APA (7th ed.) Citation

Berg, T., Metzner, K., Bahrami, N., Wang, E., Koch, M., Eaton, P., . . . Kallenborn-Gerhardt, W. Redox-dependent activation of protein kinase G1α contributes to transient receptor potential cation channel subfamily V member 1-mediated acute nociceptive pain behavior. Taylor & Francis Group.

Chicago Style (17th ed.) Citation

Berg, Tim, Katharina Metzner, Nabil Bahrami, Elena Wang, Maximilian Koch, Philip Eaton, Achim Schmidtko, and Wiebke Kallenborn-Gerhardt. Redox-dependent Activation of Protein Kinase G1α Contributes to Transient Receptor Potential Cation Channel Subfamily V Member 1-mediated Acute Nociceptive Pain Behavior. Taylor & Francis Group.

MLA (9th ed.) Citation

Berg, Tim, et al. Redox-dependent Activation of Protein Kinase G1α Contributes to Transient Receptor Potential Cation Channel Subfamily V Member 1-mediated Acute Nociceptive Pain Behavior. Taylor & Francis Group.