Tryptophan pathway profiling in multiple sclerosis patients treated with ocrelizumab

IntroductionL-Tryptophan (Trp) metabolism is impaired across various chronic inflammatory pathologies, including Multiple Sclerosis (MS). Trp processing relies on three metabolic routes, namely Kynurenine, Serotonin and Indole pathways. The host microbiota significantly impacts Trp metabolism, prima...

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Main Authors: Carolina Ricci, Matteo Pivetta, Eleonora Martinis, Viviana Valeri, Carolina Colliva, Nicola Giacchè, Simone Lorenzut, Daniela Cargnelutti, Barbara Frossi, Silvia Tonon
Format: Article
Language:English
Published: Frontiers Media S.A. 2025-06-01
Series:Frontiers in Immunology
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Online Access:https://www.frontiersin.org/articles/10.3389/fimmu.2025.1603663/full
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author Carolina Ricci
Matteo Pivetta
Eleonora Martinis
Viviana Valeri
Carolina Colliva
Nicola Giacchè
Simone Lorenzut
Daniela Cargnelutti
Barbara Frossi
Silvia Tonon
author_facet Carolina Ricci
Matteo Pivetta
Eleonora Martinis
Viviana Valeri
Carolina Colliva
Nicola Giacchè
Simone Lorenzut
Daniela Cargnelutti
Barbara Frossi
Silvia Tonon
author_sort Carolina Ricci
collection DOAJ
description IntroductionL-Tryptophan (Trp) metabolism is impaired across various chronic inflammatory pathologies, including Multiple Sclerosis (MS). Trp processing relies on three metabolic routes, namely Kynurenine, Serotonin and Indole pathways. The host microbiota significantly impacts Trp metabolism, primarily by being responsible for Indole metabolites production and secondarily by shaping both Kynurenine and Serotonin pathways. Pathological conditions and pharmaceutical treatments can elicit changes in microbial populations, leading to alterations in metabolites production and therefore determining rearrangements in host metabolism. Currently, no simultaneous exploration and comparison of all three Trp related metabolic routes has been performed in the context of MS patients before and after Ocrelizumab (OCR) treatment.MethodsBy performing mass spectrometry on plasma samples collected from healthy controls and MS patients before and six months after OCR treatment we provided a comparative investigation of Trp metabolomics profile.Results and discussionOur data points out to concurrent alterations of Trp-related pathways among both OCR treated and untreated MS patients. Furthermore, MS treated patients presented a pattern resembling health state for various metabolites across the pathways. The results reported in our research may contribute to unveiling new perspectives and understanding regarding MS pathogenetic mechanisms.
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spelling doaj-art-f2ccc7865dc74f56a1cdc9ce171d0bcf2025-08-20T03:09:57ZengFrontiers Media S.A.Frontiers in Immunology1664-32242025-06-011610.3389/fimmu.2025.16036631603663Tryptophan pathway profiling in multiple sclerosis patients treated with ocrelizumabCarolina Ricci0Matteo Pivetta1Eleonora Martinis2Viviana Valeri3Carolina Colliva4Nicola Giacchè5Simone Lorenzut6Daniela Cargnelutti7Barbara Frossi8Silvia Tonon9Department of Medicine, University of Udine, Udine, ItalyDepartment of Medicine, University of Udine, Udine, ItalyDepartment of Medicine, University of Udine, Udine, ItalyDepartment of Medicine, University of Udine, Udine, ItalyTes Pharma S.r.l., Corciano, ItalyTes Pharma S.r.l., Corciano, ItalyNeurology Unit, “Head, Neck and Neurosciences” Department, University Hospital of Udine, Udine, ItalyNeurology Unit, “Head, Neck and Neurosciences” Department, University Hospital of Udine, Udine, ItalyDepartment of Medicine, University of Udine, Udine, ItalyDepartment of Medicine, University of Udine, Udine, ItalyIntroductionL-Tryptophan (Trp) metabolism is impaired across various chronic inflammatory pathologies, including Multiple Sclerosis (MS). Trp processing relies on three metabolic routes, namely Kynurenine, Serotonin and Indole pathways. The host microbiota significantly impacts Trp metabolism, primarily by being responsible for Indole metabolites production and secondarily by shaping both Kynurenine and Serotonin pathways. Pathological conditions and pharmaceutical treatments can elicit changes in microbial populations, leading to alterations in metabolites production and therefore determining rearrangements in host metabolism. Currently, no simultaneous exploration and comparison of all three Trp related metabolic routes has been performed in the context of MS patients before and after Ocrelizumab (OCR) treatment.MethodsBy performing mass spectrometry on plasma samples collected from healthy controls and MS patients before and six months after OCR treatment we provided a comparative investigation of Trp metabolomics profile.Results and discussionOur data points out to concurrent alterations of Trp-related pathways among both OCR treated and untreated MS patients. Furthermore, MS treated patients presented a pattern resembling health state for various metabolites across the pathways. The results reported in our research may contribute to unveiling new perspectives and understanding regarding MS pathogenetic mechanisms.https://www.frontiersin.org/articles/10.3389/fimmu.2025.1603663/fullmultiple sclerosisocrelizumabtryptophankynurenineserotoninindole
spellingShingle Carolina Ricci
Matteo Pivetta
Eleonora Martinis
Viviana Valeri
Carolina Colliva
Nicola Giacchè
Simone Lorenzut
Daniela Cargnelutti
Barbara Frossi
Silvia Tonon
Tryptophan pathway profiling in multiple sclerosis patients treated with ocrelizumab
Frontiers in Immunology
multiple sclerosis
ocrelizumab
tryptophan
kynurenine
serotonin
indole
title Tryptophan pathway profiling in multiple sclerosis patients treated with ocrelizumab
title_full Tryptophan pathway profiling in multiple sclerosis patients treated with ocrelizumab
title_fullStr Tryptophan pathway profiling in multiple sclerosis patients treated with ocrelizumab
title_full_unstemmed Tryptophan pathway profiling in multiple sclerosis patients treated with ocrelizumab
title_short Tryptophan pathway profiling in multiple sclerosis patients treated with ocrelizumab
title_sort tryptophan pathway profiling in multiple sclerosis patients treated with ocrelizumab
topic multiple sclerosis
ocrelizumab
tryptophan
kynurenine
serotonin
indole
url https://www.frontiersin.org/articles/10.3389/fimmu.2025.1603663/full
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