Neuromodulation of voltage-gated sodium channels by Gβ1γ2 subunits: Implications for GNB1-linked encephalopathy

Summary: Guanine nucleotide-binding protein Gβγ subunits are ubiquitous signaling molecules that interact with numerous effector proteins in neurons, including voltage-gated sodium, calcium, and potassium channels. We show that Gβγ subunits associate with voltage-gated sodium channels (Navs) in mous...

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Main Authors: Nicholas Denomme, Samantha L. Hodges, Luis Lopez-Santiago, Yukun Yuan, Julie M. Ziobro, Joe Minton, Chunling Chen, Yan Chen, Jacob M. Hull, James Offord, Alan V. Smrcka, Lori L. Isom
Format: Article
Language:English
Published: Elsevier 2025-09-01
Series:Neurobiology of Disease
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Online Access:http://www.sciencedirect.com/science/article/pii/S0969996125002062
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author Nicholas Denomme
Samantha L. Hodges
Luis Lopez-Santiago
Yukun Yuan
Julie M. Ziobro
Joe Minton
Chunling Chen
Yan Chen
Jacob M. Hull
James Offord
Alan V. Smrcka
Lori L. Isom
author_facet Nicholas Denomme
Samantha L. Hodges
Luis Lopez-Santiago
Yukun Yuan
Julie M. Ziobro
Joe Minton
Chunling Chen
Yan Chen
Jacob M. Hull
James Offord
Alan V. Smrcka
Lori L. Isom
author_sort Nicholas Denomme
collection DOAJ
description Summary: Guanine nucleotide-binding protein Gβγ subunits are ubiquitous signaling molecules that interact with numerous effector proteins in neurons, including voltage-gated sodium, calcium, and potassium channels. We show that Gβγ subunits associate with voltage-gated sodium channels (Navs) in mouse brain, and co-expression of a prominent Gβγ complex, Gβ1γ2, leads to functional inhibition of brain Nav α subunit subtypes Nav1.1 and Nav1.6 in heterologous cells. Gβ1γ2 co-expression shows subtype-selective effects on Nav1.1 and Nav1.6 in the presence of Navβ1 subunit co-expression, and in response to prepulse voltage changes. De novo variants in GNB1, encoding the Gβ1 subunit, are linked to GNB1 encephalopathy (GNB1-E). Using cortical slice electrophysiology, we show that the Gnb1K78R/+ mouse model of GNB1-E has reduced spontaneous GABAergic, but not glutamatergic, transmission and decreased sodium current density in dissociated parvalbumin-expressing GABAergic interneurons. This work advances our understanding of the epileptic mechanisms present in GNB1-E, including a previously unrecognized role for Navs.
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spelling doaj-art-796bca3e4fc14466bddc3c84bdd61c9f2025-08-20T03:32:23ZengElsevierNeurobiology of Disease1095-953X2025-09-0121310699010.1016/j.nbd.2025.106990Neuromodulation of voltage-gated sodium channels by Gβ1γ2 subunits: Implications for GNB1-linked encephalopathyNicholas Denomme0Samantha L. Hodges1Luis Lopez-Santiago2Yukun Yuan3Julie M. Ziobro4Joe Minton5Chunling Chen6Yan Chen7Jacob M. Hull8James Offord9Alan V. Smrcka10Lori L. Isom11Department of Pharmacology, University of Michigan Medical School, Ann Arbor, MI 48109, USADepartment of Pharmacology, University of Michigan Medical School, Ann Arbor, MI 48109, USADepartment of Pharmacology, University of Michigan Medical School, Ann Arbor, MI 48109, USADepartment of Pharmacology, University of Michigan Medical School, Ann Arbor, MI 48109, USADepartment of Pediatrics, University of Michigan Medical School, Ann Arbor, MI 48109, USADepartment of Pharmacology, University of Michigan Medical School, Ann Arbor, MI 48109, USADepartment of Pharmacology, University of Michigan Medical School, Ann Arbor, MI 48109, USADepartment of Pharmacology, University of Michigan Medical School, Ann Arbor, MI 48109, USADepartment of Pharmacology, University of Michigan Medical School, Ann Arbor, MI 48109, USADepartment of Pharmacology, University of Michigan Medical School, Ann Arbor, MI 48109, USADepartment of Pharmacology, University of Michigan Medical School, Ann Arbor, MI 48109, USADepartment of Pharmacology, University of Michigan Medical School, Ann Arbor, MI 48109, USA; Corresponding author.Summary: Guanine nucleotide-binding protein Gβγ subunits are ubiquitous signaling molecules that interact with numerous effector proteins in neurons, including voltage-gated sodium, calcium, and potassium channels. We show that Gβγ subunits associate with voltage-gated sodium channels (Navs) in mouse brain, and co-expression of a prominent Gβγ complex, Gβ1γ2, leads to functional inhibition of brain Nav α subunit subtypes Nav1.1 and Nav1.6 in heterologous cells. Gβ1γ2 co-expression shows subtype-selective effects on Nav1.1 and Nav1.6 in the presence of Navβ1 subunit co-expression, and in response to prepulse voltage changes. De novo variants in GNB1, encoding the Gβ1 subunit, are linked to GNB1 encephalopathy (GNB1-E). Using cortical slice electrophysiology, we show that the Gnb1K78R/+ mouse model of GNB1-E has reduced spontaneous GABAergic, but not glutamatergic, transmission and decreased sodium current density in dissociated parvalbumin-expressing GABAergic interneurons. This work advances our understanding of the epileptic mechanisms present in GNB1-E, including a previously unrecognized role for Navs.http://www.sciencedirect.com/science/article/pii/S0969996125002062Voltage-gated sodium channelG-proteinGNB1Developmental and epileptic encephalopathy
spellingShingle Nicholas Denomme
Samantha L. Hodges
Luis Lopez-Santiago
Yukun Yuan
Julie M. Ziobro
Joe Minton
Chunling Chen
Yan Chen
Jacob M. Hull
James Offord
Alan V. Smrcka
Lori L. Isom
Neuromodulation of voltage-gated sodium channels by Gβ1γ2 subunits: Implications for GNB1-linked encephalopathy
Neurobiology of Disease
Voltage-gated sodium channel
G-protein
GNB1
Developmental and epileptic encephalopathy
title Neuromodulation of voltage-gated sodium channels by Gβ1γ2 subunits: Implications for GNB1-linked encephalopathy
title_full Neuromodulation of voltage-gated sodium channels by Gβ1γ2 subunits: Implications for GNB1-linked encephalopathy
title_fullStr Neuromodulation of voltage-gated sodium channels by Gβ1γ2 subunits: Implications for GNB1-linked encephalopathy
title_full_unstemmed Neuromodulation of voltage-gated sodium channels by Gβ1γ2 subunits: Implications for GNB1-linked encephalopathy
title_short Neuromodulation of voltage-gated sodium channels by Gβ1γ2 subunits: Implications for GNB1-linked encephalopathy
title_sort neuromodulation of voltage gated sodium channels by gβ1γ2 subunits implications for gnb1 linked encephalopathy
topic Voltage-gated sodium channel
G-protein
GNB1
Developmental and epileptic encephalopathy
url http://www.sciencedirect.com/science/article/pii/S0969996125002062
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