MPV17 Loss Causes Deoxynucleotide Insufficiency and Slow DNA Replication in Mitochondria.

MPV17 is a mitochondrial inner membrane protein whose dysfunction causes mitochondrial DNA abnormalities and disease by an unknown mechanism. Perturbations of deoxynucleoside triphosphate (dNTP) pools are a recognized cause of mitochondrial genomic instability; therefore, we determined DNA copy numb...

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Main Authors: Ilaria Dalla Rosa, Yolanda Cámara, Romina Durigon, Chloe F Moss, Sara Vidoni, Gokhan Akman, Lilian Hunt, Mark A Johnson, Sarah Grocott, Liya Wang, David R Thorburn, Michio Hirano, Joanna Poulton, Robert W Taylor, Greg Elgar, Ramon Martí, Peter Voshol, Ian J Holt, Antonella Spinazzola
Format: Article
Language:English
Published: Public Library of Science (PLoS) 2016-01-01
Series:PLoS Genetics
Online Access:https://doi.org/10.1371/journal.pgen.1005779
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author Ilaria Dalla Rosa
Yolanda Cámara
Romina Durigon
Chloe F Moss
Sara Vidoni
Gokhan Akman
Lilian Hunt
Mark A Johnson
Sarah Grocott
Liya Wang
David R Thorburn
Michio Hirano
Joanna Poulton
Robert W Taylor
Greg Elgar
Ramon Martí
Peter Voshol
Ian J Holt
Antonella Spinazzola
author_facet Ilaria Dalla Rosa
Yolanda Cámara
Romina Durigon
Chloe F Moss
Sara Vidoni
Gokhan Akman
Lilian Hunt
Mark A Johnson
Sarah Grocott
Liya Wang
David R Thorburn
Michio Hirano
Joanna Poulton
Robert W Taylor
Greg Elgar
Ramon Martí
Peter Voshol
Ian J Holt
Antonella Spinazzola
author_sort Ilaria Dalla Rosa
collection DOAJ
description MPV17 is a mitochondrial inner membrane protein whose dysfunction causes mitochondrial DNA abnormalities and disease by an unknown mechanism. Perturbations of deoxynucleoside triphosphate (dNTP) pools are a recognized cause of mitochondrial genomic instability; therefore, we determined DNA copy number and dNTP levels in mitochondria of two models of MPV17 deficiency. In Mpv17 ablated mice, liver mitochondria showed substantial decreases in the levels of dGTP and dTTP and severe mitochondrial DNA depletion, whereas the dNTP pool was not significantly altered in kidney and brain mitochondria that had near normal levels of DNA. The shortage of mitochondrial dNTPs in Mpv17-/- liver slows the DNA replication in the organelle, as evidenced by the elevated level of replication intermediates. Quiescent fibroblasts of MPV17-mutant patients recapitulate key features of the primary affected tissue of the Mpv17-/- mice, displaying virtual absence of the protein, decreased dNTP levels and mitochondrial DNA depletion. Notably, the mitochondrial DNA loss in the patients' quiescent fibroblasts was prevented and rescued by deoxynucleoside supplementation. Thus, our study establishes dNTP insufficiency in the mitochondria as the cause of mitochondrial DNA depletion in MPV17 deficiency, and identifies deoxynucleoside supplementation as a potential therapeutic strategy for MPV17-related disease. Moreover, changes in the expression of factors involved in mitochondrial deoxynucleotide homeostasis indicate a remodeling of nucleotide metabolism in MPV17 disease models, which suggests mitochondria lacking functional MPV17 have a restricted purine mitochondrial salvage pathway.
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spelling doaj-art-fd0c8fb28a5f40f98a92dee74e9682c62025-08-20T03:10:08ZengPublic Library of Science (PLoS)PLoS Genetics1553-73901553-74042016-01-01121e100577910.1371/journal.pgen.1005779MPV17 Loss Causes Deoxynucleotide Insufficiency and Slow DNA Replication in Mitochondria.Ilaria Dalla RosaYolanda CámaraRomina DurigonChloe F MossSara VidoniGokhan AkmanLilian HuntMark A JohnsonSarah GrocottLiya WangDavid R ThorburnMichio HiranoJoanna PoultonRobert W TaylorGreg ElgarRamon MartíPeter VosholIan J HoltAntonella SpinazzolaMPV17 is a mitochondrial inner membrane protein whose dysfunction causes mitochondrial DNA abnormalities and disease by an unknown mechanism. Perturbations of deoxynucleoside triphosphate (dNTP) pools are a recognized cause of mitochondrial genomic instability; therefore, we determined DNA copy number and dNTP levels in mitochondria of two models of MPV17 deficiency. In Mpv17 ablated mice, liver mitochondria showed substantial decreases in the levels of dGTP and dTTP and severe mitochondrial DNA depletion, whereas the dNTP pool was not significantly altered in kidney and brain mitochondria that had near normal levels of DNA. The shortage of mitochondrial dNTPs in Mpv17-/- liver slows the DNA replication in the organelle, as evidenced by the elevated level of replication intermediates. Quiescent fibroblasts of MPV17-mutant patients recapitulate key features of the primary affected tissue of the Mpv17-/- mice, displaying virtual absence of the protein, decreased dNTP levels and mitochondrial DNA depletion. Notably, the mitochondrial DNA loss in the patients' quiescent fibroblasts was prevented and rescued by deoxynucleoside supplementation. Thus, our study establishes dNTP insufficiency in the mitochondria as the cause of mitochondrial DNA depletion in MPV17 deficiency, and identifies deoxynucleoside supplementation as a potential therapeutic strategy for MPV17-related disease. Moreover, changes in the expression of factors involved in mitochondrial deoxynucleotide homeostasis indicate a remodeling of nucleotide metabolism in MPV17 disease models, which suggests mitochondria lacking functional MPV17 have a restricted purine mitochondrial salvage pathway.https://doi.org/10.1371/journal.pgen.1005779
spellingShingle Ilaria Dalla Rosa
Yolanda Cámara
Romina Durigon
Chloe F Moss
Sara Vidoni
Gokhan Akman
Lilian Hunt
Mark A Johnson
Sarah Grocott
Liya Wang
David R Thorburn
Michio Hirano
Joanna Poulton
Robert W Taylor
Greg Elgar
Ramon Martí
Peter Voshol
Ian J Holt
Antonella Spinazzola
MPV17 Loss Causes Deoxynucleotide Insufficiency and Slow DNA Replication in Mitochondria.
PLoS Genetics
title MPV17 Loss Causes Deoxynucleotide Insufficiency and Slow DNA Replication in Mitochondria.
title_full MPV17 Loss Causes Deoxynucleotide Insufficiency and Slow DNA Replication in Mitochondria.
title_fullStr MPV17 Loss Causes Deoxynucleotide Insufficiency and Slow DNA Replication in Mitochondria.
title_full_unstemmed MPV17 Loss Causes Deoxynucleotide Insufficiency and Slow DNA Replication in Mitochondria.
title_short MPV17 Loss Causes Deoxynucleotide Insufficiency and Slow DNA Replication in Mitochondria.
title_sort mpv17 loss causes deoxynucleotide insufficiency and slow dna replication in mitochondria
url https://doi.org/10.1371/journal.pgen.1005779
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