Pre-ciliated tubal epithelial cells are prone to initiation of high-grade serous ovarian carcinoma

Abstract The distal region of the uterine (Fallopian) tube is commonly associated with high-grade serous carcinoma (HGSC), the predominant and most aggressive form of ovarian or extra-uterine cancer. Specific cell states and lineage dynamics of the adult tubal epithelium (TE) remain insufficiently u...

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Main Authors: Andrea Flesken-Nikitin, Coulter Q. Ralston, Dah-Jiun Fu, Andrea J. De Micheli, Daryl J. Phuong, Blaine A. Harlan, Christopher S. Ashe, Amanda P. Armstrong, David W. McKellar, Sangeeta Ghuwalewala, Lora H. Ellenson, John C. Schimenti, Benjamin D. Cosgrove, Alexander Yu. Nikitin
Format: Article
Language:English
Published: Nature Portfolio 2024-10-01
Series:Nature Communications
Online Access:https://doi.org/10.1038/s41467-024-52984-1
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author Andrea Flesken-Nikitin
Coulter Q. Ralston
Dah-Jiun Fu
Andrea J. De Micheli
Daryl J. Phuong
Blaine A. Harlan
Christopher S. Ashe
Amanda P. Armstrong
David W. McKellar
Sangeeta Ghuwalewala
Lora H. Ellenson
John C. Schimenti
Benjamin D. Cosgrove
Alexander Yu. Nikitin
author_facet Andrea Flesken-Nikitin
Coulter Q. Ralston
Dah-Jiun Fu
Andrea J. De Micheli
Daryl J. Phuong
Blaine A. Harlan
Christopher S. Ashe
Amanda P. Armstrong
David W. McKellar
Sangeeta Ghuwalewala
Lora H. Ellenson
John C. Schimenti
Benjamin D. Cosgrove
Alexander Yu. Nikitin
author_sort Andrea Flesken-Nikitin
collection DOAJ
description Abstract The distal region of the uterine (Fallopian) tube is commonly associated with high-grade serous carcinoma (HGSC), the predominant and most aggressive form of ovarian or extra-uterine cancer. Specific cell states and lineage dynamics of the adult tubal epithelium (TE) remain insufficiently understood, hindering efforts to determine the cell of origin for HGSC. Here, we report a comprehensive census of cell types and states of the mouse uterine tube. We show that distal TE cells expressing the stem/progenitor cell marker Slc1a3 can differentiate into both secretory (Ovgp1+) and ciliated (Fam183b+) cells. Inactivation of Trp53 and Rb1, whose pathways are commonly altered in HGSC, leads to elimination of targeted Slc1a3+ cells by apoptosis, thereby preventing their malignant transformation. In contrast, pre-ciliated cells (Krt5+, Prom1+, Trp73+) remain cancer-prone and give rise to serous tubal intraepithelial carcinomas and overt HGSC. These findings identify transitional pre-ciliated cells as a cancer-prone cell state and point to pre-ciliation mechanisms as diagnostic and therapeutic targets.
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spelling doaj-art-90512a4f897946e0b160380da2b3bd0c2024-11-24T12:32:29ZengNature PortfolioNature Communications2041-17232024-10-0115111610.1038/s41467-024-52984-1Pre-ciliated tubal epithelial cells are prone to initiation of high-grade serous ovarian carcinomaAndrea Flesken-Nikitin0Coulter Q. Ralston1Dah-Jiun Fu2Andrea J. De Micheli3Daryl J. Phuong4Blaine A. Harlan5Christopher S. Ashe6Amanda P. Armstrong7David W. McKellar8Sangeeta Ghuwalewala9Lora H. Ellenson10John C. Schimenti11Benjamin D. Cosgrove12Alexander Yu. Nikitin13Department of Biomedical Sciences, Cornell UniversityDepartment of Biomedical Sciences, Cornell UniversityDepartment of Biomedical Sciences, Cornell UniversityDepartment of Oncology and Children’s Research Center, University Children’s Hospital ZürichDepartment of Biomedical Sciences, Cornell UniversityDepartment of Biomedical Sciences, Cornell UniversityDepartment of Biomedical Sciences, Cornell UniversityDepartment of Biomedical Sciences, Cornell UniversityMeinig School of Biomedical Engineering, Cornell UniversityDepartment of Molecular Biology and Genetics, Cornell UniversityMemorial Sloan Kettering Cancer CenterDepartment of Biomedical Sciences, Cornell UniversityMeinig School of Biomedical Engineering, Cornell UniversityDepartment of Biomedical Sciences, Cornell UniversityAbstract The distal region of the uterine (Fallopian) tube is commonly associated with high-grade serous carcinoma (HGSC), the predominant and most aggressive form of ovarian or extra-uterine cancer. Specific cell states and lineage dynamics of the adult tubal epithelium (TE) remain insufficiently understood, hindering efforts to determine the cell of origin for HGSC. Here, we report a comprehensive census of cell types and states of the mouse uterine tube. We show that distal TE cells expressing the stem/progenitor cell marker Slc1a3 can differentiate into both secretory (Ovgp1+) and ciliated (Fam183b+) cells. Inactivation of Trp53 and Rb1, whose pathways are commonly altered in HGSC, leads to elimination of targeted Slc1a3+ cells by apoptosis, thereby preventing their malignant transformation. In contrast, pre-ciliated cells (Krt5+, Prom1+, Trp73+) remain cancer-prone and give rise to serous tubal intraepithelial carcinomas and overt HGSC. These findings identify transitional pre-ciliated cells as a cancer-prone cell state and point to pre-ciliation mechanisms as diagnostic and therapeutic targets.https://doi.org/10.1038/s41467-024-52984-1
spellingShingle Andrea Flesken-Nikitin
Coulter Q. Ralston
Dah-Jiun Fu
Andrea J. De Micheli
Daryl J. Phuong
Blaine A. Harlan
Christopher S. Ashe
Amanda P. Armstrong
David W. McKellar
Sangeeta Ghuwalewala
Lora H. Ellenson
John C. Schimenti
Benjamin D. Cosgrove
Alexander Yu. Nikitin
Pre-ciliated tubal epithelial cells are prone to initiation of high-grade serous ovarian carcinoma
Nature Communications
title Pre-ciliated tubal epithelial cells are prone to initiation of high-grade serous ovarian carcinoma
title_full Pre-ciliated tubal epithelial cells are prone to initiation of high-grade serous ovarian carcinoma
title_fullStr Pre-ciliated tubal epithelial cells are prone to initiation of high-grade serous ovarian carcinoma
title_full_unstemmed Pre-ciliated tubal epithelial cells are prone to initiation of high-grade serous ovarian carcinoma
title_short Pre-ciliated tubal epithelial cells are prone to initiation of high-grade serous ovarian carcinoma
title_sort pre ciliated tubal epithelial cells are prone to initiation of high grade serous ovarian carcinoma
url https://doi.org/10.1038/s41467-024-52984-1
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