BHLHE40 contributes to allergic asthma progression in mice through NRTN downregulation in macrophages

Abstract Alternatively activated M2-like macrophages have a profound impact on asthma pathogenesis. Basic Helix-Loop-Helix Family Member E40 (BHLHE40), a dimeric transcriptional factor, plays a key regulation in macrophage functions. Here we show that ovalbumin (OVA)-challenged mice exhibited greate...

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Main Authors: Bing Dai, Li Chen, Xiaowen Li, Qianlan Zhou, Qinzhen Zhang, Lina Han, Wenxin Shen, Qing Chang, Yuhong Zhao, Si Liu, Lishen Shan
Format: Article
Language:English
Published: Nature Portfolio 2025-06-01
Series:Communications Biology
Online Access:https://doi.org/10.1038/s42003-025-08288-1
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author Bing Dai
Li Chen
Xiaowen Li
Qianlan Zhou
Qinzhen Zhang
Lina Han
Wenxin Shen
Qing Chang
Yuhong Zhao
Si Liu
Lishen Shan
author_facet Bing Dai
Li Chen
Xiaowen Li
Qianlan Zhou
Qinzhen Zhang
Lina Han
Wenxin Shen
Qing Chang
Yuhong Zhao
Si Liu
Lishen Shan
author_sort Bing Dai
collection DOAJ
description Abstract Alternatively activated M2-like macrophages have a profound impact on asthma pathogenesis. Basic Helix-Loop-Helix Family Member E40 (BHLHE40), a dimeric transcriptional factor, plays a key regulation in macrophage functions. Here we show that ovalbumin (OVA)-challenged mice exhibited greater expression of BHLHE40 in lung tissues. Bhlhe40 knockdown reduced the pulmonary lesions and allergy-induced inflammation in asthmatic mice. Moreover, an inhibitory effect of Bhlhe40 knockdown on alternative activation was observed in vivo and in vitro. We show a downstream target Neurturin (Nrtn) of Bhlhe40. Dual luciferase assay and ChIP-qPCR assay indicated that BHLHE40 bound to Nrtn promoter and reduced its transcriptional activity. Simultaneous knockdown of Bhlhe40 and Nrtn recovered the alternative activation of macrophages and rescued the OVA-elicited asthma phenotype. NRTN downregulation offset the alleviative effects of Bhlhe40 knockdown on asthma. This study demonstrates that BHLHE40 promotes allergic asthma, and contributes to the alternative activation of macrophages in asthma by inhibiting Nrtn transcription.
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issn 2399-3642
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publisher Nature Portfolio
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spelling doaj-art-101174fa397f45fda571c50bbcf8cd782025-08-20T03:25:19ZengNature PortfolioCommunications Biology2399-36422025-06-018111510.1038/s42003-025-08288-1BHLHE40 contributes to allergic asthma progression in mice through NRTN downregulation in macrophagesBing Dai0Li Chen1Xiaowen Li2Qianlan Zhou3Qinzhen Zhang4Lina Han5Wenxin Shen6Qing Chang7Yuhong Zhao8Si Liu9Lishen Shan10Department of Pediatrics, Shengjing Hospital of China Medical UniversityDepartment of Pediatrics, Shengjing Hospital of China Medical UniversityDepartment of Pediatrics, Shengjing Hospital of China Medical UniversityDepartment of Pediatrics, Shengjing Hospital of China Medical UniversityDepartment of Pediatrics, Shengjing Hospital of China Medical UniversityDepartment of Pediatrics, Shengjing Hospital of China Medical UniversityDepartment of Pediatrics, Shengjing Hospital of China Medical UniversityLiaoning Key Laboratory of Precision Medical Research on Major Chronic Disease, Clinical Research Center of Shengjing Hospital of China Medical UniversityDepartment of Clinical Epidemiology, Shengjing Hospital of China Medical UniversityDepartment of Pediatrics, Shengjing Hospital of China Medical UniversityDepartment of Pediatrics, Shengjing Hospital of China Medical UniversityAbstract Alternatively activated M2-like macrophages have a profound impact on asthma pathogenesis. Basic Helix-Loop-Helix Family Member E40 (BHLHE40), a dimeric transcriptional factor, plays a key regulation in macrophage functions. Here we show that ovalbumin (OVA)-challenged mice exhibited greater expression of BHLHE40 in lung tissues. Bhlhe40 knockdown reduced the pulmonary lesions and allergy-induced inflammation in asthmatic mice. Moreover, an inhibitory effect of Bhlhe40 knockdown on alternative activation was observed in vivo and in vitro. We show a downstream target Neurturin (Nrtn) of Bhlhe40. Dual luciferase assay and ChIP-qPCR assay indicated that BHLHE40 bound to Nrtn promoter and reduced its transcriptional activity. Simultaneous knockdown of Bhlhe40 and Nrtn recovered the alternative activation of macrophages and rescued the OVA-elicited asthma phenotype. NRTN downregulation offset the alleviative effects of Bhlhe40 knockdown on asthma. This study demonstrates that BHLHE40 promotes allergic asthma, and contributes to the alternative activation of macrophages in asthma by inhibiting Nrtn transcription.https://doi.org/10.1038/s42003-025-08288-1
spellingShingle Bing Dai
Li Chen
Xiaowen Li
Qianlan Zhou
Qinzhen Zhang
Lina Han
Wenxin Shen
Qing Chang
Yuhong Zhao
Si Liu
Lishen Shan
BHLHE40 contributes to allergic asthma progression in mice through NRTN downregulation in macrophages
Communications Biology
title BHLHE40 contributes to allergic asthma progression in mice through NRTN downregulation in macrophages
title_full BHLHE40 contributes to allergic asthma progression in mice through NRTN downregulation in macrophages
title_fullStr BHLHE40 contributes to allergic asthma progression in mice through NRTN downregulation in macrophages
title_full_unstemmed BHLHE40 contributes to allergic asthma progression in mice through NRTN downregulation in macrophages
title_short BHLHE40 contributes to allergic asthma progression in mice through NRTN downregulation in macrophages
title_sort bhlhe40 contributes to allergic asthma progression in mice through nrtn downregulation in macrophages
url https://doi.org/10.1038/s42003-025-08288-1
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