Zwitterionic poly-carboxybetaine-dexamethasone conjugates do not alleviate cartilage degeneration and synovitis in the collagenase-induced osteoarthritis model in rats

Abstract Osteoarthritis is a degenerative joint disease for which there is yet to be a disease-modifying drug available in clinics. New drug candidates often fail due to a combination of poor pharmacokinetics as well as an inability to address the complex, multifactorial nature of osteoarthritis. To...

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Main Authors: Patrick Weber, Maryam Asadikorayem, Shipin Zhang, David Fercher, Kajetana Bevc, Sami Kauppinen, Tuomas Frondelius, Tianqi Zhang, Marina Fonti, Gonçalo Barreto, Mikko A.J. Finnilä, Marcy Zenobi-Wong
Format: Article
Language:English
Published: Nature Portfolio 2025-07-01
Series:Scientific Reports
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Online Access:https://doi.org/10.1038/s41598-025-93247-3
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author Patrick Weber
Maryam Asadikorayem
Shipin Zhang
David Fercher
Kajetana Bevc
Sami Kauppinen
Tuomas Frondelius
Tianqi Zhang
Marina Fonti
Gonçalo Barreto
Mikko A.J. Finnilä
Marcy Zenobi-Wong
author_facet Patrick Weber
Maryam Asadikorayem
Shipin Zhang
David Fercher
Kajetana Bevc
Sami Kauppinen
Tuomas Frondelius
Tianqi Zhang
Marina Fonti
Gonçalo Barreto
Mikko A.J. Finnilä
Marcy Zenobi-Wong
author_sort Patrick Weber
collection DOAJ
description Abstract Osteoarthritis is a degenerative joint disease for which there is yet to be a disease-modifying drug available in clinics. New drug candidates often fail due to a combination of poor pharmacokinetics as well as an inability to address the complex, multifactorial nature of osteoarthritis. To address these issues, we developed a zwitterionic poly-carboxybetaine acrylamide-dexamethasone (pCBAA-DEX) conjugate showing good cartilage penetration as well as anti-inflammatory and lubricating properties in previous in vitro studies. Here, we investigate the therapeutic potential of pCBAA-DEX in the collagenase-induced osteoarthritis (CIOA) model in rats. Upon induction of the model, animals received one-time, unilateral injections of either saline, DEX or pCBAA-DEX on day 4 (N = 8). On day 70, joint tissues were harvested and analyzed. While pCBAA-DEX achieved ~ 50% cartilage retention at the terminal timepoint, it did not prevent cartilage degeneration, synovial inflammation and synovial fibrosis, nor did DEX alone. Nevertheless, DEX and pCBAA-DEX slightly decreased the fibrosis levels in the synovium with DEX also decreasing the number of synovial lining layers. For the cartilage, DEX did not cause any notable differences, instead we observed an increase in cartilage degeneration in the pCBAA-DEX group. These findings challenge the previous in vitro results and motivate a substantial redesign of these conjugates and associated in vitro methods to reconsider them for the treatment of osteoarthritis.
