TRIM47 promotes head and neck squamous cell carcinoma malignant progression by degrading XAF1 through ubiquitination

Summary: Tripartite motif-containing 47 (TRIM47) is a member of the TRIM family, which has E3 ligase activity and has been demonstrated to be involved in tumor development. In this work, we found that TRIM47 is highly expressed in head and neck squamous cell carcinomas (HNSCC) tissues. TRIM47 overex...

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Main Authors: Changyun Yu, Chen Zhang, Qianqian Zhang, Cai Zhang, Jingjie Han, Jinying Li
Format: Article
Language:English
Published: Elsevier 2025-01-01
Series:iScience
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Online Access:http://www.sciencedirect.com/science/article/pii/S2589004224028177
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author Changyun Yu
Chen Zhang
Qianqian Zhang
Cai Zhang
Jingjie Han
Jinying Li
author_facet Changyun Yu
Chen Zhang
Qianqian Zhang
Cai Zhang
Jingjie Han
Jinying Li
author_sort Changyun Yu
collection DOAJ
description Summary: Tripartite motif-containing 47 (TRIM47) is a member of the TRIM family, which has E3 ligase activity and has been demonstrated to be involved in tumor development. In this work, we found that TRIM47 is highly expressed in head and neck squamous cell carcinomas (HNSCC) tissues. TRIM47 overexpression promoted HNSCC cell proliferation. Downregulation of TRIM47 suppressed HNSCC cell proliferation and promoted apoptosis and autophagy. TRIM47 suppression caused cell proliferation inhibition and apoptosis promotion could be reversed by 3-MA, an autophagy inhibitor. In mechanism, TRIM47 interacted with XIAP-associated factor 1 (XAF1), promoting its ubiquitination and degradation. XAF1 promoted HNSCC cell apoptosis and autophagy. TRIM47 overexpression caused cell proliferation promotion and autophagy inhibition could be reversed by XAF1 overexpression. Animal experiments confirmed that the knockdown of TRIM47 inhibits tumor growth in vivo. Ultimately, TRIM47 promotes the ubiquitination and degradation of XAF1 and suppresses apoptosis and autophagy to promote the progression of HNSCC.
format Article
id doaj-art-0e958432b3f046a5a6a2d6adfc7b41fb
institution DOAJ
issn 2589-0042
language English
publishDate 2025-01-01
publisher Elsevier
record_format Article
series iScience
spelling doaj-art-0e958432b3f046a5a6a2d6adfc7b41fb2025-08-20T02:39:48ZengElsevieriScience2589-00422025-01-0128111159010.1016/j.isci.2024.111590TRIM47 promotes head and neck squamous cell carcinoma malignant progression by degrading XAF1 through ubiquitinationChangyun Yu0Chen Zhang1Qianqian Zhang2Cai Zhang3Jingjie Han4Jinying Li5Department of Otolaryngology Head and Neck Surgery, The First Affiliated Hospital of Zhengzhou University, Zhengzhou 450052, China; Corresponding authorDepartment of Otolaryngology Head and Neck Surgery, The First Affiliated Hospital of Zhengzhou University, Zhengzhou 450052, ChinaDepartment of Otolaryngology Head and Neck Surgery, The First Affiliated Hospital of Zhengzhou University, Zhengzhou 450052, ChinaDepartment of Otolaryngology Head and Neck Surgery, The First Affiliated Hospital of Zhengzhou University, Zhengzhou 450052, ChinaDepartment of Otolaryngology Head and Neck Surgery, The First Affiliated Hospital of Zhengzhou University, Zhengzhou 450052, ChinaDepartment of Otolaryngology Head and Neck Surgery, The First Affiliated Hospital of Zhengzhou University, Zhengzhou 450052, China; Department of Otolaryngology Head and Neck Surgery, Children’s Hospital Affiliated to Zhengzhou University, Zhengzhou 450018, ChinaSummary: Tripartite motif-containing 47 (TRIM47) is a member of the TRIM family, which has E3 ligase activity and has been demonstrated to be involved in tumor development. In this work, we found that TRIM47 is highly expressed in head and neck squamous cell carcinomas (HNSCC) tissues. TRIM47 overexpression promoted HNSCC cell proliferation. Downregulation of TRIM47 suppressed HNSCC cell proliferation and promoted apoptosis and autophagy. TRIM47 suppression caused cell proliferation inhibition and apoptosis promotion could be reversed by 3-MA, an autophagy inhibitor. In mechanism, TRIM47 interacted with XIAP-associated factor 1 (XAF1), promoting its ubiquitination and degradation. XAF1 promoted HNSCC cell apoptosis and autophagy. TRIM47 overexpression caused cell proliferation promotion and autophagy inhibition could be reversed by XAF1 overexpression. Animal experiments confirmed that the knockdown of TRIM47 inhibits tumor growth in vivo. Ultimately, TRIM47 promotes the ubiquitination and degradation of XAF1 and suppresses apoptosis and autophagy to promote the progression of HNSCC.http://www.sciencedirect.com/science/article/pii/S2589004224028177Molecular biologyCell biologyCancer
spellingShingle Changyun Yu
Chen Zhang
Qianqian Zhang
Cai Zhang
Jingjie Han
Jinying Li
TRIM47 promotes head and neck squamous cell carcinoma malignant progression by degrading XAF1 through ubiquitination
iScience
Molecular biology
Cell biology
Cancer
title TRIM47 promotes head and neck squamous cell carcinoma malignant progression by degrading XAF1 through ubiquitination
title_full TRIM47 promotes head and neck squamous cell carcinoma malignant progression by degrading XAF1 through ubiquitination
title_fullStr TRIM47 promotes head and neck squamous cell carcinoma malignant progression by degrading XAF1 through ubiquitination
title_full_unstemmed TRIM47 promotes head and neck squamous cell carcinoma malignant progression by degrading XAF1 through ubiquitination
title_short TRIM47 promotes head and neck squamous cell carcinoma malignant progression by degrading XAF1 through ubiquitination
title_sort trim47 promotes head and neck squamous cell carcinoma malignant progression by degrading xaf1 through ubiquitination
topic Molecular biology
Cell biology
Cancer
url http://www.sciencedirect.com/science/article/pii/S2589004224028177
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