Exogenous adenosine counteracts tigecycline resistance in tet(X3)-harboring Escherichia coli

ABSTRACT The rapid spread of antibiotic resistance poses a global health crisis. Tigecycline is a last-resort antibiotic, but the recent emergence of the plasmid-borne tet(X3) gene conferring high-level tigecycline resistance is deeply concerning. Here, we report a metabolomics-guided approach to ov...

Full description

Saved in:
Bibliographic Details
Main Authors: Jing Sun, Yiming Liu, Jiashen Chang, Ying Liu, Luqi Li, Ran Jiang, Yihan Luo, Shuo Yang, Mei Yang, Xinglong Wang, Juan Wang, Xi Xia, Kangkang Guo, Zengqi Yang, Dongyang Ye
Format: Article
Language:English
Published: American Society for Microbiology 2025-08-01
Series:Microbiology Spectrum
Subjects:
Online Access:https://journals.asm.org/doi/10.1128/spectrum.02382-24
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1850039264763445248
author Jing Sun
Yiming Liu
Jiashen Chang
Ying Liu
Luqi Li
Ran Jiang
Yihan Luo
Shuo Yang
Mei Yang
Xinglong Wang
Juan Wang
Xi Xia
Kangkang Guo
Zengqi Yang
Dongyang Ye
author_facet Jing Sun
Yiming Liu
Jiashen Chang
Ying Liu
Luqi Li
Ran Jiang
Yihan Luo
Shuo Yang
Mei Yang
Xinglong Wang
Juan Wang
Xi Xia
Kangkang Guo
Zengqi Yang
Dongyang Ye
author_sort Jing Sun
collection DOAJ
description ABSTRACT The rapid spread of antibiotic resistance poses a global health crisis. Tigecycline is a last-resort antibiotic, but the recent emergence of the plasmid-borne tet(X3) gene conferring high-level tigecycline resistance is deeply concerning. Here, we report a metabolomics-guided approach to overcome tet(X3)-mediated resistance. Using untargeted metabolomics, we identified adenosine as a key metabolic biomarker associated with tet(X3) expression. Remarkably, supplementation with exogenous adenosine was able to restore tigecycline susceptibility in tet(X3)-positive Escherichia coli both in vitro and in vivo. Our mechanistic investigations reveal that adenosine enhances the bactericidal effects of tigecycline by inducing oxidative stress, DNA/RNA damage, and cell membrane disruption in resistant bacteria. This study establishes a powerful metabolomics-driven strategy to potentiate antibiotic efficacy against drug-resistant pathogens. The adenosine-based adjuvant therapy represents a promising approach to combat the global crisis of antibiotic resistance.IMPORTANCEThe emergence and widespread dissemination of the high-level tigecycline resistance gene tet(X3) have posed a significant challenge to the efficacy of tigecycline, which serves as the “last line of defense” against antimicrobial-resistant bacteria. Although tigecycline has not been approved for veterinary clinical use, constant detection of tet(X3) genes and new subtypes in livestock farming environments poses a substantial threat to public health safety. While developing novel antibiotics is an effective approach to eradicate resistance genes/bacteria, it entails considerable costs and a lengthy timeframe. This study discovered that exogenous adenosine can effectively restore the sensitivity of tet(X3)-positive Escherichia coli to tigecycline through metabolic reprogramming based on a non-targeted metabolomics strategy. The findings are highly significant for exploring comprehensive mechanisms underlying bacterial multidrug resistance, utilizing metabolic reprogramming strategies to curb the spread of novel resistant genes, and treating clinical infections caused by tet(X3)-positive bacteria.
format Article
id doaj-art-9c0f46dc3dfe4f379628a68c31a0d65e
institution DOAJ
issn 2165-0497
language English
publishDate 2025-08-01
publisher American Society for Microbiology
record_format Article
series Microbiology Spectrum
spelling doaj-art-9c0f46dc3dfe4f379628a68c31a0d65e2025-08-20T02:56:24ZengAmerican Society for MicrobiologyMicrobiology Spectrum2165-04972025-08-0113810.1128/spectrum.02382-24Exogenous adenosine counteracts tigecycline resistance in tet(X3)-harboring Escherichia coliJing Sun0Yiming Liu1Jiashen Chang2Ying Liu3Luqi Li4Ran Jiang5Yihan Luo6Shuo Yang7Mei Yang8Xinglong Wang9Juan Wang10Xi Xia11Kangkang Guo12Zengqi Yang13Dongyang Ye14College of Veterinary Medicine, Northwest A&F University, Yangling, Shaanxi, ChinaCollege of Veterinary Medicine, Northwest A&F University, Yangling, Shaanxi, ChinaCollege of Veterinary Medicine, Northwest A&F University, Yangling, Shaanxi, ChinaCollege of Veterinary Medicine, Northwest A&F University, Yangling, Shaanxi, ChinaLife Science Research Core Services, Northwest A&F University, Yangling, Shaanxi, ChinaCollege of Veterinary Medicine, Northwest A&F University, Yangling, Shaanxi, ChinaCollege of Veterinary Medicine, Northwest A&F University, Yangling, Shaanxi, ChinaCollege of Veterinary Medicine, Northwest A&F University, Yangling, Shaanxi, ChinaCollege of Veterinary Medicine, Northwest A&F University, Yangling, Shaanxi, ChinaCollege of Veterinary Medicine, Northwest A&F University, Yangling, Shaanxi, ChinaCollege of Veterinary Medicine, Northwest A&F University, Yangling, Shaanxi, ChinaNational Key Laboratory of Veterinary Public Health and Safety, College of Veterinary Medicine, China