Identification of MEG3 and MAPK3 as potential therapeutic targets for osteoarthritis through multiomics integration and machine learning

Abstract Knee osteoarthritis (KOA) is a prevalent degenerative joint disorder, yet its underlying molecular mechanisms remain puzzling. This study aimed to uncover the genes with a causal relationship to KOA using Mendelian randomization (MR), transcriptomic profiling, and machine learning methods....

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Main Authors: Bing Ma, Xiaoru Wang, Chengfei Xu, Zelin Xu, Fei Zhang, Wendan Cheng
Format: Article
Language:English
Published: Nature Portfolio 2025-07-01
Series:Scientific Reports
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Online Access:https://doi.org/10.1038/s41598-025-06175-7
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author Bing Ma
Xiaoru Wang
Chengfei Xu
Zelin Xu
Fei Zhang
Wendan Cheng
author_facet Bing Ma
Xiaoru Wang
Chengfei Xu
Zelin Xu
Fei Zhang
Wendan Cheng
author_sort Bing Ma
collection DOAJ
description Abstract Knee osteoarthritis (KOA) is a prevalent degenerative joint disorder, yet its underlying molecular mechanisms remain puzzling. This study aimed to uncover the genes with a causal relationship to KOA using Mendelian randomization (MR), transcriptomic profiling, and machine learning methods. MR analysis was conducted utilizing expression quantitative trait loci (eQTL) data from the eQTLGen consortium alongside KOA-related GWAS summary statistics to identify candidate genes. Subsequently, differential expression analysis and WGCNA were applied to synovial tissue microarray datasets obtained from the GEO database. The intersecting genes were further refined using three machine learning algorithms: LASSO, random forest, and SVM–RFE. Diagnostic efficacy was assessed via ROC curve analysis and nomogram construction. Validation was ultimately performed using qPCR on clinical synovial tissue samples. Twelve genes with putative causal associations to KOA were identified, with MEG3 and MAPK3 emerging as the most diagnostically robust. Both exhibited high sensitivity and specificity in ROC analysis, and their differential expression was corroborated by qPCR. This study underscores the diagnostic utility of MEG3 and MAPK3 in KOA and offers a promising molecular framework for early disease detection. Nonetheless, validation in larger, independent cohorts and further mechanistic investigations are warranted to substantiate these findings.
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spelling doaj-art-033f336cf62040a6ae5db954930c16822025-08-20T03:38:12ZengNature PortfolioScientific Reports2045-23222025-07-0115111410.1038/s41598-025-06175-7Identification of MEG3 and MAPK3 as potential therapeutic targets for osteoarthritis through multiomics integration and machine learningBing Ma0Xiaoru Wang1Chengfei Xu2Zelin Xu3Fei Zhang4Wendan Cheng5Department of Orthopaedics, The Second Affiliated Hospital of Anhui Medical UniversityBengbu Third People’s Hospital attached to Bengbu Medical UniversityBengbu Third People’s Hospital attached to Bengbu Medical UniversityDepartment of Orthopaedics, The Second Affiliated Hospital of Anhui Medical UniversityDepartment of Orthopaedics, The Second Affiliated Hospital of Anhui Medical UniversityDepartment of Orthopaedics, The Second Affiliated Hospital of Anhui Medical UniversityAbstract Knee osteoarthritis (KOA) is a prevalent degenerative joint disorder, yet its underlying molecular mechanisms remain puzzling. This study aimed to uncover the genes with a causal relationship to KOA using Mendelian randomization (MR), transcriptomic profiling, and machine learning methods. MR analysis was conducted utilizing expression quantitative trait loci (eQTL) data from the eQTLGen consortium alongside KOA-related GWAS summary statistics to identify candidate genes. Subsequently, differential expression analysis and WGCNA were applied to synovial tissue microarray datasets obtained from the GEO database. The intersecting genes were further refined using three machine learning algorithms: LASSO, random forest, and SVM–RFE. Diagnostic efficacy was assessed via ROC curve analysis and nomogram construction. Validation was ultimately performed using qPCR on clinical synovial tissue samples. Twelve genes with putative causal associations to KOA were identified, with MEG3 and MAPK3 emerging as the most diagnostically robust. Both exhibited high sensitivity and specificity in ROC analysis, and their differential expression was corroborated by qPCR. This study underscores the diagnostic utility of MEG3 and MAPK3 in KOA and offers a promising molecular framework for early disease detection. Nonetheless, validation in larger, independent cohorts and further mechanistic investigations are warranted to substantiate these findings.https://doi.org/10.1038/s41598-025-06175-7Knee osteoarthritisMEG3MAPK3Mendelian randomizationEQTL
spellingShingle Bing Ma
Xiaoru Wang
Chengfei Xu
Zelin Xu
Fei Zhang
Wendan Cheng
Identification of MEG3 and MAPK3 as potential therapeutic targets for osteoarthritis through multiomics integration and machine learning
Scientific Reports
Knee osteoarthritis
MEG3
MAPK3
Mendelian randomization
EQTL
title Identification of MEG3 and MAPK3 as potential therapeutic targets for osteoarthritis through multiomics integration and machine learning
title_full Identification of MEG3 and MAPK3 as potential therapeutic targets for osteoarthritis through multiomics integration and machine learning
title_fullStr Identification of MEG3 and MAPK3 as potential therapeutic targets for osteoarthritis through multiomics integration and machine learning
title_full_unstemmed Identification of MEG3 and MAPK3 as potential therapeutic targets for osteoarthritis through multiomics integration and machine learning
title_short Identification of MEG3 and MAPK3 as potential therapeutic targets for osteoarthritis through multiomics integration and machine learning
title_sort identification of meg3 and mapk3 as potential therapeutic targets for osteoarthritis through multiomics integration and machine learning
topic Knee osteoarthritis
MEG3
MAPK3
Mendelian randomization
EQTL
url https://doi.org/10.1038/s41598-025-06175-7
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