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spelling doaj-art-57bf03d59bbf4c92a395b039954bd7c62025-08-20T04:01:34ZengNature PortfolioScientific Reports2045-23222025-07-0115111410.1038/s41598-025-93247-3Zwitterionic poly-carboxybetaine-dexamethasone conjugates do not alleviate cartilage degeneration and synovitis in the collagenase-induced osteoarthritis model in ratsPatrick Weber0Maryam Asadikorayem1Shipin Zhang2David Fercher3Kajetana Bevc4Sami Kauppinen5Tuomas Frondelius6Tianqi Zhang7Marina Fonti8Gonçalo Barreto9Mikko A.J. Finnilä10Marcy Zenobi-Wong11Tissue Engineering + Biofabrication Laboratory, Department of Health Sciences and Technology, ETH ZürichTissue Engineering + Biofabrication Laboratory, Department of Health Sciences and Technology, ETH ZürichTissue Engineering + Biofabrication Laboratory, Department of Health Sciences and Technology, ETH ZürichTissue Engineering + Biofabrication Laboratory, Department of Health Sciences and Technology, ETH ZürichTissue Engineering + Biofabrication Laboratory, Department of Health Sciences and Technology, ETH ZürichResearch Unit of Health Sciences and Technology, University of OuluResearch Unit of Health Sciences and Technology, University of OuluResearch Unit of Health Sciences and Technology, University of OuluTissue Engineering + Biofabrication Laboratory, Department of Health Sciences and Technology, ETH ZürichClinicum, Faculty of Medicine, University of Helsinki and Helsinki University HospitalResearch Unit of Health Sciences and Technology, University of OuluTissue Engineering + Biofabrication Laboratory, Department of Health Sciences and Technology, ETH ZürichAbstract Osteoarthritis is a degenerative joint disease for which there is yet to be a disease-modifying drug available in clinics. New drug candidates often fail due to a combination of poor pharmacokinetics as well as an inability to address the complex, multifactorial nature of osteoarthritis. To address these issues, we developed a zwitterionic poly-carboxybetaine acrylamide-dexamethasone (pCBAA-DEX) conjugate showing good cartilage penetration as well as anti-inflammatory and lubricating properties in previous in vitro studies. Here, we investigate the therapeutic potential of pCBAA-DEX in the collagenase-induced osteoarthritis (CIOA) model in rats. Upon induction of the model, animals received one-time, unilateral injections of either saline, DEX or pCBAA-DEX on day 4 (N = 8). On day 70, joint tissues were harvested and analyzed. While pCBAA-DEX achieved ~ 50% cartilage retention at the terminal timepoint, it did not prevent cartilage degeneration, synovial inflammation and synovial fibrosis, nor did DEX alone. Nevertheless, DEX and pCBAA-DEX slightly decreased the fibrosis levels in the synovium with DEX also decreasing the number of synovial lining layers. For the cartilage, DEX did not cause any notable differences, instead we observed an increase in cartilage degeneration in the pCBAA-DEX group. These findings challenge the previous in vitro results and motivate a substantial redesign of these conjugates and associated in vitro methods to reconsider them for the treatment of osteoarthritis.https://doi.org/10.1038/s41598-025-93247-3Zwitterionic polymersDexamethasoneCIOAComputed tomographySynovitis
spellingShingle Patrick Weber
Maryam Asadikorayem
Shipin Zhang
David Fercher
Kajetana Bevc
Sami Kauppinen
Tuomas Frondelius
Tianqi Zhang
Marina Fonti
Gonçalo Barreto
Mikko A.J. Finnilä
Marcy Zenobi-Wong
Zwitterionic poly-carboxybetaine-dexamethasone conjugates do not alleviate cartilage degeneration and synovitis in the collagenase-induced osteoarthritis model in rats
Scientific Reports
Zwitterionic polymers
Dexamethasone
CIOA
Computed tomography
Synovitis
title Zwitterionic poly-carboxybetaine-dexamethasone conjugates do not alleviate cartilage degeneration and synovitis in the collagenase-induced osteoarthritis model in rats
title_full Zwitterionic poly-carboxybetaine-dexamethasone conjugates do not alleviate cartilage degeneration and synovitis in the collagenase-induced osteoarthritis model in rats
title_fullStr Zwitterionic poly-carboxybetaine-dexamethasone conjugates do not alleviate cartilage degeneration and synovitis in the collagenase-induced osteoarthritis model in rats
title_full_unstemmed Zwitterionic poly-carboxybetaine-dexamethasone conjugates do not alleviate cartilage degeneration and synovitis in the collagenase-induced osteoarthritis model in rats
title_short Zwitterionic poly-carboxybetaine-dexamethasone conjugates do not alleviate cartilage degeneration and synovitis in the collagenase-induced osteoarthritis model in rats
title_sort zwitterionic poly carboxybetaine dexamethasone conjugates do not alleviate cartilage degeneration and synovitis in the collagenase induced osteoarthritis model in rats
topic Zwitterionic polymers
Dexamethasone
CIOA
Computed tomography
Synovitis
url https://doi.org/10.1038/s41598-025-93247-3
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