PGE2-Induced IDO1 Inhibits the Capacity of Fully Mature DCs to Elicit an In Vitro Antileukemic Immune Response

In the last years, dendritic cells (DC) have been evaluated for antitumor vaccination. Although DC-based vaccines have raised great expectations, their clinical translation has been largely disappointing. For these results, several explanations have been proposed. In particular,...

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Main Authors: Sara Trabanelli, Mariangela Lecciso, Valentina Salvestrini, Michele Cavo, Darina Očadlíková, Roberto M. Lemoli, Antonio Curti
Format: Article
Language:English
Published: Wiley 2015-01-01
Series:Journal of Immunology Research
Online Access:http://dx.doi.org/10.1155/2015/253191
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author Sara Trabanelli
Mariangela Lecciso
Valentina Salvestrini
Michele Cavo
Darina Očadlíková
Roberto M. Lemoli
Antonio Curti
author_facet Sara Trabanelli
Mariangela Lecciso
Valentina Salvestrini
Michele Cavo
Darina Očadlíková
Roberto M. Lemoli
Antonio Curti
author_sort Sara Trabanelli
collection DOAJ
description In the last years, dendritic cells (DC) have been evaluated for antitumor vaccination. Although DC-based vaccines have raised great expectations, their clinical translation has been largely disappointing. For these results, several explanations have been proposed. In particular, the concomitant expression by DCs of tolerogenic pathways, such as the immunosuppressive agent indoleamine 2,3-dioxygenase-1 (IDO1), has been demonstrated. The aim of this study is to evaluate both the stimulatory and the tolerogenic feature of monocyte-derived DCs (Mo-DCs) after maturation with PGE2. In particular, the role of IDO1 expression in PGE2-matured Mo-DCs has been addressed. Here we show that PGE2, which is required for full maturation of DCs, is one mediator of DC tolerance by enhancing IDO1. PGE2-mediated expression of IDO1 results in the production of kynurenine, in the generation of Tregs, and in the inhibition of either the allogeneic or the autologous antigen-specific stimulatory capacity of DCs. When pulsed with leukemic lysates and matured with PGE2, DCs are impaired in the induction of IFN-γ secreting CD4+ and CD8+ T cells due to IDO1 upregulation. Moreover, the inhibition of IDO1 enhances the antileukemic response. Overall, these results point toward the use of IDO1 inhibitors to enhance the vaccination capacity of DCs, matured with PGE2.
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spelling doaj-art-591510b08fbf4adcbae8a69e8031843d2025-08-20T02:05:21ZengWileyJournal of Immunology Research2314-88612314-71562015-01-01201510.1155/2015/253191253191PGE2-Induced IDO1 Inhibits the Capacity of Fully Mature DCs to Elicit an In Vitro Antileukemic Immune ResponseSara Trabanelli0Mariangela Lecciso1Valentina Salvestrini2Michele Cavo3Darina Očadlíková4Roberto M. Lemoli5Antonio Curti6Department of Specialistic, Diagnostic and Experimental Medicine, Institute of Hematology “L. & A. Seràgnoli”, University of Bologna, 40138 Bologna, ItalyDepartment of Specialistic, Diagnostic and Experimental Medicine, Institute of Hematology “L. & A. Seràgnoli”, University of Bologna, 40138 Bologna, ItalyDepartment of Specialistic, Diagnostic and Experimental Medicine, Institute of Hematology “L. & A. Seràgnoli”, University of Bologna, 40138 Bologna, ItalyDepartment of Specialistic, Diagnostic and Experimental Medicine, Institute of Hematology “L. & A. Seràgnoli”, University of Bologna, 40138 Bologna, ItalyDepartment of Specialistic, Diagnostic and Experimental Medicine, Institute of Hematology “L. & A. Seràgnoli”, University of Bologna, 40138 Bologna, ItalyDepartment of Internal Medicine (DiMI), University of Genoa and IRCCS Azienda Ospedaliera Universitaria S. Martino-IST, 16132 Genoa, ItalyDepartment of Specialistic, Diagnostic and Experimental Medicine, Institute of Hematology “L. & A. Seràgnoli”, University of Bologna, 40138 Bologna, ItalyIn the last years, dendritic cells (DC) have been evaluated for antitumor vaccination. Although DC-based vaccines have raised great expectations, their clinical translation has been largely disappointing. For these results, several explanations have been proposed. In particular, the concomitant expression by DCs of tolerogenic pathways, such as the immunosuppressive agent indoleamine 2,3-dioxygenase-1 (IDO1), has been demonstrated. The aim of this study is to evaluate both the stimulatory and the tolerogenic feature of monocyte-derived DCs (Mo-DCs) after maturation with PGE2. In particular, the role of IDO1 expression in PGE2-matured Mo-DCs has been addressed. Here we show that PGE2, which is required for full maturation of DCs, is one mediator of DC tolerance by enhancing IDO1. PGE2-mediated expression of IDO1 results in the production of kynurenine, in the generation of Tregs, and in the inhibition of either the allogeneic or the autologous antigen-specific stimulatory capacity of DCs. When pulsed with leukemic lysates and matured with PGE2, DCs are impaired in the induction of IFN-γ secreting CD4+ and CD8+ T cells due to IDO1 upregulation. Moreover, the inhibition of IDO1 enhances the antileukemic response. Overall, these results point toward the use of IDO1 inhibitors to enhance the vaccination capacity of DCs, matured with PGE2.http://dx.doi.org/10.1155/2015/253191
spellingShingle Sara Trabanelli
Mariangela Lecciso
Valentina Salvestrini
Michele Cavo
Darina Očadlíková
Roberto M. Lemoli
Antonio Curti
PGE2-Induced IDO1 Inhibits the Capacity of Fully Mature DCs to Elicit an In Vitro Antileukemic Immune Response
Journal of Immunology Research
title PGE2-Induced IDO1 Inhibits the Capacity of Fully Mature DCs to Elicit an In Vitro Antileukemic Immune Response
title_full PGE2-Induced IDO1 Inhibits the Capacity of Fully Mature DCs to Elicit an In Vitro Antileukemic Immune Response
title_fullStr PGE2-Induced IDO1 Inhibits the Capacity of Fully Mature DCs to Elicit an In Vitro Antileukemic Immune Response
title_full_unstemmed PGE2-Induced IDO1 Inhibits the Capacity of Fully Mature DCs to Elicit an In Vitro Antileukemic Immune Response
title_short PGE2-Induced IDO1 Inhibits the Capacity of Fully Mature DCs to Elicit an In Vitro Antileukemic Immune Response
title_sort pge2 induced ido1 inhibits the capacity of fully mature dcs to elicit an in vitro antileukemic immune response
url http://dx.doi.org/10.1155/2015/253191
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