Showing 1 - 5 results of 5 for search 'pathogen-most interaction', query time: 0.09s Refine Results
  1. 1

    Stalks and roots are the main battlefield for the coevolution between maize and Fusarium verticillioides by Hao Xiong, Xiaobin Xing, Muyuan Liu, Zhaoyu Zhang, Qingjun Wang, Xuemei Zhang, Xiangjian Gou, Yanli Lu, Yanli Lu, Xuanjun Feng, Xuanjun Feng

    Published 2024-10-01
    “…Theoretically, the organ where the host interacts with the pathogen most frequently should exhibit the highest degree of symptom-genotype correlation. …”
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  2. 2

    Manipulating adenovirus hexon hypervariable loops dictates immune neutralisation and coagulation factor X-dependent cell interaction in vitro and in vivo. by Jiangtao Ma, Margaret R Duffy, Lin Deng, Rachel S Dakin, Taco Uil, Jerome Custers, Sharon M Kelly, John H McVey, Stuart A Nicklin, Andrew H Baker

    Published 2015-02-01
    “…Adenoviruses are common pathogens, mostly targeting ocular, gastrointestinal and respiratory cells, but in some cases infection disseminates, presenting in severe clinical outcomes. …”
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  3. 3

    New advances in the science and use of Trichoderma spp. by Gary Harman

    Published 2004-07-01
    “…The increase in plant growth and yields is strongly interactive with plant genotype, at least in maize. …”
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  4. 4

    Breaking the bad: Bacillus blocks fungal virulence factors by François L. Mayer, James W. Kronstad

    Published 2017-10-01
    “…Inhibiting such factors generally results in reduced fungal pathogenicity. Most studies in the past have focused on understanding the molecular mechanisms of fungal virulence factor expression during mono-culture, or during interaction with the host. …”
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  5. 5

    Characterization of temporal expression of immune genes in female locust challenged by fungal pathogen, Aspergillus sp. by Muhammad Saad Waqas, Xiao Xu, Pengfei Zhang, Jin Guo, Shaojing Hu, Yinwei You, Long Zhang

    Published 2025-04-01
    “…This means that fat body cells, but not hemocytes, would be able to precisely recognize invading pathogens. Most serine protease inhibitors (SERPINs) genes and clip domain serine proteinase (CLIP) genes were up-regulated in fat body cells. …”
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