Antiproliferative Factor-Induced Changes in Phosphorylation and Palmitoylation of Cytoskeleton-Associated Protein-4 Regulate Its Nuclear Translocation and DNA Binding

Cytoskeleton-associated protein 4 (CKAP4) is a reversibly palmitoylated and phosphorylated transmembrane protein that functions as a high-affinity receptor for antiproliferative factor (APF)—a sialoglycopeptide secreted from bladder epithelial cells of patients with interstitial cystitis (IC). Palmi...

Full description

Saved in:
Bibliographic Details
Main Authors: David A. Zacharias, Matthew Mullen, Sonia Lobo Planey
Format: Article
Language:English
Published: Wiley 2012-01-01
Series:International Journal of Cell Biology
Online Access:http://dx.doi.org/10.1155/2012/150918
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1849468148461338624
author David A. Zacharias
Matthew Mullen
Sonia Lobo Planey
author_facet David A. Zacharias
Matthew Mullen
Sonia Lobo Planey
author_sort David A. Zacharias
collection DOAJ
description Cytoskeleton-associated protein 4 (CKAP4) is a reversibly palmitoylated and phosphorylated transmembrane protein that functions as a high-affinity receptor for antiproliferative factor (APF)—a sialoglycopeptide secreted from bladder epithelial cells of patients with interstitial cystitis (IC). Palmitoylation of CKAP4 by the palmitoyl acyltransferase, DHHC2, is required for its cell surface localization and subsequent APF signal transduction; however, the mechanism for APF signal transduction by CKAP4 is unknown. In this paper, we demonstrate that APF treatment induces serine phosphorylation of residues S3, S17, and S19 of CKAP4 and nuclear translocation of CKAP4. Additionally, we demonstrate that CKAP4 binds gDNA in a phosphorylation-dependent manner in response to APF treatment, and that a phosphomimicking, constitutively nonpalmitoylated form of CKAP4 localizes to the nucleus, binds DNA, and mimics the inhibitory effects of APF on cellular proliferation. These results reveal a novel role for CKAP4 as a downstream effecter for APF signal transduction.
format Article
id doaj-art-ffaa679583f84271a2ad6517b2ab23b2
institution Kabale University
issn 1687-8876
1687-8884
language English
publishDate 2012-01-01
publisher Wiley
record_format Article
series International Journal of Cell Biology
spelling doaj-art-ffaa679583f84271a2ad6517b2ab23b22025-08-20T03:25:56ZengWileyInternational Journal of Cell Biology1687-88761687-88842012-01-01201210.1155/2012/150918150918Antiproliferative Factor-Induced Changes in Phosphorylation and Palmitoylation of Cytoskeleton-Associated Protein-4 Regulate Its Nuclear Translocation and DNA BindingDavid A. Zacharias0Matthew Mullen1Sonia Lobo Planey2Whitney Laboratory, Department of Neuroscience, University of Florida, St. Augustine, FL 32080, USADepartment of Basic Sciences, The Commonwealth Medical College, Scranton, PA 18509, USADepartment of Basic Sciences, The Commonwealth Medical College, Scranton, PA 18509, USACytoskeleton-associated protein 4 (CKAP4) is a reversibly palmitoylated and phosphorylated transmembrane protein that functions as a high-affinity receptor for antiproliferative factor (APF)—a sialoglycopeptide secreted from bladder epithelial cells of patients with interstitial cystitis (IC). Palmitoylation of CKAP4 by the palmitoyl acyltransferase, DHHC2, is required for its cell surface localization and subsequent APF signal transduction; however, the mechanism for APF signal transduction by CKAP4 is unknown. In this paper, we demonstrate that APF treatment induces serine phosphorylation of residues S3, S17, and S19 of CKAP4 and nuclear translocation of CKAP4. Additionally, we demonstrate that CKAP4 binds gDNA in a phosphorylation-dependent manner in response to APF treatment, and that a phosphomimicking, constitutively nonpalmitoylated form of CKAP4 localizes to the nucleus, binds DNA, and mimics the inhibitory effects of APF on cellular proliferation. These results reveal a novel role for CKAP4 as a downstream effecter for APF signal transduction.http://dx.doi.org/10.1155/2012/150918
spellingShingle David A. Zacharias
Matthew Mullen
Sonia Lobo Planey
Antiproliferative Factor-Induced Changes in Phosphorylation and Palmitoylation of Cytoskeleton-Associated Protein-4 Regulate Its Nuclear Translocation and DNA Binding
International Journal of Cell Biology
title Antiproliferative Factor-Induced Changes in Phosphorylation and Palmitoylation of Cytoskeleton-Associated Protein-4 Regulate Its Nuclear Translocation and DNA Binding
title_full Antiproliferative Factor-Induced Changes in Phosphorylation and Palmitoylation of Cytoskeleton-Associated Protein-4 Regulate Its Nuclear Translocation and DNA Binding
title_fullStr Antiproliferative Factor-Induced Changes in Phosphorylation and Palmitoylation of Cytoskeleton-Associated Protein-4 Regulate Its Nuclear Translocation and DNA Binding
title_full_unstemmed Antiproliferative Factor-Induced Changes in Phosphorylation and Palmitoylation of Cytoskeleton-Associated Protein-4 Regulate Its Nuclear Translocation and DNA Binding
title_short Antiproliferative Factor-Induced Changes in Phosphorylation and Palmitoylation of Cytoskeleton-Associated Protein-4 Regulate Its Nuclear Translocation and DNA Binding
title_sort antiproliferative factor induced changes in phosphorylation and palmitoylation of cytoskeleton associated protein 4 regulate its nuclear translocation and dna binding
url http://dx.doi.org/10.1155/2012/150918
work_keys_str_mv AT davidazacharias antiproliferativefactorinducedchangesinphosphorylationandpalmitoylationofcytoskeletonassociatedprotein4regulateitsnucleartranslocationanddnabinding
AT matthewmullen antiproliferativefactorinducedchangesinphosphorylationandpalmitoylationofcytoskeletonassociatedprotein4regulateitsnucleartranslocationanddnabinding
AT sonialoboplaney antiproliferativefactorinducedchangesinphosphorylationandpalmitoylationofcytoskeletonassociatedprotein4regulateitsnucleartranslocationanddnabinding