Dopamine-Sensitive Anterior Cingulate Cortical Glucose-Monitoring Neurons as Potential Therapeutic Targets for Gustatory and Other Behavior Alterations

<b>Background</b>: The anterior cingulate cortex (ACC) is known for its involvement in various regulatory functions, including in the central control of feeding. Activation of local elements of the central glucose-monitoring (GM) neuronal network appears to be indispensable in these regu...

Full description

Saved in:
Bibliographic Details
Main Authors: Edina Hormay, Bettina László, István Szabó, Kitti Mintál, Beáta Berta, Tamás Ollmann, László Péczely, Bernadett Nagy, Attila Tóth, Kristóf László, László Lénárd, Zoltán Karádi
Format: Article
Language:English
Published: MDPI AG 2024-12-01
Series:Biomedicines
Subjects:
Online Access:https://www.mdpi.com/2227-9059/12/12/2803
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1850036347843117056
author Edina Hormay
Bettina László
István Szabó
Kitti Mintál
Beáta Berta
Tamás Ollmann
László Péczely
Bernadett Nagy
Attila Tóth
Kristóf László
László Lénárd
Zoltán Karádi
author_facet Edina Hormay
Bettina László
István Szabó
Kitti Mintál
Beáta Berta
Tamás Ollmann
László Péczely
Bernadett Nagy
Attila Tóth
Kristóf László
László Lénárd
Zoltán Karádi
author_sort Edina Hormay
collection DOAJ
description <b>Background</b>: The anterior cingulate cortex (ACC) is known for its involvement in various regulatory functions, including in the central control of feeding. Activation of local elements of the central glucose-monitoring (GM) neuronal network appears to be indispensable in these regulatory processes. Destruction of these type 2 glucose transporter protein (GLUT2)-equipped chemosensory cells results in multiple feeding-associated functional alterations. <b>Methods</b>: In order to examine this complex symptomatology, (1) dopamine sensitivity was studied in laboratory rats by means of the single-neuron-recording multibarreled microelectrophoretic technique, and (2) after local bilateral microinjection of the selective type 2 glucose transporter proteindemolishing streptozotocin (STZ), open-field, elevated plus maze, two-bottle and taste reactivity tests were performed. <b>Results</b>: A high proportion of the anterior cingulate cortical neurons changed their firing rate in response to microelectrophoretic administration of D-glucose, thus verifying them as local elements of the central glucose-monitoring network. Approximately 20% of the recorded cells displayed activity changes in response to microelectrophoretic application of dopamine, and almost 50% of the glucose-monitoring units here proved to be dopamine-sensitive. Moreover, taste stimulation experiments revealed even higher (80%) gustatory sensitivity dominance of these chemosensory cells. The anterior cingulate cortical STZ microinjections resulted in extensive behavioral and taste-associated functional deficits. <b>Conclusions</b>: The present findings provided evidence for the selective loss of glucose-monitoring neurons in the anterior cingulate cortex leading to motivated behavioral and gustatory alterations. This complex dataset also underlines the varied significance of the type 2 glucose transporter protein-equipped, dopamine-sensitive glucose-monitoring neurons as potential therapeutic targets. These units appear to be indispensable in adaptive control mechanisms of the homeostatic–motivational–emotional–cognitive balance for the overall well-being of the organism.
format Article
id doaj-art-ff84cba0e6a5437e887f9726c856dd04
institution DOAJ
issn 2227-9059
language English
publishDate 2024-12-01
publisher MDPI AG
record_format Article
series Biomedicines
spelling doaj-art-ff84cba0e6a5437e887f9726c856dd042025-08-20T02:57:12ZengMDPI AGBiomedicines2227-90592024-12-011212280310.3390/biomedicines12122803Dopamine-Sensitive Anterior Cingulate Cortical Glucose-Monitoring Neurons as Potential Therapeutic Targets for Gustatory and Other Behavior AlterationsEdina Hormay0Bettina László1István Szabó2Kitti Mintál3Beáta Berta4Tamás Ollmann5László Péczely6Bernadett Nagy7Attila Tóth8Kristóf László9László Lénárd10Zoltán Karádi11Institute of Physiology, Medical School, University of Pécs, H-7624 Pécs, HungaryInstitute of Physiology, Medical School, University of Pécs, H-7624 Pécs, HungaryInstitute of Physiology, Medical School, University of Pécs, H-7624 Pécs, HungaryInstitute of Physiology, Medical School, University of Pécs, H-7624 Pécs, HungaryInstitute of Physiology, Medical School, University of Pécs, H-7624 Pécs, HungaryInstitute of Physiology, Medical School, University of Pécs, H-7624 Pécs, HungaryInstitute of Physiology, Medical School, University of Pécs, H-7624 Pécs, HungaryInstitute of Physiology, Medical School, University of Pécs, H-7624 Pécs, HungaryInstitute of Physiology, Medical School, University of Pécs, H-7624 Pécs, HungaryInstitute of Physiology, Medical School, University of Pécs, H-7624 Pécs, HungaryInstitute of Physiology, Medical School, University of Pécs, H-7624 Pécs, HungaryInstitute of Physiology, Medical School, University of Pécs, H-7624 Pécs, Hungary<b>Background</b>: The anterior cingulate cortex (ACC) is known for its involvement in various regulatory functions, including in the central control of feeding. Activation of local elements of the central glucose-monitoring (GM) neuronal network appears to be indispensable in