Hypoxia Induced Downregulation of Na+/H+ Exchanger-1 Activity Decreases Tumor Cell Proliferation

Objectives: Hypoxia and acidosis are the hallmarks of proliferative tumor microenvironment which can modulate the expression and function of Na+/H+ exchanger-1 (NHE1) via hypoxia inducible factor 1 (Hif). Here, we investigate the severity and time dependent effects of chronic hypoxia on NHE1 activit...

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Main Authors: Gül Şimşek, Hilmi Burak Kandilci
Format: Article
Language:English
Published: Galenos Publishing House 2025-03-01
Series:Ankara Üniversitesi Tıp Fakültesi Mecmuas
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Online Access:https://www.ankaratipfakultesimecmuasi.net/articles/hypoxia-induced-downregulation-of-nalesssupgreaterlesssupgreaterhlesssupgreaterlesssupgreater-exchanger-1-activity-decreases-tumor-cell-proliferation/doi/atfm.galenos.2025.98624
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author Gül Şimşek
Hilmi Burak Kandilci
author_facet Gül Şimşek
Hilmi Burak Kandilci
author_sort Gül Şimşek
collection DOAJ
description Objectives: Hypoxia and acidosis are the hallmarks of proliferative tumor microenvironment which can modulate the expression and function of Na+/H+ exchanger-1 (NHE1) via hypoxia inducible factor 1 (Hif). Here, we investigate the severity and time dependent effects of chronic hypoxia on NHE1 activity and its correlation with cell proliferation in mouse atrium tumor derived HL-1 cells. Materials and Methods: NHE1 activity was recorded using intracellular pH (pHi) sensitive dye cSNARF-1 (Leica SP5). Cell proliferation was assessed by live cell movie analyzer (Nanoentek, JuLI Br®) or immunofluorescence method. Results: According to our results, mild chronic hypoxia (2% O2, 48 hours) or shorter duration severe chronic hypoxia (1% O2, 24 hours) did not affect cell proliferation and NHE1 activity. In contrast, long term dimethyloxalylglycine (DMOG, Hif stabilizer) or zoniporide (NHE1 inhibitor) incubations (21% O2, 24/48 hours) suppressed cell proliferation. Conclusion: When our published and current results in this study interpreted together, at a critical level and duration of chronic hypoxia, Hif mediated downregulation of NHE1 activity could suppress tumor cell proliferation regardless of the well-known anti-proliferative early term direct effects of Hif. Therefore, restriction of NHE1 activity in tumor hypoxia is an important alternative target in regulating anti-proliferative action against tumor cells.
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spelling doaj-art-3ef93aa0c6244148b6c4ffa44626aafa2025-08-20T03:50:59ZengGalenos Publishing HouseAnkara Üniversitesi Tıp Fakültesi Mecmuas1307-56082025-03-01781495410.4274/atfm.galenos.2025.98624Hypoxia Induced Downregulation of Na+/H+ Exchanger-1 Activity Decreases Tumor Cell ProliferationGül Şimşek0https://orcid.org/0000-0002-3056-6414Hilmi Burak Kandilci1https://orcid.org/0000-0003-3050-6443Ankara University Faculty of Medicine Department of Biophysics, Ankara, TürkiyeAnkara University Faculty of Medicine Department of Biophysics, Ankara, TürkiyeObjectives: Hypoxia and acidosis are the hallmarks of proliferative tumor microenvironment which can modulate the expression and function of Na+/H+ exchanger-1 (NHE1) via hypoxia inducible factor 1 (Hif). Here, we investigate the severity and time dependent effects of chronic hypoxia on NHE1 activity and its correlation with cell proliferation in mouse atrium tumor derived HL-1 cells. Materials and Methods: NHE1 activity was recorded using intracellular pH (pHi) sensitive dye cSNARF-1 (Leica SP5). Cell proliferation was assessed by live cell movie analyzer (Nanoentek, JuLI Br®) or immunofluorescence method. Results: According to our results, mild chronic hypoxia (2% O2, 48 hours) or shorter duration severe chronic hypoxia (1% O2, 24 hours) did not affect cell proliferation and NHE1 activity. In contrast, long term dimethyloxalylglycine (DMOG, Hif stabilizer) or zoniporide (NHE1 inhibitor) incubations (21% O2, 24/48 hours) suppressed cell proliferation. Conclusion: When our published and current results in this study interpreted together, at a critical level and duration of chronic hypoxia, Hif mediated downregulation of NHE1 activity could suppress tumor cell proliferation regardless of the well-known anti-proliferative early term direct effects of Hif. Therefore, restriction of NHE1 activity in tumor hypoxia is an important alternative target in regulating anti-proliferative action against tumor cells.https://www.ankaratipfakultesimecmuasi.net/articles/hypoxia-induced-downregulation-of-nalesssupgreaterlesssupgreaterhlesssupgreaterlesssupgreater-exchanger-1-activity-decreases-tumor-cell-proliferation/doi/atfm.galenos.2025.98624cell culture techniqueshypoxiasodium-hydrogen exchangersneoplasms
spellingShingle Gül Şimşek
Hilmi Burak Kandilci
Hypoxia Induced Downregulation of Na+/H+ Exchanger-1 Activity Decreases Tumor Cell Proliferation
Ankara Üniversitesi Tıp Fakültesi Mecmuas
cell culture techniques
hypoxia
sodium-hydrogen exchangers
neoplasms
title Hypoxia Induced Downregulation of Na+/H+ Exchanger-1 Activity Decreases Tumor Cell Proliferation
title_full Hypoxia Induced Downregulation of Na+/H+ Exchanger-1 Activity Decreases Tumor Cell Proliferation
title_fullStr Hypoxia Induced Downregulation of Na+/H+ Exchanger-1 Activity Decreases Tumor Cell Proliferation
title_full_unstemmed Hypoxia Induced Downregulation of Na+/H+ Exchanger-1 Activity Decreases Tumor Cell Proliferation
title_short Hypoxia Induced Downregulation of Na+/H+ Exchanger-1 Activity Decreases Tumor Cell Proliferation
title_sort hypoxia induced downregulation of na h exchanger 1 activity decreases tumor cell proliferation
topic cell culture techniques
hypoxia
sodium-hydrogen exchangers
neoplasms
url https://www.ankaratipfakultesimecmuasi.net/articles/hypoxia-induced-downregulation-of-nalesssupgreaterlesssupgreaterhlesssupgreaterlesssupgreater-exchanger-1-activity-decreases-tumor-cell-proliferation/doi/atfm.galenos.2025.98624
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