Mapping essential somatic hypermutations in a CD4-binding site bNAb informs HIV-1 vaccine design

Summary: HIV-1 broadly neutralizing antibodies (bNAbs) targeting the CD4-binding site (CD4bs) contain rare features that pose challenges to elicit these bNAbs through vaccination. The IOMA class of CD4bs bNAbs includes fewer rare features and somatic hypermutations (SHMs) to achieve broad neutraliza...

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Main Authors: Kim-Marie A. Dam, Harry B. Gristick, Yancheng E. Li, Zhi Yang, Priyanthi N.P. Gnanapragasam, Anthony P. West, Jr., Michael S. Seaman, Pamela J. Bjorkman
Format: Article
Language:English
Published: Elsevier 2025-05-01
Series:Cell Reports
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Online Access:http://www.sciencedirect.com/science/article/pii/S221112472500484X
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author Kim-Marie A. Dam
Harry B. Gristick
Yancheng E. Li
Zhi Yang
Priyanthi N.P. Gnanapragasam
Anthony P. West, Jr.
Michael S. Seaman
Pamela J. Bjorkman
author_facet Kim-Marie A. Dam
Harry B. Gristick
Yancheng E. Li
Zhi Yang
Priyanthi N.P. Gnanapragasam
Anthony P. West, Jr.
Michael S. Seaman
Pamela J. Bjorkman
author_sort Kim-Marie A. Dam
collection DOAJ
description Summary: HIV-1 broadly neutralizing antibodies (bNAbs) targeting the CD4-binding site (CD4bs) contain rare features that pose challenges to elicit these bNAbs through vaccination. The IOMA class of CD4bs bNAbs includes fewer rare features and somatic hypermutations (SHMs) to achieve broad neutralization, thus presenting a potentially accessible pathway for vaccine-induced bNAb development. Here, we created a library of IOMA variants in which each SHM was individually reverted to the inferred germline counterpart to investigate the roles of SHMs in conferring IOMA’s neutralization potency and breadth. Impacts on neutralization for each variant were evaluated, and this information was used to design minimally mutated IOMA-class variants (IOMAmin) that incorporated the fewest SHMs required for achieving IOMA’s neutralization breadth. A cryoelectron microscopy (cryo-EM) structure of an IOMAmin variant bound to Env was used to further interpret characteristics of IOMA variants to elucidate how IOMA’s structural features correlate with its neutralization mechanism, informing the design of IOMA-targeting immunogens.
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spelling doaj-art-001d5eebf4fc4d748c6d03a1fb01a52f2025-08-20T03:09:43ZengElsevierCell Reports2211-12472025-05-0144511571310.1016/j.celrep.2025.115713Mapping essential somatic hypermutations in a CD4-binding site bNAb informs HIV-1 vaccine designKim-Marie A. Dam0Harry B. Gristick1Yancheng E. Li2Zhi Yang3Priyanthi N.P. Gnanapragasam4Anthony P. West, Jr.5Michael S. Seaman6Pamela J. Bjorkman7Division of Biology and Biological Engineering, California Institute of Technology, Pasadena, CA 91125, USADivision of Biology and Biological Engineering, California Institute of Technology, Pasadena, CA 91125, USADivision of Biology and Biological Engineering, California Institute of Technology, Pasadena, CA 91125, USA; Division of Chemistry and Chemical Engineering, California Institute of Technology, Pasadena, CA 91125, USADivision of Biology and Biological Engineering, California Institute of Technology, Pasadena, CA 91125, USADivision of Biology and Biological Engineering, California Institute of Technology, Pasadena, CA 91125, USADivision of Biology and Biological Engineering, California Institute of Technology, Pasadena, CA 91125, USACenter for Virology and Vaccine Research, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, MA 02215, USADivision of Biology and Biological Engineering, California Institute of Technology, Pasadena, CA 91125, USA; Corresponding authorSummary: HIV-1 broadly neutralizing antibodies (bNAbs) targeting the CD4-binding site (CD4bs) contain rare features that pose challenges to elicit these bNAbs through vaccination. The IOMA class of CD4bs bNAbs includes fewer rare features and somatic hypermutations (SHMs) to achieve broad neutralization, thus presenting a potentially accessible pathway for vaccine-induced bNAb development. Here, we created a library of IOMA variants in which each SHM was individually reverted to the inferred germline counterpart to investigate the roles of SHMs in conferring IOMA’s neutralization potency and breadth. Impacts on neutralization for each variant were evaluated, and this information was used to design minimally mutated IOMA-class variants (IOMAmin) that incorporated the fewest SHMs required for achieving IOMA’s neutralization breadth. A cryoelectron microscopy (cryo-EM) structure of an IOMAmin variant bound to Env was used to further interpret characteristics of IOMA variants to elucidate how IOMA’s structural features correlate with its neutralization mechanism, informing the design of IOMA-targeting immunogens.http://www.sciencedirect.com/science/article/pii/S221112472500484XCP: ImmunologyCP: Microbiology
spellingShingle Kim-Marie A. Dam
Harry B. Gristick
Yancheng E. Li
Zhi Yang
Priyanthi N.P. Gnanapragasam
Anthony P. West, Jr.
Michael S. Seaman
Pamela J. Bjorkman
Mapping essential somatic hypermutations in a CD4-binding site bNAb informs HIV-1 vaccine design
Cell Reports
CP: Immunology
CP: Microbiology
title Mapping essential somatic hypermutations in a CD4-binding site bNAb informs HIV-1 vaccine design
title_full Mapping essential somatic hypermutations in a CD4-binding site bNAb informs HIV-1 vaccine design
title_fullStr Mapping essential somatic hypermutations in a CD4-binding site bNAb informs HIV-1 vaccine design
title_full_unstemmed Mapping essential somatic hypermutations in a CD4-binding site bNAb informs HIV-1 vaccine design
title_short Mapping essential somatic hypermutations in a CD4-binding site bNAb informs HIV-1 vaccine design
title_sort mapping essential somatic hypermutations in a cd4 binding site bnab informs hiv 1 vaccine design
topic CP: Immunology
CP: Microbiology
url http://www.sciencedirect.com/science/article/pii/S221112472500484X
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