these regulatory processes. Destruction of these type 2 glucose transporter protein (GLUT2)-equipped chemosensory cells results in multiple feeding-associated functional alterations. <b>Methods</b>: In order to examine this complex symptomatology, (1) dopamine sensitivity was studied in laboratory rats by means of the single-neuron-recording multibarreled microelectrophoretic technique, and (2) after local bilateral microinjection of the selective type 2 glucose transporter proteindemolishing streptozotocin (STZ), open-field, elevated plus maze, two-bottle and taste reactivity tests were performed. <b>Results</b>: A high proportion of the anterior cingulate cortical neurons changed their firing rate in response to microelectrophoretic administration of D-glucose, thus verifying them as local elements of the central glucose-monitoring network. Approximately 20% of the recorded cells displayed activity changes in response to microelectrophoretic application of dopamine, and almost 50% of the glucose-monitoring units here proved to be dopamine-sensitive. Moreover, taste stimulation experiments revealed even higher (80%) gustatory sensitivity dominance of these chemosensory cells. The anterior cingulate cortical STZ microinjections resulted in extensive behavioral and taste-associated functional deficits. <b>Conclusions</b>: The present findings provided evidence for the selective loss of glucose-monitoring neurons in the anterior cingulate cortex leading to motivated behavioral and gustatory alterations. This complex dataset also underlines the varied significance of the type 2 glucose transporter protein-equipped, dopamine-sensitive glucose-monitoring neurons as potential therapeutic targets. These units appear to be indispensable in adaptive control mechanisms of the homeostatic–motivational–emotional–cognitive balance for the overall well-being of the organism.https://www.mdpi.com/2227-9059/12/12/2803anterior cingulate cortexglucose-monitoring neurondopaminestreptozotocinbehavior
spellingShingle Edina Hormay
Bettina László
István Szabó
Kitti Mintál
Beáta Berta
Tamás Ollmann
László Péczely
Bernadett Nagy
Attila Tóth
Kristóf László
László Lénárd
Zoltán Karádi
Dopamine-Sensitive Anterior Cingulate Cortical Glucose-Monitoring Neurons as Potential Therapeutic Targets for Gustatory and Other Behavior Alterations
Biomedicines
anterior cingulate cortex
glucose-monitoring neuron
dopamine
streptozotocin
behavior
title Dopamine-Sensitive Anterior Cingulate Cortical Glucose-Monitoring Neurons as Potential Therapeutic Targets for Gustatory and Other Behavior Alterations
title_full Dopamine-Sensitive Anterior Cingulate Cortical Glucose-Monitoring Neurons as Potential Therapeutic Targets for Gustatory and Other Behavior Alterations
title_fullStr Dopamine-Sensitive Anterior Cingulate Cortical Glucose-Monitoring Neurons as Potential Therapeutic Targets for Gustatory and Other Behavior Alterations
title_full_unstemmed Dopamine-Sensitive Anterior Cingulate Cortical Glucose-Monitoring Neurons as Potential Therapeutic Targets for Gustatory and Other Behavior Alterations
title_short Dopamine-Sensitive Anterior Cingulate Cortical Glucose-Monitoring Neurons as Potential Therapeutic Targets for Gustatory and Other Behavior Alterations
title_sort dopamine sensitive anterior cingulate cortical glucose monitoring neurons as potential therapeutic targets for gustatory and other behavior alterations
topic anterior cingulate cortex
glucose-monitoring neuron
dopamine
streptozotocin
behavior
url https://www.mdpi.com/2227-9059/12/12/2803
work_keys_str_mv AT edinahormay dopaminesensitiveanteriorcingulatecorticalglucosemonitoringneuronsaspotentialtherapeutictargetsforgustatoryandotherbehavioralterations
AT bettinalaszlo dopaminesensitiveanteriorcingulatecorticalglucosemonitoringneuronsaspotentialtherapeutictargetsforgustatoryandotherbehavioralterations
AT istvanszabo dopaminesensitiveanteriorcingulatecorticalglucosemonitoringneuronsaspotentialtherapeutictargetsforgustatoryandotherbehavioralterations
AT kittimintal dopaminesensitiveanteriorcingulatecorticalglucosemonitoringneuronsaspotentialtherapeutictargetsforgustatoryandotherbehavioralterations
AT beataberta dopaminesensitiveanteriorcingulatecorticalglucosemonitoringneuronsaspotentialtherapeutictargetsforgustatoryandotherbehavioralterations
AT tamasollmann dopaminesensitiveanteriorcingulatecorticalglucosemonitoringneuronsaspotentialtherapeutictargetsforgustatoryandotherbehavioralterations
AT laszlopeczely dopaminesensitiveanteriorcingulatecorticalglucosemonitoringneuronsaspotentialtherapeutictargetsforgustatoryandotherbehavioralterations
AT bernadettnagy dopaminesensitiveanteriorcingulatecorticalglucosemonitoringneuronsaspotentialtherapeutictargetsforgustatoryandotherbehavioralterations
AT attilatoth dopaminesensitiveanteriorcingulatecorticalglucosemonitoringneuronsaspotentialtherapeutictargetsforgustatoryandotherbehavioralterations
AT kristoflaszlo dopaminesensitiveanteriorcingulatecorticalglucosemonitoringneuronsaspotentialtherapeutictargetsforgustatoryandotherbehavioralterations
AT laszlolenard dopaminesensitiveanteriorcingulatecorticalglucosemonitoringneuronsaspotentialtherapeutictargetsforgustatoryandotherbehavioralterations
AT zoltankaradi dopaminesensitiveanteriorcingulatecorticalglucosemonitoringneuronsaspotentialtherapeutictargetsforgustatoryandotherbehavioralterations