Discovery of Specific Inhibitors for Intestinal E. coli  β-Glucuronidase through In Silico Virtual Screening

Glucuronidation is a major metabolism process of detoxification for carcinogens, 4-(methylnitrosamino)-1-(3-pyridy)-1-butanone (NNK) and 1,2-dimethylhydrazine (DMH), of reactive oxygen species (ROS). However, intestinal E. coli   β-glucuronidase (eβG) has been considered pivotal to colorectal carcin...

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Main Authors: Ta-Chun Cheng, Kuo-Hsiang Chuang, Steve R. Roffler, Kai-Wen Cheng, Yu-Lin Leu, Chih-Hung Chuang, Chien-Chaio Huang, Chien-Han Kao, Yuan-Chin Hsieh, Long-Sen Chang, Tian-Lu Cheng, Chien-Shu Chen
Format: Article
Language:English
Published: Wiley 2015-01-01
Series:The Scientific World Journal
Online Access:http://dx.doi.org/10.1155/2015/740815
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author Ta-Chun Cheng
Kuo-Hsiang Chuang
Steve R. Roffler
Kai-Wen Cheng
Yu-Lin Leu
Chih-Hung Chuang
Chien-Chaio Huang
Chien-Han Kao
Yuan-Chin Hsieh
Long-Sen Chang
Tian-Lu Cheng
Chien-Shu Chen
author_facet Ta-Chun Cheng
Kuo-Hsiang Chuang
Steve R. Roffler
Kai-Wen Cheng
Yu-Lin Leu
Chih-Hung Chuang
Chien-Chaio Huang
Chien-Han Kao
Yuan-Chin Hsieh
Long-Sen Chang
Tian-Lu Cheng
Chien-Shu Chen
author_sort Ta-Chun Cheng
collection DOAJ
description Glucuronidation is a major metabolism process of detoxification for carcinogens, 4-(methylnitrosamino)-1-(3-pyridy)-1-butanone (NNK) and 1,2-dimethylhydrazine (DMH), of reactive oxygen species (ROS). However, intestinal E. coli   β-glucuronidase (eβG) has been considered pivotal to colorectal carcinogenesis. Specific inhibition of eβG may prevent reactivating the glucuronide-carcinogen and protect the intestine from ROS-mediated carcinogenesis. In order to develop specific eβG inhibitors, we found that 59 candidate compounds obtained from the initial virtual screening had high inhibition specificity against eβG but not human βG. In particular, we found that compounds 7145 and 4041 with naphthalenylidene-benzenesulfonamide (NYBS) are highly effective and selective to inhibit eβG activity. Compound 4041  (IC50=2.8 μM) shows a higher inhibiting ability than compound 7145  (IC50=31.6 μM) against eβG. Furthermore, the molecular docking analysis indicates that compound 4041 has two hydrophobic contacts to residues L361 and I363 in the bacterial loop, but 7145 has one contact to L361. Only compound 4041 can bind to key residue (E413) at active site of eβG via hydrogen-bonding interactions. These novel NYBS-based eβG specific inhibitors may provide as novel candidate compounds, which specifically inhibit eβG to reduce eβG-based carcinogenesis and intestinal injury.
