Influence of Peripheral Artery Disease and Statin Therapy on Apolipoprotein Profiles

Apolipoprotein B is a stronger predictor of myocardial infarction than LDL cholesterol, and it is inversely related to physical activity and modifiable with exercise training. As such, apolipoprotein measures may be of particular relevance for subjects with PAD and claudication. We compared plasma a...

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Main Authors: Andrew W. Gardner, Petar Alaupovic, Donald E. Parker, Polly S. Montgomery, Omar L. Esponda, Ana I. Casanegra
Format: Article
Language:English
Published: Wiley 2013-01-01
Series:International Journal of Vascular Medicine
Online Access:http://dx.doi.org/10.1155/2013/548764
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author Andrew W. Gardner
Petar Alaupovic
Donald E. Parker
Polly S. Montgomery
Omar L. Esponda
Ana I. Casanegra
author_facet Andrew W. Gardner
Petar Alaupovic
Donald E. Parker
Polly S. Montgomery
Omar L. Esponda
Ana I. Casanegra
author_sort Andrew W. Gardner
collection DOAJ
description Apolipoprotein B is a stronger predictor of myocardial infarction than LDL cholesterol, and it is inversely related to physical activity and modifiable with exercise training. As such, apolipoprotein measures may be of particular relevance for subjects with PAD and claudication. We compared plasma apolipoprotein profiles in 29 subjects with peripheral artery disease (PAD) and intermittent claudication and in 39 control subjects. Furthermore, we compared the plasma apolipoprotein profiles of subjects with PAD either treated (n=17) or untreated (n=12) with statin medications. For the apolipoprotein subparticle analyses, subjects with PAD had higher age-adjusted Lp-B:C (P<0.05) and lower values of Lp-A-I:A-II (P<0.05) than controls. The PAD group taking statins had lower age-adjusted values for apoB (P<0.05), Lp-A-II:B:C:D:E (P<0.05), Lp-B:E + Lp-B:C:E (P<0.05), Lp-B:C (P<0.05), and Lp-A-I (P<0.05) than the untreated PAD group. Subjects with PAD have impaired apolipoprotein profiles than controls, characterized by Lp-B:C and Lp-A-I:A-II. Furthermore, subjects with PAD on statin medications have a more favorable risk profile, particularly noted in multiple apolipoprotein subparticles. The efficacy of statin therapy to improve cardiovascular risk appears more evident in the apolipoprotein sub-particle profile than in the more traditional lipid profile of subjects with PAD and claudication. This trial is registered with ClinicalTrials.gov NCT00618670.
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spelling doaj-art-fb9e78445c9a44bc8b20387cbcf8eaf72025-08-20T02:22:55ZengWileyInternational Journal of Vascular Medicine2090-28242090-28322013-01-01201310.1155/2013/548764548764Influence of Peripheral Artery Disease and Statin Therapy on Apolipoprotein ProfilesAndrew W. Gardner0Petar Alaupovic1Donald E. Parker2Polly S. Montgomery3Omar L. Esponda4Ana I. Casanegra5Reynolds Oklahoma Center on Aging, Donald W. Reynolds Department of Geriatric Medicine, University of Oklahoma Health Sciences Center (OUHSC), Oklahoma City, OK 73117, USAOklahoma Medical Research Foundation, Oklahoma City, OK 73104, USADepartment of Biostatistics and Epidemiology, OUHSC, Oklahoma City, OK 73117, USAReynolds Oklahoma Center on Aging, Donald W. Reynolds Department of Geriatric Medicine, University of Oklahoma Health Sciences Center (OUHSC), Oklahoma City, OK 73117, USACardiovascular Section, Department of Medicine, OUHSC, Oklahoma City, OK 73117, USACardiovascular Section, Department of Medicine, OUHSC, Oklahoma City, OK 73117, USAApolipoprotein B is a stronger predictor of myocardial infarction than LDL cholesterol, and it is inversely related to physical activity and modifiable with exercise training. As such, apolipoprotein measures may be of particular relevance for subjects with PAD and claudication. We compared plasma apolipoprotein profiles in 29 subjects with peripheral artery disease (PAD) and intermittent claudication and in 39 control subjects. Furthermore, we compared the plasma apolipoprotein profiles of subjects with PAD either treated (n=17) or untreated (n=12) with statin medications. For the apolipoprotein subparticle analyses, subjects with PAD had higher age-adjusted Lp-B:C (P<0.05) and lower values of Lp-A-I:A-II (P<0.05) than controls. The PAD group taking statins had lower age-adjusted values for apoB (P<0.05), Lp-A-II:B:C:D:E (P<0.05), Lp-B:E + Lp-B:C:E (P<0.05), Lp-B:C (P<0.05), and Lp-A-I (P<0.05) than the untreated PAD group. Subjects with PAD have impaired apolipoprotein profiles than controls, characterized by Lp-B:C and Lp-A-I:A-II. Furthermore, subjects with PAD on statin medications have a more favorable risk profile, particularly noted in multiple apolipoprotein subparticles. The efficacy of statin therapy to improve cardiovascular risk appears more evident in the apolipoprotein sub-particle profile than in the more traditional lipid profile of subjects with PAD and claudication. This trial is registered with ClinicalTrials.gov NCT00618670.http://dx.doi.org/10.1155/2013/548764
spellingShingle Andrew W. Gardner
Petar Alaupovic
Donald E. Parker
Polly S. Montgomery
Omar L. Esponda
Ana I. Casanegra
Influence of Peripheral Artery Disease and Statin Therapy on Apolipoprotein Profiles
International Journal of Vascular Medicine
title Influence of Peripheral Artery Disease and Statin Therapy on Apolipoprotein Profiles
title_full Influence of Peripheral Artery Disease and Statin Therapy on Apolipoprotein Profiles
title_fullStr Influence of Peripheral Artery Disease and Statin Therapy on Apolipoprotein Profiles
title_full_unstemmed Influence of Peripheral Artery Disease and Statin Therapy on Apolipoprotein Profiles
title_short Influence of Peripheral Artery Disease and Statin Therapy on Apolipoprotein Profiles
title_sort influence of peripheral artery disease and statin therapy on apolipoprotein profiles
url http://dx.doi.org/10.1155/2013/548764
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