Inactivation of by promoter methylation correlated with lymph node metastasis and genomic instability in nasopharyngeal carcinoma

This study aimed to investigate the inactivation of the parkin gene by promoter methylation and its relationship with genome instability in nasopharyngeal carcinoma. Parkin was considered as a tumor suppressor gene in various types of cancers. However, its role in nasopharyngeal carcinoma is unexplo...

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Main Authors: Haifeng Ni, Zhen Zhou, Bo Jiang, Xiaoyang Yuan, Xiaolin Cao, Guangwu Huang, Yong Li
Format: Article
Language:English
Published: SAGE Publishing 2017-03-01
Series:Tumor Biology
Online Access:https://doi.org/10.1177/1010428317695025
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author Haifeng Ni
Zhen Zhou
Bo Jiang
Xiaoyang Yuan
Xiaolin Cao
Guangwu Huang
Yong Li
author_facet Haifeng Ni
Zhen Zhou
Bo Jiang
Xiaoyang Yuan
Xiaolin Cao
Guangwu Huang
Yong Li
author_sort Haifeng Ni
collection DOAJ
description This study aimed to investigate the inactivation of the parkin gene by promoter methylation and its relationship with genome instability in nasopharyngeal carcinoma. Parkin was considered as a tumor suppressor gene in various types of cancers. However, its role in nasopharyngeal carcinoma is unexplored. Genomic instabilities were detected in nasopharyngeal carcinoma tissues by the random amplified polymorphic DNA. The methylation-specific polymerase chain reaction, semi-quantitative reverse transcription polymerase chain reaction, and immunohistochemical analysis were used to detect methylation and mRNA and protein expression of parkin in 54 cases of nasopharyngeal carcinoma tissues and 16 cases of normal nasopharyngeal epithelia tissues, and in 5 nasopharyngeal carcinoma cell lines (CNE1, CNE2, TWO3, C666, and HONE1) and 1 normal nasopharyngeal epithelia cell line (NP69). mRNA expression of parkin in CNE1 and CNE2 was analyzed before and after methyltransferase inhibitor 5-aza-2-deoxycytidine treatment. The relationship between promoter methylation and mRNA expression, demethylation and mRNA expression, and mRNA and protein expression of the gene and clinical factors and genomic instabilities were analyzed. The mRNA and protein expression levels were significantly reduced in 54 cases of human nasopharyngeal carcinoma compared with 16 cases of normal nasopharyngeal epithelia. Parkin -methylated cases showed significantly lower mRNA and protein expression levels compared with unmethylated cases. After 5-aza-2-deoxycytidine treatment, parkin mRNA expression was restored in CNE1 and CNE2; 92.59% (50/54) of nasopharyngeal carcinoma demonstrated genomic instability. Parkin is frequently inactivated by promoter methylation, and its mRNA and protein expression correlate with lymph node metastasis and genomic instability. Parkin deficiency probably promotes tumorigenesis in nasopharyngeal carcinoma.
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spelling doaj-art-fe730eb06738470da334ffd7eece66df2025-08-20T03:22:26ZengSAGE PublishingTumor Biology1423-03802017-03-013910.1177/1010428317695025Inactivation of by promoter methylation correlated with lymph node metastasis and genomic instability in nasopharyngeal carcinomaHaifeng Ni0Zhen Zhou1Bo Jiang2Xiaoyang Yuan3Xiaolin Cao4Guangwu Huang5Yong Li6Department of Otolaryngology, Hangzhou First People’s Hospital, Nanjing Medical University, Hangzhou, ChinaDepartment of Otolaryngology, Hangzhou First People’s Hospital, Nanjing Medical University, Hangzhou, ChinaDepartment of Otolaryngology, Hangzhou First People’s Hospital, Nanjing Medical University, Hangzhou, ChinaDepartment of Otolaryngology, Hangzhou First People’s Hospital, Nanjing Medical University, Hangzhou, ChinaDepartment of Otolaryngology, Hangzhou First People’s Hospital, Nanjing Medical University, Hangzhou, ChinaDepartment of Otolaryngology, First Affiliated Hospital, Guangxi Medical University, Nanning, ChinaDepartment of Otolaryngology, Hangzhou First People’s Hospital, Nanjing Medical University, Hangzhou, ChinaThis study aimed to investigate the inactivation of the parkin gene by promoter methylation and its relationship with genome instability in nasopharyngeal carcinoma. Parkin was considered as a tumor suppressor gene in various types of cancers. However, its role in nasopharyngeal carcinoma is unexplored. Genomic instabilities were detected in nasopharyngeal carcinoma tissues by the random amplified polymorphic DNA. The methylation-specific polymerase chain reaction, semi-quantitative reverse transcription polymerase chain reaction, and immunohistochemical analysis were used to detect methylation and mRNA and protein expression of parkin in 54 cases of nasopharyngeal carcinoma tissues and 16 cases of normal nasopharyngeal epithelia tissues, and in 5 nasopharyngeal carcinoma cell lines (CNE1, CNE2, TWO3, C666, and HONE1) and 1 normal nasopharyngeal epithelia cell line (NP69). mRNA expression of parkin in CNE1 and CNE2 was analyzed before and after methyltransferase inhibitor 5-aza-2-deoxycytidine treatment. The relationship between promoter methylation and mRNA expression, demethylation and mRNA expression, and mRNA and protein expression of the gene and clinical factors and genomic instabilities were analyzed. The mRNA and protein expression levels were significantly reduced in 54 cases of human nasopharyngeal carcinoma compared with 16 cases of normal nasopharyngeal epithelia. Parkin -methylated cases showed significantly lower mRNA and protein expression levels compared with unmethylated cases. After 5-aza-2-deoxycytidine treatment, parkin mRNA expression was restored in CNE1 and CNE2; 92.59% (50/54) of nasopharyngeal carcinoma demonstrated genomic instability. Parkin is frequently inactivated by promoter methylation, and its mRNA and protein expression correlate with lymph node metastasis and genomic instability. Parkin deficiency probably promotes tumorigenesis in nasopharyngeal carcinoma.https://doi.org/10.1177/1010428317695025
spellingShingle Haifeng Ni
Zhen Zhou
Bo Jiang
Xiaoyang Yuan
Xiaolin Cao
Guangwu Huang
Yong Li
Inactivation of by promoter methylation correlated with lymph node metastasis and genomic instability in nasopharyngeal carcinoma
Tumor Biology
title Inactivation of by promoter methylation correlated with lymph node metastasis and genomic instability in nasopharyngeal carcinoma
title_full Inactivation of by promoter methylation correlated with lymph node metastasis and genomic instability in nasopharyngeal carcinoma
title_fullStr Inactivation of by promoter methylation correlated with lymph node metastasis and genomic instability in nasopharyngeal carcinoma
title_full_unstemmed Inactivation of by promoter methylation correlated with lymph node metastasis and genomic instability in nasopharyngeal carcinoma
title_short Inactivation of by promoter methylation correlated with lymph node metastasis and genomic instability in nasopharyngeal carcinoma
title_sort inactivation of by promoter methylation correlated with lymph node metastasis and genomic instability in nasopharyngeal carcinoma
url https://doi.org/10.1177/1010428317695025
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