Targeting the major pro-inflammatory interleukin-6-type cytokine receptor gp130 by antagonistic single domain antibodies

IntroductionAlthough Interleukin (IL)-6-type cytokine signaling is critical for maintaining the body's homeostasis, aberrant signaling has been observed in numerous diseases including autoimmunity and cancer. Currently, all approved biologics that inhibit IL-6-type cytokines specifically target...

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Main Authors: Silke Pudewell, Julia Heuser, Inna Dorogobed, Britta Lipinski, Thi Hong Hue Tran, Pia Metzenmacher, Richard Kunze, Felix Geyer, Stefan Zielonka, Doreen M. Floss, Harald Kolmar, Jens M. Moll, Jürgen Scheller
Format: Article
Language:English
Published: Frontiers Media S.A. 2025-08-01
Series:Frontiers in Immunology
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Online Access:https://www.frontiersin.org/articles/10.3389/fimmu.2025.1613004/full
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author Silke Pudewell
Julia Heuser
Inna Dorogobed
Britta Lipinski
Thi Hong Hue Tran
Pia Metzenmacher
Richard Kunze
Felix Geyer
Stefan Zielonka
Doreen M. Floss
Harald Kolmar
Jens M. Moll
Jürgen Scheller
author_facet Silke Pudewell
Julia Heuser
Inna Dorogobed
Britta Lipinski
Thi Hong Hue Tran
Pia Metzenmacher
Richard Kunze
Felix Geyer
Stefan Zielonka
Doreen M. Floss
Harald Kolmar
Jens M. Moll
Jürgen Scheller
author_sort Silke Pudewell
collection DOAJ
description IntroductionAlthough Interleukin (IL)-6-type cytokine signaling is critical for maintaining the body's homeostasis, aberrant signaling has been observed in numerous diseases including autoimmunity and cancer. Currently, all approved biologics that inhibit IL-6-type cytokines specifically target the key pro-inflammatory mediator IL-6 or its receptor (IL6R). Historically, direct inhibition of glycoprotein 130 (gp130)—the shared transmembrane receptor for IL-6-type cytokines—was avoided due to concerns that broad suppression might cause more harm than benefit. However, this view is being reconsidered in light of the clinical success of Janus kinase (JAK) inhibitors, which broadly disrupt cytokine signaling, including pathways mediated by gp130.MethodsHere we developed four single domain antibodies (sdAb), consisting out of a camelid-derived nanobody and a human Fc-fragment, and characterized them by direct protein interaction analysis, epitope binding, epitope binning, as well as inhibition of cytokine-induced stimulation and proliferation of appropriate Ba/F3 cell lines and trans-migration in HT-29 cells.ResultsThe four sdAb-Fc constructs GP01-, GP11- GP13- and GP20-Fc bind directly to gp130 in the cytokine binding module (CBM) and largely inhibit IL-6-type cytokine signaling by interfering with the high-affinity binding site of IL-6, IL-11, CLCF1, CT1, CNTF, OSM and LIF. Furthermore, we functionally demonstrate the inhibitory effect of the selected nanobodies in cell-based transmigration assays of the human colorectal cancer cell line HT-29.DiscussionIn summary, our study has identified and characterized four novel inhibitory high-affinity gp130 nanobodies with potential for use in cytokine-dependent autoimmunity or cancer therapy.
