miR-1, miR-133a, miR-29b and skeletal muscle fibrosis in chronic limb-threatening ischaemia

Abstract Chronic limb-threatening ischaemia (CLTI), the most severe manifestation of peripheral arterial disease (PAD), is associated with a poor prognosis and high amputation rates. Despite novel therapeutic approaches being investigated, no significant clinical benefits have been observed yet. Und...

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Main Authors: Alan J. Keane, Clara Sanz-Nogués, Dulan Jayasooriya, Michael Creane, Xizhe Chen, Caomhán J. Lyons, Isha Sikri, Katarzyna Goljanek-Whysall, Timothy O’Brien
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Language:English
Published: Nature Portfolio 2024-11-01
Series:Scientific Reports
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Online Access:https://doi.org/10.1038/s41598-024-76415-9
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author Alan J. Keane
Clara Sanz-Nogués
Dulan Jayasooriya
Michael Creane
Xizhe Chen
Caomhán J. Lyons
Isha Sikri
Katarzyna Goljanek-Whysall
Timothy O’Brien
author_facet Alan J. Keane
Clara Sanz-Nogués
Dulan Jayasooriya
Michael Creane
Xizhe Chen
Caomhán J. Lyons
Isha Sikri
Katarzyna Goljanek-Whysall
Timothy O’Brien
author_sort Alan J. Keane
collection DOAJ
description Abstract Chronic limb-threatening ischaemia (CLTI), the most severe manifestation of peripheral arterial disease (PAD), is associated with a poor prognosis and high amputation rates. Despite novel therapeutic approaches being investigated, no significant clinical benefits have been observed yet. Understanding the molecular pathways of skeletal muscle dysfunction in CLTI is crucial for designing successful treatments. This study aimed to identify miRNAs dysregulated in muscle biopsies from PAD cohorts. Using MIcroRNA ENrichment TURned NETwork (MIENTURNET) on a publicly accessible RNA-sequencing dataset of PAD cohorts, we identified a list of miRNAs that were over-represented among the upregulated differentially expressed genes (DEGs) in CLTI. Next, we validated the altered expression of these miRNAs and their targets in mice with hindlimb ischaemia (HLI). Our results showed a significant downregulation of miR-1, miR-133a, and miR-29b levels in the ischaemic limbs versus the contralateral non-ischaemic limb. A miRNA target protein-protein interaction network identified extracellular matrix components, including collagen-1a1, -3a1, and -4a1, fibronectin-1, fibrin-1, matrix metalloproteinase-2 and -14, and Sparc, which were upregulated in the ischaemic muscle of mice. This is the first study to identify miR-1, miR-133a, and miR-29b as potential contributors to fibrosis and vascular pathology in CLTI muscle, which supports their potential as novel therapeutic agents for this condition.
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spelling doaj-art-166524dc20094da3b7a8d6a8fafaef6e2025-08-20T02:49:17ZengNature PortfolioScientific Reports2045-23222024-11-0114111710.1038/s41598-024-76415-9miR-1, miR-133a, miR-29b and skeletal muscle fibrosis in chronic limb-threatening ischaemiaAlan J. Keane0Clara Sanz-Nogués1Dulan Jayasooriya2Michael Creane3Xizhe Chen4Caomhán J. Lyons5Isha Sikri6Katarzyna Goljanek-Whysall7Timothy O’Brien8Regenerative Medicine Institute (REMEDI), University of GalwayRegenerative Medicine Institute (REMEDI), University of GalwayRegenerative Medicine Institute (REMEDI), University of GalwayRegenerative Medicine Institute (REMEDI), University of GalwayRegenerative Medicine Institute (REMEDI), University of GalwayRegenerative Medicine Institute (REMEDI), University of GalwayRegenerative Medicine Institute (REMEDI), University of GalwayRegenerative Medicine Institute (REMEDI), University of GalwayRegenerative Medicine Institute (REMEDI), University of GalwayAbstract Chronic limb-threatening ischaemia (CLTI), the most severe manifestation of peripheral arterial disease (PAD), is associated with a poor prognosis and high amputation rates. Despite novel therapeutic approaches being investigated, no significant clinical benefits have been observed yet. Understanding the molecular pathways of skeletal muscle dysfunction in CLTI is crucial for designing successful treatments. This study aimed to identify miRNAs dysregulated in muscle biopsies from PAD cohorts. Using MIcroRNA ENrichment TURned NETwork (MIENTURNET) on a publicly accessible RNA-sequencing dataset of PAD cohorts, we identified a list of miRNAs that were over-represented among the upregulated differentially expressed genes (DEGs) in CLTI. Next, we validated the altered expression of these miRNAs and their targets in mice with hindlimb ischaemia (HLI). Our results showed a significant downregulation of miR-1, miR-133a, and miR-29b levels in the ischaemic limbs versus the contralateral non-ischaemic limb. A miRNA target protein-protein interaction network identified extracellular matrix components, including collagen-1a1, -3a1, and -4a1, fibronectin-1, fibrin-1, matrix metalloproteinase-2 and -14, and Sparc, which were upregulated in the ischaemic muscle of mice. This is the first study to identify miR-1, miR-133a, and miR-29b as potential contributors to fibrosis and vascular pathology in CLTI muscle, which supports their potential as novel therapeutic agents for this condition.https://doi.org/10.1038/s41598-024-76415-9Chronic limb-threatening ischaemiaFibrosisMuscle regenerationMicroRNAs
spellingShingle Alan J. Keane
Clara Sanz-Nogués
Dulan Jayasooriya
Michael Creane
Xizhe Chen
Caomhán J. Lyons
Isha Sikri
Katarzyna Goljanek-Whysall
Timothy O’Brien
miR-1, miR-133a, miR-29b and skeletal muscle fibrosis in chronic limb-threatening ischaemia
Scientific Reports
Chronic limb-threatening ischaemia
Fibrosis
Muscle regeneration
MicroRNAs
title miR-1, miR-133a, miR-29b and skeletal muscle fibrosis in chronic limb-threatening ischaemia
title_full miR-1, miR-133a, miR-29b and skeletal muscle fibrosis in chronic limb-threatening ischaemia
title_fullStr miR-1, miR-133a, miR-29b and skeletal muscle fibrosis in chronic limb-threatening ischaemia
title_full_unstemmed miR-1, miR-133a, miR-29b and skeletal muscle fibrosis in chronic limb-threatening ischaemia
title_short miR-1, miR-133a, miR-29b and skeletal muscle fibrosis in chronic limb-threatening ischaemia
title_sort mir 1 mir 133a mir 29b and skeletal muscle fibrosis in chronic limb threatening ischaemia
topic Chronic limb-threatening ischaemia
Fibrosis
Muscle regeneration
MicroRNAs
url https://doi.org/10.1038/s41598-024-76415-9
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