Oxygen metabolism abnormalities and high-altitude cerebral edema

Oxygen metabolism abnormalities and high-altitude cerebral edema

Hypobaric hypoxia is widely recognized as a prominent risk factor for high-altitude cerebral edema (HACE), which contributes to the exacerbation of multiple pathological mechanisms, including oxidative stress, mitochondrial dysfunction, disruption of blood−;brain barrier integrity, neuroinflammation...

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Bibliographic Details
Main Authors: Zhi Li, Jianping Zhang, Xiaoxia Zhang, Qiaoying Jin, Xingxing Zheng, Li Mo, Zejiao Da
Format: Article
Language:English
Published: Frontiers Media S.A. 2025-03-01
Series:Frontiers in Immunology
Subjects:
oxygen metabolism
hypobaric hypoxia
oxidative stress
mitochondrial dysfunction
high-altitude cerebral edema
Online Access:https://www.frontiersin.org/articles/10.3389/fimmu.2025.1555910/full
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https://www.frontiersin.org/articles/10.3389/fimmu.2025.1555910/full

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