Managing Inflammation after Spinal Cord Injury through Manipulation of Macrophage Function

Spinal cord injury (SCI) triggers inflammation with activation of innate immune responses that contribute to secondary injury including oligodendrocyte apoptosis, demyelination, axonal degeneration, and neuronal death. Macrophage activation, accumulation, and persistent inflammation occur in SCI. Ma...

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Main Authors: Yi Ren, Wise Young
Format: Article
Language:English
Published: Wiley 2013-01-01
Series:Neural Plasticity
Online Access:http://dx.doi.org/10.1155/2013/945034
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author Yi Ren
Wise Young
author_facet Yi Ren
Wise Young
author_sort Yi Ren
collection DOAJ
description Spinal cord injury (SCI) triggers inflammation with activation of innate immune responses that contribute to secondary injury including oligodendrocyte apoptosis, demyelination, axonal degeneration, and neuronal death. Macrophage activation, accumulation, and persistent inflammation occur in SCI. Macrophages are heterogeneous cells with extensive functional plasticity and have the capacity to switch phenotypes by factors present in the inflammatory microenvironment of the injured spinal cord. This review will discuss the role of different polarized macrophages and the potential effect of macrophage-based therapies for SCI.
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spelling doaj-art-fe74c063a4e147b89a01767f3ac183e32025-08-20T03:25:46ZengWileyNeural Plasticity2090-59041687-54432013-01-01201310.1155/2013/945034945034Managing Inflammation after Spinal Cord Injury through Manipulation of Macrophage FunctionYi Ren0Wise Young1Department of Biomedical Sciences, College of Medicine, Florida State University, 1115 West Call Street, Tallahassee, FL 32306-4300, USAW. M. Keck Center for Collaborative Neuroscience, Rutgers, The State University of New Jersey, Nelson Labs D-251, 604 Allison Road, Piscataway, NJ 08854, USASpinal cord injury (SCI) triggers inflammation with activation of innate immune responses that contribute to secondary injury including oligodendrocyte apoptosis, demyelination, axonal degeneration, and neuronal death. Macrophage activation, accumulation, and persistent inflammation occur in SCI. Macrophages are heterogeneous cells with extensive functional plasticity and have the capacity to switch phenotypes by factors present in the inflammatory microenvironment of the injured spinal cord. This review will discuss the role of different polarized macrophages and the potential effect of macrophage-based therapies for SCI.http://dx.doi.org/10.1155/2013/945034
spellingShingle Yi Ren
Wise Young
Managing Inflammation after Spinal Cord Injury through Manipulation of Macrophage Function
Neural Plasticity
title Managing Inflammation after Spinal Cord Injury through Manipulation of Macrophage Function
title_full Managing Inflammation after Spinal Cord Injury through Manipulation of Macrophage Function
title_fullStr Managing Inflammation after Spinal Cord Injury through Manipulation of Macrophage Function
title_full_unstemmed Managing Inflammation after Spinal Cord Injury through Manipulation of Macrophage Function
title_short Managing Inflammation after Spinal Cord Injury through Manipulation of Macrophage Function
title_sort managing inflammation after spinal cord injury through manipulation of macrophage function
url http://dx.doi.org/10.1155/2013/945034
work_keys_str_mv AT yiren managinginflammationafterspinalcordinjurythroughmanipulationofmacrophagefunction
AT wiseyoung managinginflammationafterspinalcordinjurythroughmanipulationofmacrophagefunction