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spelling doaj-art-0a2b3e96bd444eb69a82c2dac885205c2025-08-20T03:55:27ZengWileyThe Scientific World Journal2356-61401537-744X2015-01-01201510.1155/2015/740815740815Discovery of Specific Inhibitors for Intestinal E. coli  β-Glucuronidase through In Silico Virtual ScreeningTa-Chun Cheng0Kuo-Hsiang Chuang1Steve R. Roffler2Kai-Wen Cheng3Yu-Lin Leu4Chih-Hung Chuang5Chien-Chaio Huang6Chien-Han Kao7Yuan-Chin Hsieh8Long-Sen Chang9Tian-Lu Cheng10Chien-Shu Chen11Graduate Institute of Pharmacognosy, Taipei Medical University, 252 Wu Hsing Street, Taipei 11031, TaiwanGraduate Institute of Pharmacognosy, Taipei Medical University, 252 Wu Hsing Street, Taipei 11031, TaiwanInstitute of Biomedical Sciences, Academia Sinica, 128 Section 2, Academia Road, Nankang, Taipei 11529, TaiwanInstitute of Biomedical Science, 70 Lienhai Road, Kaohsiung 80424, National Sun Yat-Sen University, TaiwanDepartment of Pharmacy, Chia Nan University of Pharmacy and Science, 60 Section 1, Erh-Ren Road, Tainan 71710, TaiwanDepartment of Biomedical and Environmental Biology, Kaohsiung Medical University, 100 Shih-Chuan 1st Road, Kaohsiung 80708, TaiwanDepartment of Biomedical and Environmental Biology, Kaohsiung Medical University, 100 Shih-Chuan 1st Road, Kaohsiung 80708, TaiwanGraduate Institute of Medicine, Kaohsiung Medical University, 100 Shih-Chuan 1st Road, Kaohsiung 80708, TaiwanGraduate Institute of Medicine, Kaohsiung Medical University, 100 Shih-Chuan 1st Road, Kaohsiung 80708, TaiwanInstitute of Biomedical Science, 70 Lienhai Road, Kaohsiung 80424, National Sun Yat-Sen University, TaiwanDepartment of Biomedical and Environmental Biology, Kaohsiung Medical University, 100 Shih-Chuan 1st Road, Kaohsiung 80708, TaiwanSchool of Pharmacy, China Medical University, 91 Hsueh-Shih Road, Taichung 40402, TaiwanGlucuronidation is a major metabolism process of detoxification for carcinogens, 4-(methylnitrosamino)-1-(3-pyridy)-1-butanone (NNK) and 1,2-dimethylhydrazine (DMH), of reactive oxygen species (ROS). However, intestinal E. coli   β-glucuronidase (eβG) has been considered pivotal to colorectal carcinogenesis. Specific inhibition of eβG may prevent reactivating the glucuronide-carcinogen and protect the intestine from ROS-mediated carcinogenesis. In order to develop specific eβG inhibitors, we found that 59 candidate compounds obtained from the initial virtual screening had high inhibition specificity against eβG but not human βG. In particular, we found that compounds 7145 and 4041 with naphthalenylidene-benzenesulfonamide (NYBS) are highly effective and selective to inhibit eβG activity. Compound 4041  (IC50=2.8 μM) shows a higher inhibiting ability than compound 7145  (IC50=31.6 μM) against eβG. Furthermore, the molecular docking analysis indicates that compound 4041 has two hydrophobic contacts to residues L361 and I363 in the bacterial loop, but 7145 has one contact to L361. Only compound 4041 can bind to key residue (E413) at active site of eβG via hydrogen-bonding interactions. These novel NYBS-based eβG specific inhibitors may provide as novel candidate compounds, which specifically inhibit eβG to reduce eβG-based carcinogenesis and intestinal injury.http://dx.doi.org/10.1155/2015/740815
spellingShingle Ta-Chun Cheng
Kuo-Hsiang Chuang
Steve R. Roffler
Kai-Wen Cheng
Yu-Lin Leu
Chih-Hung Chuang
Chien-Chaio Huang
Chien-Han Kao
Yuan-Chin Hsieh
Long-Sen Chang
Tian-Lu Cheng
Chien-Shu Chen
Discovery of Specific Inhibitors for Intestinal E. coli  β-Glucuronidase through In Silico Virtual Screening
The Scientific World Journal
title Discovery of Specific Inhibitors for Intestinal E. coli  β-Glucuronidase through In Silico Virtual Screening
title_full Discovery of Specific Inhibitors for Intestinal E. coli  β-Glucuronidase through In Silico Virtual Screening
title_fullStr Discovery of Specific Inhibitors for Intestinal E. coli  β-Glucuronidase through In Silico Virtual Screening
title_full_unstemmed Discovery of Specific Inhibitors for Intestinal E. coli  β-Glucuronidase through In Silico Virtual Screening
title_short Discovery of Specific Inhibitors for Intestinal E. coli  β-Glucuronidase through In Silico Virtual Screening
title_sort discovery of specific inhibitors for intestinal e coli β glucuronidase through in silico virtual screening
url http://dx.doi.org/10.1155/2015/740815
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