Spinal changes of a newly isolated neuropeptide endomorphin-2 concomitant with vincristine-induced allodynia.

Chemotherapy-induced neuropathic pain (CNP) is the major dose-limiting factor in cancer chemotherapy. However, the neural mechanisms underlying CNP remain unclear. There is increasing evidence implicating the involvement of spinal endomorphin-2 (EM2) in neuropathic pain. In this study, we used a vin...

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Main Authors: Yang Yang, Yong-Gang Zhang, Guo-An Lin, He-Qiu Xie, Hai-Tao Pan, Ben-Qing Huang, Ji-Dong Liu, Hui Liu, Nan Zhang, Li Li, Jian-Hua Chen
Format: Article
Language:English
Published: Public Library of Science (PLoS) 2014-01-01
Series:PLoS ONE
Online Access:https://journals.plos.org/plosone/article/file?id=10.1371/journal.pone.0089583&type=printable
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author Yang Yang
Yong-Gang Zhang
Guo-An Lin
He-Qiu Xie
Hai-Tao Pan
Ben-Qing Huang
Ji-Dong Liu
Hui Liu
Nan Zhang
Li Li
Jian-Hua Chen
author_facet Yang Yang
Yong-Gang Zhang
Guo-An Lin
He-Qiu Xie
Hai-Tao Pan
Ben-Qing Huang
Ji-Dong Liu
Hui Liu
Nan Zhang
Li Li
Jian-Hua Chen
author_sort Yang Yang
collection DOAJ
description Chemotherapy-induced neuropathic pain (CNP) is the major dose-limiting factor in cancer chemotherapy. However, the neural mechanisms underlying CNP remain unclear. There is increasing evidence implicating the involvement of spinal endomorphin-2 (EM2) in neuropathic pain. In this study, we used a vincristine-evoked rat CNP model displaying mechanical allodynia and central sensitization, and observed a significant decrease in the expression of spinal EM2 in CNP. Also, while intrathecal administration of exogenous EM2 attenuated allodynia and central sensitization, the mu-opioid receptor antagonist β-funaltrexamine facilitated these events. We found that the reduction in spinal EM2 was mediated by increased activity of dipeptidylpeptidase IV, possibly as a consequence of chemotherapy-induced oxidative stress. Taken together, our findings suggest that a decrease in spinal EM2 expression causes the loss of endogenous analgesia and leads to enhanced pain sensation in CNP.
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publisher Public Library of Science (PLoS)
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series PLoS ONE
spelling doaj-art-fa628c861ba64c1f863c01733f5cbfd02025-08-20T03:01:13ZengPublic Library of Science (PLoS)PLoS ONE1932-62032014-01-0192e8958310.1371/journal.pone.0089583Spinal changes of a newly isolated neuropeptide endomorphin-2 concomitant with vincristine-induced allodynia.Yang YangYong-Gang ZhangGuo-An LinHe-Qiu XieHai-Tao PanBen-Qing HuangJi-Dong LiuHui LiuNan ZhangLi LiJian-Hua ChenChemotherapy-induced neuropathic pain (CNP) is the major dose-limiting factor in cancer chemotherapy. However, the neural mechanisms underlying CNP remain unclear. There is increasing evidence implicating the involvement of spinal endomorphin-2 (EM2) in neuropathic pain. In this study, we used a vincristine-evoked rat CNP model displaying mechanical allodynia and central sensitization, and observed a significant decrease in the expression of spinal EM2 in CNP. Also, while intrathecal administration of exogenous EM2 attenuated allodynia and central sensitization, the mu-opioid receptor antagonist β-funaltrexamine facilitated these events. We found that the reduction in spinal EM2 was mediated by increased activity of dipeptidylpeptidase IV, possibly as a consequence of chemotherapy-induced oxidative stress. Taken together, our findings suggest that a decrease in spinal EM2 expression causes the loss of endogenous analgesia and leads to enhanced pain sensation in CNP.https://journals.plos.org/plosone/article/file?id=10.1371/journal.pone.0089583&type=printable
spellingShingle Yang Yang
Yong-Gang Zhang
Guo-An Lin
He-Qiu Xie
Hai-Tao Pan
Ben-Qing Huang
Ji-Dong Liu
Hui Liu
Nan Zhang
Li Li
Jian-Hua Chen
Spinal changes of a newly isolated neuropeptide endomorphin-2 concomitant with vincristine-induced allodynia.
PLoS ONE
title Spinal changes of a newly isolated neuropeptide endomorphin-2 concomitant with vincristine-induced allodynia.
title_full Spinal changes of a newly isolated neuropeptide endomorphin-2 concomitant with vincristine-induced allodynia.
title_fullStr Spinal changes of a newly isolated neuropeptide endomorphin-2 concomitant with vincristine-induced allodynia.
title_full_unstemmed Spinal changes of a newly isolated neuropeptide endomorphin-2 concomitant with vincristine-induced allodynia.
title_short Spinal changes of a newly isolated neuropeptide endomorphin-2 concomitant with vincristine-induced allodynia.
title_sort spinal changes of a newly isolated neuropeptide endomorphin 2 concomitant with vincristine induced allodynia
url https://journals.plos.org/plosone/article/file?id=10.1371/journal.pone.0089583&type=printable
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