PRMT1 Ablation in Endothelial Cells Causes Endothelial Dysfunction and Aggravates COPD Attributable to Dysregulated NF‐κB Signaling

PRMT1 Ablation in Endothelial Cells Causes Endothelial Dysfunction and Aggravates COPD Attributable to Dysregulated NF‐κB Signaling

Abstract Endothelial dysfunction and senescence are pivotal in pulmonary diseases, including chronic obstructive pulmonary disease (COPD). Protein arginine methyltransferase 1 (PRMT1) is the major enzyme responsible for asymmetric arginine dimethylation and plays a role in diverse biological process...

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Bibliographic Details
Main Authors: Thi Thuy Vy Tran, Yideul Jeong, Suwoo Kim, Ji Eun Yeom, Jinwoo Lee, Wonhwa Lee, Gyu‐Un Bae, Jong‐Sun Kang
Format: Article
Language:English
Published: Wiley 2025-05-01
Series:Advanced Science
Subjects:
COPD
endothelial dysfunction
NF‐κB
PRMT1
senescence
Online Access:https://doi.org/10.1002/advs.202411514
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https://doi.org/10.1002/advs.202411514

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