Helicobacter pylori CagA elevates FTO to induce gastric cancer progression via a “hit‐and‐run” paradigm

Helicobacter pylori CagA elevates FTO to induce gastric cancer progression via a “hit‐and‐run” paradigm

Abstract Background Helicobacter pylori (H. pylori) infection contributes significantly to gastric cancer (GC) progression. The intrinsic mechanisms of H. pylori‐host interactions and their role in promoting GC progression need further investigation. In this study, we explored the potential role of...

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Bibliographic Details
Main Authors: Bing He, Yiyang Hu, Yuyun Wu, Chao Wang, Limin Gao, Chunli Gong, Zhibin Li, Nannan Gao, Huan Yang, Yufeng Xiao, Shiming Yang
Format: Article
Language:English
Published: Wiley 2025-05-01
Series:Cancer Communications
Subjects:
Epithelial‐Mesenchymal transition
FTO
Gastric cancer
Helicobacter pylori
m6A modification
Online Access:https://doi.org/10.1002/cac2.70004
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https://doi.org/10.1002/cac2.70004

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