Obtusifolin ameliorates dry eye model in rats by reducing inflammation and blocking MAPK/NF-κB pathways

AIM: To investigate the functions and potential mechanisms of obtusifolin in dry eye disease (DED) in a rat model. METHODS: A rat DED model was established via topical administration of benzalkonium chloride (BAC), followed by administration of obtusifolin. Conjunctival irritation score and tear pro...

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Main Authors: Dan Zhu, Xiao-Yang Wu, Liang-Chang Li
Format: Article
Language:English
Published: Press of International Journal of Ophthalmology (IJO PRESS) 2025-08-01
Series:International Journal of Ophthalmology
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Online Access:http://ies.ijo.cn/en_publish/2025/8/20250802.pdf
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author Dan Zhu
Xiao-Yang Wu
Liang-Chang Li
author_facet Dan Zhu
Xiao-Yang Wu
Liang-Chang Li
author_sort Dan Zhu
collection DOAJ
description AIM: To investigate the functions and potential mechanisms of obtusifolin in dry eye disease (DED) in a rat model. METHODS: A rat DED model was established via topical administration of benzalkonium chloride (BAC), followed by administration of obtusifolin. Conjunctival irritation score and tear production were measured to evaluate DED symptoms. Enzyme-linked immunosorbent assay (ELISA) was employed for determining inflammatory cytokine levels in rat conjunctiva. Periodic acid-Schiff staining and corneal fluorescein staining were implemented for assessing goblet cell numbers and corneal epithelial defects, respectively. Western blotting showed zonula occludens-1 (ZO-1), matrix metalloproteinase-9 (MMP-9), and mitogen-activated protein kinase (MAPK)/nuclear factor kappa B (NF-κB) signaling-related protein levels in the conjunctiva. RESULTS: Topical application of obtusifolin alleviated conjunctival irritation and enhanced tear production in BAC-induced DED rats. Obtusifolin attenuated conjunctival inflammatory response and goblet cell loss as well as corneal epithelial barrier disruption in DED rats. Obtusifolin suppressed extracellular signal-regulated kinase (ERK), p38, and NF-κB phosphorylation in the conjunctiva of DED rats. CONCLUSION: Obtusifolin ameliorates DED in rats possibly by alleviating inflammation via the inactivation of MAPK/NF-κB signaling.
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institution Kabale University
issn 2222-3959
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language English
publishDate 2025-08-01
publisher Press of International Journal of Ophthalmology (IJO PRESS)
record_format Article
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spelling doaj-art-fe4aebdc722a479688b4eaf52bc278cc2025-08-20T03:27:28ZengPress of International Journal of Ophthalmology (IJO PRESS)International Journal of Ophthalmology2222-39592227-48982025-08-011881426143210.18240/ijo.2025.08.0220250802Obtusifolin ameliorates dry eye model in rats by reducing inflammation and blocking MAPK/NF-κB pathwaysDan Zhu0Xiao-Yang Wu1Liang-Chang Li2Dan Zhu. Hubei Provincial Hospital of Traditional Chinese Medicine, Wuhan 430061, Hubei Province, China. zhudan@hbhtcm.comDepartment of Ophthalmology, Hubei Provincial Hospital of Traditional Chinese Medicine, Wuhan 430061, Hubei Province, China; Affiliated Hospital of Hubei University of Chinese Medicine, Wuhan 430061, Hubei Province, China; Hubei Provincial Key Laboratory of Liver and Kidney Research and Application of Traditional Chinese Medicine, Wuhan 430061, Hubei Province, ChinaDepartment of Ophthalmology, Hubei Provincial Hospital of Traditional Chinese Medicine, Wuhan 430061, Hubei Province, China; Affiliated Hospital of Hubei University of Chinese Medicine, Wuhan 430061, Hubei Province, China; Hubei Provincial Key Laboratory of Liver and Kidney Research and Application of Traditional Chinese Medicine, Wuhan 430061, Hubei Province, ChinaAIM: To investigate the functions and potential mechanisms of obtusifolin in dry eye disease (DED) in a rat model. METHODS: A rat DED model was established via topical administration of benzalkonium chloride (BAC), followed by administration of obtusifolin. Conjunctival irritation score and tear production were measured to evaluate DED symptoms. Enzyme-linked immunosorbent assay (ELISA) was employed for determining inflammatory cytokine levels in rat conjunctiva. Periodic acid-Schiff staining and corneal fluorescein staining were implemented for assessing goblet cell numbers and corneal epithelial defects, respectively. Western blotting showed zonula occludens-1 (ZO-1), matrix metalloproteinase-9 (MMP-9), and mitogen-activated protein kinase (MAPK)/nuclear factor kappa B (NF-κB) signaling-related protein levels in the conjunctiva. RESULTS: Topical application of obtusifolin alleviated conjunctival irritation and enhanced tear production in BAC-induced DED rats. Obtusifolin attenuated conjunctival inflammatory response and goblet cell loss as well as corneal epithelial barrier disruption in DED rats. Obtusifolin suppressed extracellular signal-regulated kinase (ERK), p38, and NF-κB phosphorylation in the conjunctiva of DED rats. CONCLUSION: Obtusifolin ameliorates DED in rats possibly by alleviating inflammation via the inactivation of MAPK/NF-κB signaling.http://ies.ijo.cn/en_publish/2025/8/20250802.pdfdry eye diseaseobtusifolininflammationtear productionmitogen-activated protein kinase/nuclear factor kappa brats
spellingShingle Dan Zhu
Xiao-Yang Wu
Liang-Chang Li
Obtusifolin ameliorates dry eye model in rats by reducing inflammation and blocking MAPK/NF-κB pathways
International Journal of Ophthalmology
dry eye disease
obtusifolin
inflammation
tear production
mitogen-activated protein kinase/nuclear factor kappa b
rats
title Obtusifolin ameliorates dry eye model in rats by reducing inflammation and blocking MAPK/NF-κB pathways
title_full Obtusifolin ameliorates dry eye model in rats by reducing inflammation and blocking MAPK/NF-κB pathways
title_fullStr Obtusifolin ameliorates dry eye model in rats by reducing inflammation and blocking MAPK/NF-κB pathways
title_full_unstemmed Obtusifolin ameliorates dry eye model in rats by reducing inflammation and blocking MAPK/NF-κB pathways
title_short Obtusifolin ameliorates dry eye model in rats by reducing inflammation and blocking MAPK/NF-κB pathways
title_sort obtusifolin ameliorates dry eye model in rats by reducing inflammation and blocking mapk nf κb pathways
topic dry eye disease
obtusifolin
inflammation
tear production
mitogen-activated protein kinase/nuclear factor kappa b
rats
url http://ies.ijo.cn/en_publish/2025/8/20250802.pdf
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AT xiaoyangwu obtusifolinamelioratesdryeyemodelinratsbyreducinginflammationandblockingmapknfkbpathways
AT liangchangli obtusifolinamelioratesdryeyemodelinratsbyreducinginflammationandblockingmapknfkbpathways