Redox-dependent activation of protein kinase G1α contributes to transient receptor potential cation channel subfamily V member 1-mediated acute nociceptive pain behavior

Redox-dependent activation of protein kinase G1α contributes to transient receptor potential cation channel subfamily V member 1-mediated acute nociceptive pain behavior

Background Sensory neurons relay the pain signals to the brain via the nociceptive system. Notably, reactive oxygen species (ROS) serve as signaling molecules in the somatosensory system; however, their contribution to sensing noxious stimuli remains poorly understood.Methods Herein, the role of pro...

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Bibliographic Details
Main Authors: Tim Berg, Katharina Metzner, Nabil Bahrami, Elena Wang, Maximilian Koch, Philip Eaton, Achim Schmidtko, Wiebke Kallenborn-Gerhardt
Format: Article
Language:English
Published: Taylor & Francis Group 2025-12-01
Series:Redox Report
Subjects:
Pain
nociception
PKG1α
reactive oxygen species
TRPV1
capsaicin
Online Access:https://www.tandfonline.com/doi/10.1080/13510002.2025.2549954
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https://www.tandfonline.com/doi/10.1080/13510002.2025.2549954

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