Stromal vascular fraction inhibits renal fibrosis by regulating metabolism and inflammation in obstructive nephropathy

Obstructive nephropathy is one of the leading causes of kidney injury and fibrosis, which can lead to end-stage renal disease (ESRD). Stromal vascular fraction (SVF), a heterogeneous cell mixture derived from adipose tissue, has been widely used for regenerative medicine across many preclinical mode...

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Main Authors: Guang Yue, Yunjie Yang, Hongshuai Jia, Yangyang Wu, Lifei Ma, Xiaoyu Yi, Yuandong Tao, Huixia Zhou
Format: Article
Language:English
Published: Frontiers Media S.A. 2025-05-01
Series:Frontiers in Pharmacology
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Online Access:https://www.frontiersin.org/articles/10.3389/fphar.2025.1559446/full
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author Guang Yue
Guang Yue
Yunjie Yang
Hongshuai Jia
Hongshuai Jia
Yangyang Wu
Yangyang Wu
Lifei Ma
Xiaoyu Yi
Yuandong Tao
Huixia Zhou
Huixia Zhou
author_facet Guang Yue
Guang Yue
Yunjie Yang
Hongshuai Jia
Hongshuai Jia
Yangyang Wu
Yangyang Wu
Lifei Ma
Xiaoyu Yi
Yuandong Tao
Huixia Zhou
Huixia Zhou
author_sort Guang Yue
collection DOAJ
description Obstructive nephropathy is one of the leading causes of kidney injury and fibrosis, which can lead to end-stage renal disease (ESRD). Stromal vascular fraction (SVF), a heterogeneous cell mixture derived from adipose tissue, has been widely used for regenerative medicine across many preclinical models and clinical applications. Recent studies have suggested that SVF can alleviate acute kidney injury in mice. However, to our knowledge, the therapeutic effects of SVF on obstructive nephropathy have not been studied before. In this study, we evaluated the therapeutic potential of SVF on obstructive nephropathy in mice with unilateral ureteral obstruction (UUO). We revealed that autologous SVF administration mitigated UUO-induced renal fibrosis. SVF treatment inhibited both the infiltration of neutrophils and CD4+ T cells, as well as the production of inflammatory cytokines. Moreover, SVF promoted metabolic reprogramming and improved mitochondrial function in the obstructed kidneys, partially through PPAR pathway activation. Mechanistically, SVF-mediated PPAR activation inhibited the epithelial-mesenchymal transition (EMT) process of tubular cells, thus alleviating renal fibrosis in UUO mice. We further confirmed that pharmacological activation of PPAR pathway significantly reduced fibrosis in UUO kidneys. Therefore, our study suggests that SVF may represent a promising therapeutic strategy for obstructive nephropathy.
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publisher Frontiers Media S.A.
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spelling doaj-art-33fa336e9a704c13b1c0c8ce041a86e82025-08-20T02:57:36ZengFrontiers Media S.A.Frontiers in Pharmacology1663-98122025-05-011610.3389/fphar.2025.15594461559446Stromal vascular fraction inhibits renal fibrosis by regulating metabolism and inflammation in obstructive nephropathyGuang Yue0Guang Yue1Yunjie Yang2Hongshuai Jia3Hongshuai Jia4Yangyang Wu5Yangyang Wu6Lifei Ma7Xiaoyu Yi8Yuandong Tao9Huixia Zhou10Huixia Zhou11Department of Pediatric Urology, The Seventh Medical Center of Chinese PLA General Hospital, Beijing, ChinaMedical School of Chinese PLA, Beijing, ChinaDepartment of Pediatric Surgery, The Sixth Affiliated Hospital, School of Medicine, South China University of Technology, Foshan, ChinaDepartment of Pediatric Urology, The Seventh Medical Center of Chinese PLA General Hospital, Beijing, ChinaMedical School of Chinese PLA, Beijing, ChinaDepartment of Pediatric Urology, The Seventh Medical Center of Chinese PLA General Hospital, Beijing, ChinaMedical School of Chinese PLA, Beijing, ChinaDepartment of Pediatric Urology, The Seventh Medical Center of Chinese PLA General Hospital, Beijing, ChinaDepartment of Pediatric Urology, The Seventh Medical Center of Chinese PLA General Hospital, Beijing, ChinaDepartment of Pediatric Urology, The Seventh Medical Center of Chinese PLA General Hospital, Beijing, ChinaDepartment of Pediatric Urology, The Seventh Medical Center of Chinese PLA General Hospital, Beijing, ChinaMedical School of Chinese PLA, Beijing, ChinaObstructive nephropathy is one of the leading causes of kidney injury and fibrosis, which can lead to end-stage renal disease (ESRD). Stromal vascular fraction (SVF), a heterogeneous cell mixture derived from adipose tissue, has been widely used for regenerative medicine across many preclinical models and clinical applications. Recent studies have suggested that SVF can alleviate acute kidney injury in mice. However, to our knowledge, the therapeutic effects of SVF on obstructive nephropathy have not been studied before. In this study, we evaluated the therapeutic potential of SVF on obstructive nephropathy in mice with unilateral ureteral obstruction (UUO). We revealed that autologous SVF administration mitigated UUO-induced renal fibrosis. SVF treatment inhibited both the infiltration of neutrophils and CD4+ T cells, as well as the production of inflammatory cytokines. Moreover, SVF promoted metabolic reprogramming and improved mitochondrial function in the obstructed kidneys, partially through PPAR pathway activation. Mechanistically, SVF-mediated PPAR activation inhibited the epithelial-mesenchymal transition (EMT) process of tubular cells, thus alleviating renal fibrosis in UUO mice. We further confirmed that pharmacological activation of PPAR pathway significantly reduced fibrosis in UUO kidneys. Therefore, our study suggests that SVF may represent a promising therapeutic strategy for obstructive nephropathy.https://www.frontiersin.org/articles/10.3389/fphar.2025.1559446/fullSVFobstructive nephropathyrenal fibrosisinflammationPPAR
spellingShingle Guang Yue
Guang Yue
Yunjie Yang
Hongshuai Jia
Hongshuai Jia
Yangyang Wu
Yangyang Wu
Lifei Ma
Xiaoyu Yi
Yuandong Tao
Huixia Zhou
Huixia Zhou
Stromal vascular fraction inhibits renal fibrosis by regulating metabolism and inflammation in obstructive nephropathy
Frontiers in Pharmacology
SVF
obstructive nephropathy
renal fibrosis
inflammation
PPAR
title Stromal vascular fraction inhibits renal fibrosis by regulating metabolism and inflammation in obstructive nephropathy
title_full Stromal vascular fraction inhibits renal fibrosis by regulating metabolism and inflammation in obstructive nephropathy
title_fullStr Stromal vascular fraction inhibits renal fibrosis by regulating metabolism and inflammation in obstructive nephropathy
title_full_unstemmed Stromal vascular fraction inhibits renal fibrosis by regulating metabolism and inflammation in obstructive nephropathy
title_short Stromal vascular fraction inhibits renal fibrosis by regulating metabolism and inflammation in obstructive nephropathy
title_sort stromal vascular fraction inhibits renal fibrosis by regulating metabolism and inflammation in obstructive nephropathy
topic SVF
obstructive nephropathy
renal fibrosis
inflammation
PPAR
url https://www.frontiersin.org/articles/10.3389/fphar.2025.1559446/full
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