Deficiency of Rab26 causes behavioral defects in mice through impaired trafficking of serotonin (5-HT) transporter

Summary: Rab proteins are key regulators of membrane trafficking. Dysregulated Rab proteins are associated with neurological diseases through the regulation of receptor endocytosis, recycling, and/or degradation. Rab26 is highly expressed in the brain, but its physiological function remains poorly e...

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Bibliographic Details
Main Authors: Yandan Ren, Ziyan Wang, Ziheng Wei, Ruijuan Zhuang, Yongtao Zhang, Xiaoxi Liu, Cong Jiang, Xuan Liu, Ye Yun, Yanfang Li, Wanjin Hong, Tuanlao Wang
Format: Article
Language:English
Published: Elsevier 2025-07-01
Series:iScience
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Online Access:http://www.sciencedirect.com/science/article/pii/S2589004225011927
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