Complement-Inhibiting Therapy of Atypical Haemolytic Uremic Syndrome in a Patient With Factor H Mutation

Atypical haemolytic uremic syndrome (aHUS) is an extremely rare pathology with the development of complement-mediated thrombotic microangiopathy (TMA). Before eculizumab, a humanized IgG monoclonal antibody to the complement component C5, the prognosis of total and renal survival with aHUS was unfav...

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Main Authors: Irina N. Lupan, Alexander Yu. Pischalnikov, Hadizha M. Emirova, Ekaterina S. Stolyarevich, Alexander M. Volyansky, Larisa V. Glukhova
Format: Article
Language:Russian
Published: Union of pediatricians of Russia 2019-02-01
Series:Педиатрическая фармакология
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Online Access:https://www.pedpharma.ru/jour/article/view/1682
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author Irina N. Lupan
Alexander Yu. Pischalnikov
Hadizha M. Emirova
Ekaterina S. Stolyarevich
Alexander M. Volyansky
Larisa V. Glukhova
author_facet Irina N. Lupan
Alexander Yu. Pischalnikov
Hadizha M. Emirova
Ekaterina S. Stolyarevich
Alexander M. Volyansky
Larisa V. Glukhova
author_sort Irina N. Lupan
collection DOAJ
description Atypical haemolytic uremic syndrome (aHUS) is an extremely rare pathology with the development of complement-mediated thrombotic microangiopathy (TMA). Before eculizumab, a humanized IgG monoclonal antibody to the complement component C5, the prognosis of total and renal survival with aHUS was unfavourable due to the high probability of death and the development of end-stage chronic renal failure in surviving patients. This article presents a clinical observation of a patient with aHUS who had an identified heterozygous factor H (CFH) mutation — c.3653G>A(p.Cys1218Tyr), and two heterozygous variants of polymorphism in the same gene — c.2016A>G; c.2808G>T. Despite the achievement of haematological remission of TMA against the background of plasma therapy, the child developed dialysis-dependent renal failure. Treatment with eculizumab in a patient with chronic kidney damage provided a significant improvement in their function, maintaining a stable remission and improving the quality of life of the patient with aHUS.Kh. М. Emirova, Е. S. Stolyarevich take part in educational events for doctors as lecturers with the support of Alexion Pharma. The other contributors confirmed the absence of a reportable conflict of interest.
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spelling doaj-art-fc6b5ba93b4b461ab2e794afaa3e25442025-08-20T03:59:20ZrusUnion of pediatricians of RussiaПедиатрическая фармакология1727-57762500-30892019-02-0115647047810.15690/pf.v15i6.19841618Complement-Inhibiting Therapy of Atypical Haemolytic Uremic Syndrome in a Patient With Factor H MutationIrina N. Lupan0Alexander Yu. Pischalnikov1Hadizha M. Emirova2Ekaterina S. Stolyarevich3Alexander M. Volyansky4Larisa V. Glukhova5South Ural State Medical University of the Ministry of Healthcare of Russia, Institute for Continuing Professional Education; Children’s City Clinical Hospital No.1; Chelyabinsk Regional Children’s Clinical HospitalSouth Ural State Medical University of the Ministry of Healthcare of Russia, Institute for Continuing Professional Education; Children’s City Clinical Hospital No.1; Chelyabinsk Regional Children’s Clinical HospitalA. I. Yevdokimov Moscow State University of Medicine and Dentistry; St. Vladimir Children’s City Clinical HospitalA. I. Yevdokimov Moscow State University of Medicine and DentistryChelyabinsk Regional Children’s Clinical HospitalChildren’s City Clinical Hospital No.1Atypical haemolytic uremic syndrome (aHUS) is an extremely rare pathology with the development of complement-mediated thrombotic microangiopathy (TMA). Before eculizumab, a humanized IgG monoclonal antibody to the complement component C5, the prognosis of total and renal survival with aHUS was unfavourable due to the high probability of death and the development of end-stage chronic renal failure in surviving patients. This article presents a clinical observation of a patient with aHUS who had an identified heterozygous factor H (CFH) mutation — c.3653G>A(p.Cys1218Tyr), and two heterozygous variants of polymorphism in the same gene — c.2016A>G; c.2808G>T. Despite the achievement of haematological remission of TMA against the background of plasma therapy, the child developed dialysis-dependent renal failure. Treatment with eculizumab in a patient with chronic kidney damage provided a significant improvement in their function, maintaining a stable remission and improving the quality of life of the patient with aHUS.Kh. М. Emirova, Е. S. Stolyarevich take part in educational events for doctors as lecturers with the support of Alexion Pharma. The other contributors confirmed the absence of a reportable conflict of interest.https://www.pedpharma.ru/jour/article/view/1682atypical haemolytic uremic syndromethrombotic microangiopathyplasma therapyeculizumabrenal failuredialysis
spellingShingle Irina N. Lupan
Alexander Yu. Pischalnikov
Hadizha M. Emirova
Ekaterina S. Stolyarevich
Alexander M. Volyansky
Larisa V. Glukhova
Complement-Inhibiting Therapy of Atypical Haemolytic Uremic Syndrome in a Patient With Factor H Mutation
Педиатрическая фармакология
atypical haemolytic uremic syndrome
thrombotic microangiopathy
plasma therapy
eculizumab
renal failure
dialysis
title Complement-Inhibiting Therapy of Atypical Haemolytic Uremic Syndrome in a Patient With Factor H Mutation
title_full Complement-Inhibiting Therapy of Atypical Haemolytic Uremic Syndrome in a Patient With Factor H Mutation
title_fullStr Complement-Inhibiting Therapy of Atypical Haemolytic Uremic Syndrome in a Patient With Factor H Mutation
title_full_unstemmed Complement-Inhibiting Therapy of Atypical Haemolytic Uremic Syndrome in a Patient With Factor H Mutation
title_short Complement-Inhibiting Therapy of Atypical Haemolytic Uremic Syndrome in a Patient With Factor H Mutation
title_sort complement inhibiting therapy of atypical haemolytic uremic syndrome in a patient with factor h mutation
topic atypical haemolytic uremic syndrome
thrombotic microangiopathy
plasma therapy
eculizumab
renal failure
dialysis
url https://www.pedpharma.ru/jour/article/view/1682
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