Dehydroacteoside rejuvenates senescence via TVP23C-CDRT4 regulation

Dehydroacteoside rejuvenates senescence via TVP23C-CDRT4 regulation

One of the major factors inducing senescence is reactive oxygen species (ROS) produced from dysfunctional mitochondria. Therapeutic strategies that reduce mitochondrial ROS generation are considered essential for rejuvenating senescence, but effective methods have not yet been established. Here, we...

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Bibliographic Details
Main Authors: Yoo Jin Lee, Eun Seon Song, Yun Haeng Lee, Kyeong Seon Lee, Byeonghyeon So, Ji Ho Park, Jee Hee Yoon, Duyeol Kim, Minseon Kim, Hyung Wook Kwon, Youngjoo Byun, Ki Yong Lee, Joon Tae Park
Format: Article
Language:English
Published: Elsevier 2025-08-01
Series:Experimental Gerontology
Subjects:
Senescence rejuvenation
Dehydroacteoside
Reactive oxygen species
TVP23C-CDRT4
Online Access:http://www.sciencedirect.com/science/article/pii/S0531556525001299
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