Agricultural University, Beijing, ChinaCollege of Veterinary Medicine, Northwest A&F University, Yangling, Shaanxi, ChinaCollege of Veterinary Medicine, Northwest A&F University, Yangling, Shaanxi, ChinaCollege of Veterinary Medicine, Northwest A&F University, Yangling, Shaanxi, ChinaABSTRACT The rapid spread of antibiotic resistance poses a global health crisis. Tigecycline is a last-resort antibiotic, but the recent emergence of the plasmid-borne tet(X3) gene conferring high-level tigecycline resistance is deeply concerning. Here, we report a metabolomics-guided approach to overcome tet(X3)-mediated resistance. Using untargeted metabolomics, we identified adenosine as a key metabolic biomarker associated with tet(X3) expression. Remarkably, supplementation with exogenous adenosine was able to restore tigecycline susceptibility in tet(X3)-positive Escherichia coli both in vitro and in vivo. Our mechanistic investigations reveal that adenosine enhances the bactericidal effects of tigecycline by inducing oxidative stress, DNA/RNA damage, and cell membrane disruption in resistant bacteria. This study establishes a powerful metabolomics-driven strategy to potentiate antibiotic efficacy against drug-resistant pathogens. The adenosine-based adjuvant therapy represents a promising approach to combat the global crisis of antibiotic resistance.IMPORTANCEThe emergence and widespread dissemination of the high-level tigecycline resistance gene tet(X3) have posed a significant challenge to the efficacy of tigecycline, which serves as the “last line of defense” against antimicrobial-resistant bacteria. Although tigecycline has not been approved for veterinary clinical use, constant detection of tet(X3) genes and new subtypes in livestock farming environments poses a substantial threat to public health safety. While developing novel antibiotics is an effective approach to eradicate resistance genes/bacteria, it entails considerable costs and a lengthy timeframe. This study discovered that exogenous adenosine can effectively restore the sensitivity of tet(X3)-positive Escherichia coli to tigecycline through metabolic reprogramming based on a non-targeted metabolomics strategy. The findings are highly significant for exploring comprehensive mechanisms underlying bacterial multidrug resistance, utilizing metabolic reprogramming strategies to curb the spread of novel resistant genes, and treating clinical infections caused by tet(X3)-positive bacteria.https://journals.asm.org/doi/10.1128/spectrum.02382-24tet(X3)tigecyclineadenosineantimicrobial resistancemetabolomics
spellingShingle Jing Sun
Yiming Liu
Jiashen Chang
Ying Liu
Luqi Li
Ran Jiang
Yihan Luo
Shuo Yang
Mei Yang
Xinglong Wang
Juan Wang
Xi Xia
Kangkang Guo
Zengqi Yang
Dongyang Ye
Exogenous adenosine counteracts tigecycline resistance in tet(X3)-harboring Escherichia coli
Microbiology Spectrum
tet(X3)
tigecycline
adenosine
antimicrobial resistance
metabolomics
title Exogenous adenosine counteracts tigecycline resistance in tet(X3)-harboring Escherichia coli
title_full Exogenous adenosine counteracts tigecycline resistance in tet(X3)-harboring Escherichia coli
title_fullStr Exogenous adenosine counteracts tigecycline resistance in tet(X3)-harboring Escherichia coli
title_full_unstemmed Exogenous adenosine counteracts tigecycline resistance in tet(X3)-harboring Escherichia coli
title_short Exogenous adenosine counteracts tigecycline resistance in tet(X3)-harboring Escherichia coli
title_sort exogenous adenosine counteracts tigecycline resistance in tet x3 harboring escherichia coli
topic tet(X3)
tigecycline
adenosine
antimicrobial resistance
metabolomics
url https://journals.asm.org/doi/10.1128/spectrum.02382-24
work_keys_str_mv AT jingsun exogenousadenosinecounteractstigecyclineresistanceintetx3harboringescherichiacoli
AT yimingliu exogenousadenosinecounteractstigecyclineresistanceintetx3harboringescherichiacoli
AT jiashenchang exogenousadenosinecounteractstigecyclineresistanceintetx3harboringescherichiacoli
AT yingliu exogenousadenosinecounteractstigecyclineresistanceintetx3harboringescherichiacoli
AT luqili exogenousadenosinecounteractstigecyclineresistanceintetx3harboringescherichiacoli
AT ranjiang exogenousadenosinecounteractstigecyclineresistanceintetx3harboringescherichiacoli
AT yihanluo exogenousadenosinecounteractstigecyclineresistanceintetx3harboringescherichiacoli
AT shuoyang exogenousadenosinecounteractstigecyclineresistanceintetx3harboringescherichiacoli
AT meiyang exogenousadenosinecounteractstigecyclineresistanceintetx3harboringescherichiacoli
AT xinglongwang exogenousadenosinecounteractstigecyclineresistanceintetx3harboringescherichiacoli
AT juanwang exogenousadenosinecounteractstigecyclineresistanceintetx3harboringescherichiacoli
AT xixia exogenousadenosinecounteractstigecyclineresistanceintetx3harboringescherichiacoli
AT kangkangguo exogenousadenosinecounteractstigecyclineresistanceintetx3harboringescherichiacoli
AT zengqiyang exogenousadenosinecounteractstigecyclineresistanceintetx3harboringescherichiacoli
AT dongyangye exogenousadenosinecounteractstigecyclineresistanceintetx3harboringescherichiacoli