REST contributes to renal fibrosis through inducing mitochondrial energy metabolism imbalance in tubular epithelial cells

REST contributes to renal fibrosis through inducing mitochondrial energy metabolism imbalance in tubular epithelial cells

Abstract Background Renal fibrosis represents the final common pathological manifestation of chronic kidney disease (CKD), yet the underlying mechanism remains elusive, and there is still a lack of effective targeted therapeutic strategy. Although previous research indicated that repressor element 1...

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Main Authors: Yingxian Yang, Shuiqin Gong, Chun Zhou, Wang Xin, Shaozong Qin, Mengying Yao, Qigang Lan, Wenhao Liao, Jinghong Zhao, Yinghui Huang
Format: Article
Language:English
Published: BMC 2025-04-01
Series:Cell Communication and Signaling
Subjects:
REST
Renal fibrosis
Fatty acid oxidation
OGDHL
Online Access:https://doi.org/10.1186/s12964-025-02166-3
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https://doi.org/10.1186/s12964-025-02166-3

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