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Stalks and roots are the main battlefield for the coevolution between maize and Fusarium verticillioides
Published 2024-10-01“…Theoretically, the organ where the host interacts with the pathogen most frequently should exhibit the highest degree of symptom-genotype correlation. …”
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Manipulating adenovirus hexon hypervariable loops dictates immune neutralisation and coagulation factor X-dependent cell interaction in vitro and in vivo.
Published 2015-02-01“…Adenoviruses are common pathogens, mostly targeting ocular, gastrointestinal and respiratory cells, but in some cases infection disseminates, presenting in severe clinical outcomes. …”
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New advances in the science and use of Trichoderma spp.
Published 2004-07-01“…The increase in plant growth and yields is strongly interactive with plant genotype, at least in maize. …”
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Breaking the bad: Bacillus blocks fungal virulence factors
Published 2017-10-01“…Inhibiting such factors generally results in reduced fungal pathogenicity. Most studies in the past have focused on understanding the molecular mechanisms of fungal virulence factor expression during mono-culture, or during interaction with the host. …”
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Analysis of epidemic trend of respiratory pathogens in children after long-term pathogen isolation
Published 2025-07-01“…The ability of children under these conditions to resist respiratory pathogens post-lifting of containment measures remained unknown. …”
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The Role and Pathogenesis of Tau Protein in Alzheimer’s Disease
Published 2025-06-01“…Tau, a microtubule-associated protein predominantly localized to neuronal axons, orchestrates microtubule stabilization and axonal transport through dynamic tubulin interactions under physiological conditions. In AD pathogenesis, however, tau undergoes pathogenic post-translational modifications (PTMs), encompassing hyperphosphorylation, lysine acetylation, methylation, ubiquitination, and glycosylation. …”
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Characterization of temporal expression of immune genes in female locust challenged by fungal pathogen, Aspergillus sp.
Published 2025-04-01“…This means that fat body cells, but not hemocytes, would be able to precisely recognize invading pathogens. Most serine protease inhibitors (SERPINs) genes and clip domain serine proteinase (CLIP) genes were up-regulated in fat body cells. …”
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