Case Report: A renal wasting disease caused by a pure deletion of nephrocystin-1

Nephronophthisis is an autosomal recessive disorder associated with the tubular interstitium of the kidney, and can lead to renal failure in children and adolescents. Mutations in the gene encoding nephrocystin-1, NPHP1, are frequently associated with the disease. Here, we describe the case of a chi...

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Main Authors: Ting Dong, Jiajia Luo, Tianhong Sun, Huimin Wu, Qing Zhao, Lina Ma, Jing Yang
Format: Article
Language:English
Published: Frontiers Media S.A. 2025-06-01
Series:Frontiers in Pediatrics
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Online Access:https://www.frontiersin.org/articles/10.3389/fped.2025.1541411/full
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author Ting Dong
Jiajia Luo
Tianhong Sun
Huimin Wu
Qing Zhao
Lina Ma
Jing Yang
author_facet Ting Dong
Jiajia Luo
Tianhong Sun
Huimin Wu
Qing Zhao
Lina Ma
Jing Yang
author_sort Ting Dong
collection DOAJ
description Nephronophthisis is an autosomal recessive disorder associated with the tubular interstitium of the kidney, and can lead to renal failure in children and adolescents. Mutations in the gene encoding nephrocystin-1, NPHP1, are frequently associated with the disease. Here, we describe the case of a child who presented to the clinic with febrile convulsions and who was ultimately diagnosed with nephronophthisis caused by a homozygous deletion of the NPHP1 gene. Alerting pediatricians to the recognition of atypical renal wasting disease and reclarifying the diagnostic value of genetic diagnosis for this disease.
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series Frontiers in Pediatrics
spelling doaj-art-ff83cb79cf384abaaa7b74655ba7dcd92025-08-20T02:36:04ZengFrontiers Media S.A.Frontiers in Pediatrics2296-23602025-06-011310.3389/fped.2025.15414111541411Case Report: A renal wasting disease caused by a pure deletion of nephrocystin-1Ting Dong0Jiajia Luo1Tianhong Sun2Huimin Wu3Qing Zhao4Lina Ma5Jing Yang6Department of Lanzhou University, Lanzhou, ChinaDepartment of Pediatrics, Lanzhou University Second Hospital, Lanzhou, ChinaDepartment of Pediatrics, Lanzhou University Second Hospital, Lanzhou, ChinaDepartment of Pediatrics, Lanzhou University Second Hospital, Lanzhou, ChinaDepartment of Pediatrics, Lanzhou University Second Hospital, Lanzhou, ChinaDepartment of Lanzhou University, Lanzhou, ChinaDepartment of Lanzhou University, Lanzhou, ChinaNephronophthisis is an autosomal recessive disorder associated with the tubular interstitium of the kidney, and can lead to renal failure in children and adolescents. Mutations in the gene encoding nephrocystin-1, NPHP1, are frequently associated with the disease. Here, we describe the case of a child who presented to the clinic with febrile convulsions and who was ultimately diagnosed with nephronophthisis caused by a homozygous deletion of the NPHP1 gene. Alerting pediatricians to the recognition of atypical renal wasting disease and reclarifying the diagnostic value of genetic diagnosis for this disease.https://www.frontiersin.org/articles/10.3389/fped.2025.1541411/fullrenal wasting diseaseNPHP1 genepediatriccase reportsequencing of genome case report (presentation)
spellingShingle Ting Dong
Jiajia Luo
Tianhong Sun
Huimin Wu
Qing Zhao
Lina Ma
Jing Yang
Case Report: A renal wasting disease caused by a pure deletion of nephrocystin-1
Frontiers in Pediatrics
renal wasting disease
NPHP1 gene
pediatric
case report
sequencing of genome case report (presentation)
title Case Report: A renal wasting disease caused by a pure deletion of nephrocystin-1
title_full Case Report: A renal wasting disease caused by a pure deletion of nephrocystin-1
title_fullStr Case Report: A renal wasting disease caused by a pure deletion of nephrocystin-1
title_full_unstemmed Case Report: A renal wasting disease caused by a pure deletion of nephrocystin-1
title_short Case Report: A renal wasting disease caused by a pure deletion of nephrocystin-1
title_sort case report a renal wasting disease caused by a pure deletion of nephrocystin 1
topic renal wasting disease
NPHP1 gene
pediatric
case report
sequencing of genome case report (presentation)
url https://www.frontiersin.org/articles/10.3389/fped.2025.1541411/full
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