Interleukin-6 Contributes to Age-Related Alteration of Cytokine Production by Macrophages

Here, we studied in vitro cytokine production by splenic macrophages obtained from young and aged BALB/c wild type (WT) and IL-6 knockout (IL-6 KO) mice. Relative to macrophages obtained from young WT mice given lipopolysaccharide (LPS), those from aged WT mice had decreased production of proinflam...

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Main Authors: Christian R. Gomez, John Karavitis, Jessica L. Palmer, Douglas E. Faunce, Luis Ramirez, Vanessa Nomellini, Elizabeth J. Kovacs
Format: Article
Language:English
Published: Wiley 2010-01-01
Series:Mediators of Inflammation
Online Access:http://dx.doi.org/10.1155/2010/475139
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author Christian R. Gomez
John Karavitis
Jessica L. Palmer
Douglas E. Faunce
Luis Ramirez
Vanessa Nomellini
Elizabeth J. Kovacs
author_facet Christian R. Gomez
John Karavitis
Jessica L. Palmer
Douglas E. Faunce
Luis Ramirez
Vanessa Nomellini
Elizabeth J. Kovacs
author_sort Christian R. Gomez
collection DOAJ
description Here, we studied in vitro cytokine production by splenic macrophages obtained from young and aged BALB/c wild type (WT) and IL-6 knockout (IL-6 KO) mice. Relative to macrophages obtained from young WT mice given lipopolysaccharide (LPS), those from aged WT mice had decreased production of proinflammatory cytokines. In contrast, when compared to macrophages from young IL-6 KO mice, LPS stimulation yielded higher levels of these cytokines by cells from aged IL-6 KO mice. Aging or IL-6 deficiency did not affected the percentage of F4/80+ macrophages, or the surface expression of Toll-like receptor 4 (TLR4) and components of the IL-6 receptor. Overall, our results indicate that IL-6 plays a role in regulating the age-related defects in macrophages through alteration of proinflammatory cytokines, adding to the complexity of IL-6-mediated impairment of immune cell function with increasing age.
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spelling doaj-art-ffd3e5ae7cea46efafafc23cd01124402025-08-20T03:25:54ZengWileyMediators of Inflammation0962-93511466-18612010-01-01201010.1155/2010/475139475139Interleukin-6 Contributes to Age-Related Alteration of Cytokine Production by MacrophagesChristian R. Gomez0John Karavitis1Jessica L. Palmer2Douglas E. Faunce3Luis Ramirez4Vanessa Nomellini5Elizabeth J. Kovacs6Department of Surgery, and Immunology and Aging Program, The Burn and Shock Trauma Institute, Loyola University Medical Center, 2160 South First Avenue, Maywood, IL 60153, USADepartment of Surgery, and Immunology and Aging Program, The Burn and Shock Trauma Institute, Loyola University Medical Center, 2160 South First Avenue, Maywood, IL 60153, USADepartment of Surgery, and Immunology and Aging Program, The Burn and Shock Trauma Institute, Loyola University Medical Center, 2160 South First Avenue, Maywood, IL 60153, USADepartment of Surgery, and Immunology and Aging Program, The Burn and Shock Trauma Institute, Loyola University Medical Center, 2160 South First Avenue, Maywood, IL 60153, USADepartment of Surgery, and Immunology and Aging Program, The Burn and Shock Trauma Institute, Loyola University Medical Center, 2160 South First Avenue, Maywood, IL 60153, USADepartment of Surgery, and Immunology and Aging Program, The Burn and Shock Trauma Institute, Loyola University Medical Center, 2160 South First Avenue, Maywood, IL 60153, USADepartment of Surgery, and Immunology and Aging Program, The Burn and Shock Trauma Institute, Loyola University Medical Center, 2160 South First Avenue, Maywood, IL 60153, USAHere, we studied in vitro cytokine production by splenic macrophages obtained from young and aged BALB/c wild type (WT) and IL-6 knockout (IL-6 KO) mice. Relative to macrophages obtained from young WT mice given lipopolysaccharide (LPS), those from aged WT mice had decreased production of proinflammatory cytokines. In contrast, when compared to macrophages from young IL-6 KO mice, LPS stimulation yielded higher levels of these cytokines by cells from aged IL-6 KO mice. Aging or IL-6 deficiency did not affected the percentage of F4/80+ macrophages, or the surface expression of Toll-like receptor 4 (TLR4) and components of the IL-6 receptor. Overall, our results indicate that IL-6 plays a role in regulating the age-related defects in macrophages through alteration of proinflammatory cytokines, adding to the complexity of IL-6-mediated impairment of immune cell function with increasing age.http://dx.doi.org/10.1155/2010/475139
spellingShingle Christian R. Gomez
John Karavitis
Jessica L. Palmer
Douglas E. Faunce
Luis Ramirez
Vanessa Nomellini
Elizabeth J. Kovacs
Interleukin-6 Contributes to Age-Related Alteration of Cytokine Production by Macrophages
Mediators of Inflammation
title Interleukin-6 Contributes to Age-Related Alteration of Cytokine Production by Macrophages
title_full Interleukin-6 Contributes to Age-Related Alteration of Cytokine Production by Macrophages
title_fullStr Interleukin-6 Contributes to Age-Related Alteration of Cytokine Production by Macrophages
title_full_unstemmed Interleukin-6 Contributes to Age-Related Alteration of Cytokine Production by Macrophages
title_short Interleukin-6 Contributes to Age-Related Alteration of Cytokine Production by Macrophages
title_sort interleukin 6 contributes to age related alteration of cytokine production by macrophages
url http://dx.doi.org/10.1155/2010/475139
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