Targeting the ERK1/2 and p38 MAPK pathways attenuates Golgi tethering factor golgin-97 depletion-induced cancer progression in breast cancer

Targeting the ERK1/2 and p38 MAPK pathways attenuates Golgi tethering factor golgin-97 depletion-induced cancer progression in breast cancer

Abstract Background The Golgi apparatus is widely considered a secretory center and a hub for different signaling pathways. Abnormalities in Golgi dynamics can perturb the tumor microenvironment and influence cell migration. Therefore, unraveling the regulatory network of the Golgi and searching for...

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Main Authors: Yu-Chin Liu, Tsung-Jen Lin, Kowit-Yu Chong, Guan-Ying Chen, Chia-Yu Kuo, Yi-Yun Lin, Chia-Wei Chang, Ting-Feng Hsiao, Chih-Liang Wang, Yo-Chen Shih, Chia-Jung Yu
Format: Article
Language:English
Published: BMC 2025-01-01
Series:Cell Communication and Signaling
Subjects:
Golgi
Golgin-97
Breast cancer
MAPK signaling
Metastasis
Inflammatory cytokine
Online Access:https://doi.org/10.1186/s12964-024-02010-0
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https://doi.org/10.1186/s12964-024-02010-0

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