The hepatocyte traffic network in the human hepatitis A virus biological cycle from an evolutionary perspective

Abstract Hepatitis A virus (HAV) egresses from hepatocytes cloaked in exosomes (eHAV). However, the traffic network used for its release from polarized hepatocytes is not completely understood. We propose that eHAV biogenesis may follow not only an ESCRT-mediated pathway but also the syndecan-synten...

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Main Authors: Albert Carcereny, Alba Arrebola, Gemma Chavarria-Miró, Montserrat de Castellarnau, Cristina Fuentes, David Garcia-Pedemonte, Adán Martínez-Velázquez, Enric Ribes, Albert Bosch, Susana Guix, Maria Isabel Costafreda, Rosa M. Pintó
Format: Article
Language:English
Published: Nature Portfolio 2025-06-01
Series:Communications Biology
Online Access:https://doi.org/10.1038/s42003-025-08344-w
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author Albert Carcereny
Alba Arrebola
Gemma Chavarria-Miró
Montserrat de Castellarnau
Cristina Fuentes
David Garcia-Pedemonte
Adán Martínez-Velázquez
Enric Ribes
Albert Bosch
Susana Guix
Maria Isabel Costafreda
Rosa M. Pintó
author_facet Albert Carcereny
Alba Arrebola
Gemma Chavarria-Miró
Montserrat de Castellarnau
Cristina Fuentes
David Garcia-Pedemonte
Adán Martínez-Velázquez
Enric Ribes
Albert Bosch
Susana Guix
Maria Isabel Costafreda
Rosa M. Pintó
author_sort Albert Carcereny
collection DOAJ
description Abstract Hepatitis A virus (HAV) egresses from hepatocytes cloaked in exosomes (eHAV). However, the traffic network used for its release from polarized hepatocytes is not completely understood. We propose that eHAV biogenesis may follow not only an ESCRT-mediated pathway but also the syndecan-syntenin-ALIX pathway. The Bro1 and the V domains of ALIX bind to the pX extension of VP1 and the VP2-late domains of the unmature capsid, respectively. A Serine-to-Glycine replacement at position 134 of VP2, closely located with the first late domain, facilitates the interaction with ALIX promoting the syndecan-syntenin-ALIX pathway and improving the basolateral egress, preferentially using RAB35. This replacement is conserved in hepatoviruses infecting a wide range of mammalian species, but not in hepatoviruses infecting chimpanzees and humans. An inefficient basolateral egress could be a strategy to escape the antiviral cellular response in apes.
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spelling doaj-art-7c27a7ed59d24df18e053ca2cf736da32025-08-20T03:21:06ZengNature PortfolioCommunications Biology2399-36422025-06-018111410.1038/s42003-025-08344-wThe hepatocyte traffic network in the human hepatitis A virus biological cycle from an evolutionary perspectiveAlbert Carcereny0Alba Arrebola1Gemma Chavarria-Miró2Montserrat de Castellarnau3Cristina Fuentes4David Garcia-Pedemonte5Adán Martínez-Velázquez6Enric Ribes7Albert Bosch8Susana Guix9Maria Isabel Costafreda10Rosa M. Pintó11Enteric Virus Laboratory, Department of Genetics, Microbiology and Statistics, School of Biology, and Institute of Nutrition and Food Safety, University of BarcelonaEnteric Virus Laboratory, Department of Genetics, Microbiology and Statistics, School of Biology, and Institute of Nutrition and Food Safety, University of BarcelonaEnteric Virus Laboratory, Department of Genetics, Microbiology and Statistics, School of Biology, and Institute of Nutrition and Food Safety, University of BarcelonaEnteric Virus Laboratory, Department of Genetics, Microbiology and Statistics, School of Biology, and Institute of Nutrition and Food Safety, University of BarcelonaEnteric Virus Laboratory, Department of Genetics, Microbiology and Statistics, School of Biology, and Institute of Nutrition and Food Safety, University of BarcelonaEnteric Virus Laboratory, Department of Genetics, Microbiology and Statistics, School of Biology, and Institute of Nutrition and Food Safety, University of BarcelonaEnteric Virus Laboratory, Department of Genetics, Microbiology and Statistics, School of Biology, and Institute of Nutrition and Food Safety, University of BarcelonaEnteric Virus Laboratory, Department of Cell Biology, Physiology and Immunology, School of Biology, University of BarcelonaEnteric Virus Laboratory, Department of Genetics, Microbiology and Statistics, School of Biology, and Institute of Nutrition and Food Safety, University of BarcelonaEnteric Virus Laboratory, Department of Genetics, Microbiology and Statistics, School of Biology, and Institute of Nutrition and Food Safety, University of BarcelonaEnteric Virus Laboratory, Department of Genetics, Microbiology and Statistics, School of Biology, and Institute of Nutrition and Food Safety, University of BarcelonaEnteric Virus Laboratory, Department of Genetics, Microbiology and Statistics, School of Biology, and Institute of Nutrition and Food Safety, University of BarcelonaAbstract Hepatitis A virus (HAV) egresses from hepatocytes cloaked in exosomes (eHAV). However, the traffic network used for its release from polarized hepatocytes is not completely understood. We propose that eHAV biogenesis may follow not only an ESCRT-mediated pathway but also the syndecan-syntenin-ALIX pathway. The Bro1 and the V domains of ALIX bind to the pX extension of VP1 and the VP2-late domains of the unmature capsid, respectively. A Serine-to-Glycine replacement at position 134 of VP2, closely located with the first late domain, facilitates the interaction with ALIX promoting the syndecan-syntenin-ALIX pathway and improving the basolateral egress, preferentially using RAB35. This replacement is conserved in hepatoviruses infecting a wide range of mammalian species, but not in hepatoviruses infecting chimpanzees and humans. An inefficient basolateral egress could be a strategy to escape the antiviral cellular response in apes.https://doi.org/10.1038/s42003-025-08344-w
spellingShingle Albert Carcereny
Alba Arrebola
Gemma Chavarria-Miró
Montserrat de Castellarnau
Cristina Fuentes
David Garcia-Pedemonte
Adán Martínez-Velázquez
Enric Ribes
Albert Bosch
Susana Guix
Maria Isabel Costafreda
Rosa M. Pintó
The hepatocyte traffic network in the human hepatitis A virus biological cycle from an evolutionary perspective
Communications Biology
title The hepatocyte traffic network in the human hepatitis A virus biological cycle from an evolutionary perspective
title_full The hepatocyte traffic network in the human hepatitis A virus biological cycle from an evolutionary perspective
title_fullStr The hepatocyte traffic network in the human hepatitis A virus biological cycle from an evolutionary perspective
title_full_unstemmed The hepatocyte traffic network in the human hepatitis A virus biological cycle from an evolutionary perspective
title_short The hepatocyte traffic network in the human hepatitis A virus biological cycle from an evolutionary perspective
title_sort hepatocyte traffic network in the human hepatitis a virus biological cycle from an evolutionary perspective
url https://doi.org/10.1038/s42003-025-08344-w
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