significant, reinforcing their functional relevance. Validation across independent datasets demonstrated the robustness of our findings. Our research provides novel insights into the molecular mechanisms underlying CRC progression and highlights the potential of progression-related lncRNAs as prognostic biomarkers and therapeutic targets.https://doi.org/10.1038/s41598-025-02096-7Colorectal cancerLncRNAsCancer progressionTumor microenvironmentImmune regulationImmune infiltration
spellingShingle Wei Xi
Xinxin Sun
Mingwei Wang
Xizi Wang
Kun Li
Runze Jiang
Xiaodong Jia
Wenxiao Wang
Identification of progression related LncRNAs in colorectal cancer aggressiveness
Scientific Reports
Colorectal cancer
LncRNAs
Cancer progression
Tumor microenvironment
Immune regulation
Immune infiltration
title Identification of progression related LncRNAs in colorectal cancer aggressiveness
title_full Identification of progression related LncRNAs in colorectal cancer aggressiveness
title_fullStr Identification of progression related LncRNAs in colorectal cancer aggressiveness
title_full_unstemmed Identification of progression related LncRNAs in colorectal cancer aggressiveness
title_short Identification of progression related LncRNAs in colorectal cancer aggressiveness
title_sort identification of progression related lncrnas in colorectal cancer aggressiveness
topic Colorectal cancer
LncRNAs
Cancer progression
Tumor microenvironment
Immune regulation
Immune infiltration
url https://doi.org/10.1038/s41598-025-02096-7
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