Genes Related to Mitochondrial Functions, Protein Degradation, and Chromatin Folding Are Differentially Expressed in Lymphomonocytes of Rett Syndrome Patients
Rett syndrome (RTT) is mainly caused by mutations in the X-linked methyl-CpG binding protein (MeCP2) gene. By binding to methylated promoters on CpG islands, MeCP2 protein is able to modulate several genes and important cellular pathways. Therefore, mutations in MeCP2 can seriously affect the cellul...
Saved in:
| Main Authors: | Alessandra Pecorelli, Guido Leoni, Franco Cervellati, Raffaella Canali, Cinzia Signorini, Silvia Leoncini, Alessio Cortelazzo, Claudio De Felice, Lucia Ciccoli, Joussef Hayek, Giuseppe Valacchi |
|---|---|
| Format: | Article |
| Language: | English |
| Published: |
Wiley
2013-01-01
|
| Series: | Mediators of Inflammation |
| Online Access: | http://dx.doi.org/10.1155/2013/137629 |
| Tags: |
Add Tag
No Tags, Be the first to tag this record!
|
Similar Items
-
Inflammatory Lung Disease in Rett Syndrome
by: Claudio De Felice, et al.
Published: (2014-01-01) -
Effects of ω-3 Polyunsaturated Fatty Acids on Plasma Proteome in Rett Syndrome
by: Claudio De Felice, et al.
Published: (2013-01-01) -
Effects of ω-3 PUFAs Supplementation on Myocardial Function and Oxidative Stress Markers in Typical Rett Syndrome
by: Silvia Maffei, et al.
Published: (2014-01-01) -
Persistent Unresolved Inflammation in the Mecp2-308 Female Mutated Mouse Model of Rett Syndrome
by: Alessio Cortelazzo, et al.
Published: (2017-01-01) -
Revealing the complexity of a monogenic disease: rett syndrome exome sequencing.
by: Elisa Grillo, et al.
Published: (2013-01-01)