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spelling doaj-art-59b3eee090e8459db916258a3399a2592025-08-20T04:01:00ZengFrontiers Media S.A.Frontiers in Immunology1664-32242025-08-011610.3389/fimmu.2025.16130041613004Targeting the major pro-inflammatory interleukin-6-type cytokine receptor gp130 by antagonistic single domain antibodiesSilke Pudewell0Julia Heuser1Inna Dorogobed2Britta Lipinski3Thi Hong Hue Tran4Pia Metzenmacher5Richard Kunze6Felix Geyer7Stefan Zielonka8Doreen M. Floss9Harald Kolmar10Jens M. Moll11Jürgen Scheller12Institute of Biochemistry and Molecular Biology II, Medical Faculty, Heinrich-Heine-University, Düsseldorf, GermanyInstitute of Biochemistry and Molecular Biology II, Medical Faculty, Heinrich-Heine-University, Düsseldorf, GermanyApplied Biochemistry, Institute for Organic Chemistry and Biochemistry, Technical University of Darmstadt, Darmstadt, GermanyBiomolecular Immunotherapy, Institute for Organic Chemistry and Biochemistry, Technical University of Darmstadt, Darmstadt, GermanyBiomolecular Immunotherapy, Institute for Organic Chemistry and Biochemistry, Technical University of Darmstadt, Darmstadt, GermanyInstitute of Biochemistry and Molecular Biology II, Medical Faculty, Heinrich-Heine-University, Düsseldorf, GermanyInstitute of Biochemistry and Molecular Biology II, Medical Faculty, Heinrich-Heine-University, Düsseldorf, GermanyApplied Biochemistry, Institute for Organic Chemistry and Biochemistry, Technical University of Darmstadt, Darmstadt, GermanyBiomolecular Immunotherapy, Institute for Organic Chemistry and Biochemistry, Technical University of Darmstadt, Darmstadt, GermanyInstitute of Biochemistry and Molecular Biology II, Medical Faculty, Heinrich-Heine-University, Düsseldorf, GermanyApplied Biochemistry, Institute for Organic Chemistry and Biochemistry, Technical University of Darmstadt, Darmstadt, GermanyInstitute of Biochemistry and Molecular Biology II, Medical Faculty, Heinrich-Heine-University, Düsseldorf, GermanyInstitute of Biochemistry and Molecular Biology II, Medical Faculty, Heinrich-Heine-University, Düsseldorf, GermanyIntroductionAlthough Interleukin (IL)-6-type cytokine signaling is critical for maintaining the body's homeostasis, aberrant signaling has been observed in numerous diseases including autoimmunity and cancer. Currently, all approved biologics that inhibit IL-6-type cytokines specifically target the key pro-inflammatory mediator IL-6 or its receptor (IL6R). Historically, direct inhibition of glycoprotein 130 (gp130)—the shared transmembrane receptor for IL-6-type cytokines—was avoided due to concerns that broad suppression might cause more harm than benefit. However, this view is being reconsidered in light of the clinical success of Janus kinase (JAK) inhibitors, which broadly disrupt cytokine signaling, including pathways mediated by gp130.MethodsHere we developed four single domain antibodies (sdAb), consisting out of a camelid-derived nanobody and a human Fc-fragment, and characterized them by direct protein interaction analysis, epitope binding, epitope binning, as well as inhibition of cytokine-induced stimulation and proliferation of appropriate Ba/F3 cell lines and trans-migration in HT-29 cells.ResultsThe four sdAb-Fc constructs GP01-, GP11- GP13- and GP20-Fc bind directly to gp130 in the cytokine binding module (CBM) and largely inhibit IL-6-type cytokine signaling by interfering with the high-affinity binding site of IL-6, IL-11, CLCF1, CT1, CNTF, OSM and LIF. Furthermore, we functionally demonstrate the inhibitory effect of the selected nanobodies in cell-based transmigration assays of the human colorectal cancer cell line HT-29.DiscussionIn summary, our study has identified and characterized four novel inhibitory high-affinity gp130 nanobodies with potential for use in cytokine-dependent autoimmunity or cancer therapy.https://www.frontiersin.org/articles/10.3389/fimmu.2025.1613004/fullIL-6-type cytokinesglycoprotein 130single domain antibodyinhibitorinflammation
spellingShingle Silke Pudewell
Julia Heuser
Inna Dorogobed
Britta Lipinski
Thi Hong Hue Tran
Pia Metzenmacher
Richard Kunze
Felix Geyer
Stefan Zielonka
Doreen M. Floss
Harald Kolmar
Jens M. Moll
Jürgen Scheller
Targeting the major pro-inflammatory interleukin-6-type cytokine receptor gp130 by antagonistic single domain antibodies
Frontiers in Immunology
IL-6-type cytokines
glycoprotein 130
single domain antibody
inhibitor
inflammation
title Targeting the major pro-inflammatory interleukin-6-type cytokine receptor gp130 by antagonistic single domain antibodies
title_full Targeting the major pro-inflammatory interleukin-6-type cytokine receptor gp130 by antagonistic single domain antibodies
title_fullStr Targeting the major pro-inflammatory interleukin-6-type cytokine receptor gp130 by antagonistic single domain antibodies
title_full_unstemmed Targeting the major pro-inflammatory interleukin-6-type cytokine receptor gp130 by antagonistic single domain antibodies
title_short Targeting the major pro-inflammatory interleukin-6-type cytokine receptor gp130 by antagonistic single domain antibodies
title_sort targeting the major pro inflammatory interleukin 6 type cytokine receptor gp130 by antagonistic single domain antibodies
topic IL-6-type cytokines
glycoprotein 130
single domain antibody
inhibitor
inflammation
url https://www.frontiersin.org/articles/10.3389/fimmu.2025.1613004/full
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