Faecal metaproteomics analysis reveals a high cardiovascular risk profile across healthy individuals and heart failure patients

The gut microbiota is a crucial link between diet and cardiovascular disease (CVD). Using fecal metaproteomics, a method that concurrently captures human gut and microbiome proteins, we determined the crosstalk between gut microbiome, diet, gut health, and CVD. Traditional CVD risk factors (age, BMI...

Full description

Saved in:
Bibliographic Details
Main Authors: Chaoran Yang, Leticia Camargo Tavares, Han-Chung Lee, Joel R. Steele, Rosilene V. Ribeiro, Anna L. Beale, Stephanie Yiallourou, Melinda J. Carrington, David M. Kaye, Geoffrey A. Head, Ralf B. Schittenhelm, Francine Z. Marques
Format: Article
Language:English
Published: Taylor & Francis Group 2025-12-01
Series:Gut Microbes
Subjects:
Online Access:https://www.tandfonline.com/doi/10.1080/19490976.2024.2441356
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1850244268256395264
author Chaoran Yang
Leticia Camargo Tavares
Han-Chung Lee
Joel R. Steele
Rosilene V. Ribeiro
Anna L. Beale
Stephanie Yiallourou
Melinda J. Carrington
David M. Kaye
Geoffrey A. Head
Ralf B. Schittenhelm
Francine Z. Marques
author_facet Chaoran Yang
Leticia Camargo Tavares
Han-Chung Lee
Joel R. Steele
Rosilene V. Ribeiro
Anna L. Beale
Stephanie Yiallourou
Melinda J. Carrington
David M. Kaye
Geoffrey A. Head
Ralf B. Schittenhelm
Francine Z. Marques
author_sort Chaoran Yang
collection DOAJ
description The gut microbiota is a crucial link between diet and cardiovascular disease (CVD). Using fecal metaproteomics, a method that concurrently captures human gut and microbiome proteins, we determined the crosstalk between gut microbiome, diet, gut health, and CVD. Traditional CVD risk factors (age, BMI, sex, blood pressure) explained < 10% of the proteome variance. However, unsupervised human protein-based clustering analysis revealed two distinct CVD risk clusters (low-risk and high-risk) with different blood pressure (by 9 mmHg) and sex-dependent dietary potassium and fiber intake. In the human proteome, the low-risk group had lower angiotensin-converting enzymes, inflammatory proteins associated with neutrophil extracellular trap formation and auto-immune diseases. In the microbial proteome, the low-risk group had higher expression of phosphate acetyltransferase that produces SCFAs, particularly in fiber-fermenting bacteria. This model identified severity across phenotypes in heart failure patients and long-term risk of cardiovascular events in a large population-based cohort. These findings underscore multifactorial gut-to-host mechanisms that may underlie risk factors for CVD.
format Article
id doaj-art-f2bfc3c6fc0342cbaa23bcaf14e97809
institution OA Journals
issn 1949-0976
1949-0984
language English
publishDate 2025-12-01
publisher Taylor & Francis Group
record_format Article
series Gut Microbes
spelling doaj-art-f2bfc3c6fc0342cbaa23bcaf14e978092025-08-20T01:59:47ZengTaylor & Francis GroupGut Microbes1949-09761949-09842025-12-0117110.1080/19490976.2024.2441356Faecal metaproteomics analysis reveals a high cardiovascular risk profile across healthy individuals and heart failure patientsChaoran Yang0Leticia Camargo Tavares1Han-Chung Lee2Joel R. Steele3Rosilene V. Ribeiro4Anna L. Beale5Stephanie Yiallourou6Melinda J. Carrington7David M. Kaye8Geoffrey A. Head9Ralf B. Schittenhelm10Francine Z. Marques11Hypertension Research Laboratory, School of Biological Sciences, Faculty of Science, Monash, Clayton, AustraliaHypertension Research Laboratory, School of Biological Sciences, Faculty of Science, Monash, Clayton, AustraliaMonash Proteomics &amp; Metabolomics Platform, Monash Biomedicine Discovery Institute &amp; Department of Biochemistry and Molecular Biology, Monash University, Melbourne, AustraliaMonash Proteomics &amp; Metabolomics Platform, Monash Biomedicine Discovery Institute &amp; Department of Biochemistry and Molecular Biology, Monash University, Melbourne, AustraliaCharles Perkins Centre, University of Sydney, Sydney, AustraliaHeart Failure Research Laboratory, Baker Heart and Diabetes Institute, Melbourne, AustraliaPreclinical Disease and Prevention Unit, Baker Heart and Diabetes Institute, Melbourne, AustraliaPreclinical Disease and Prevention Unit, Baker Heart and Diabetes Institute, Melbourne, AustraliaHeart Failure Research Laboratory, Baker Heart and Diabetes Institute, Melbourne, AustraliaNeuropharmacology Laboratory, Baker Heart and Diabetes Institute, Melbourne, AustraliaMonash Proteomics &amp; Metabolomics Platform, Monash Biomedicine Discovery Institute &amp; Department of Biochemistry and Molecular Biology, Monash University, Melbourne, AustraliaHypertension Research Laboratory, School of Biological Sciences, Faculty of Science, Monash, Clayton, AustraliaThe gut microbiota is a crucial link between diet and cardiovascular disease (CVD). Using fecal metaproteomics, a method that concurrently captures human gut and microbiome proteins, we determined the crosstalk between gut microbiome, diet, gut health, and CVD. Traditional CVD risk factors (age, BMI, sex, blood pressure) explained < 10% of the proteome variance. However, unsupervised human protein-based clustering analysis revealed two distinct CVD risk clusters (low-risk and high-risk) with different blood pressure (by 9 mmHg) and sex-dependent dietary potassium and fiber intake. In the human proteome, the low-risk group had lower angiotensin-converting enzymes, inflammatory proteins associated with neutrophil extracellular trap formation and auto-immune diseases. In the microbial proteome, the low-risk group had higher expression of phosphate acetyltransferase that produces SCFAs, particularly in fiber-fermenting bacteria. This model identified severity across phenotypes in heart failure patients and long-term risk of cardiovascular events in a large population-based cohort. These findings underscore multifactorial gut-to-host mechanisms that may underlie risk factors for CVD.https://www.tandfonline.com/doi/10.1080/19490976.2024.2441356Metaproteomedisease riskshort-chain fatty acidsmachine learning
spellingShingle Chaoran Yang
Leticia Camargo Tavares
Han-Chung Lee
Joel R. Steele
Rosilene V. Ribeiro
Anna L. Beale
Stephanie Yiallourou
Melinda J. Carrington
David M. Kaye
Geoffrey A. Head
Ralf B. Schittenhelm
Francine Z. Marques
Faecal metaproteomics analysis reveals a high cardiovascular risk profile across healthy individuals and heart failure patients
Gut Microbes
Metaproteome
disease risk
short-chain fatty acids
machine learning
title Faecal metaproteomics analysis reveals a high cardiovascular risk profile across healthy individuals and heart failure patients
title_full Faecal metaproteomics analysis reveals a high cardiovascular risk profile across healthy individuals and heart failure patients
title_fullStr Faecal metaproteomics analysis reveals a high cardiovascular risk profile across healthy individuals and heart failure patients
title_full_unstemmed Faecal metaproteomics analysis reveals a high cardiovascular risk profile across healthy individuals and heart failure patients
title_short Faecal metaproteomics analysis reveals a high cardiovascular risk profile across healthy individuals and heart failure patients
title_sort faecal metaproteomics analysis reveals a high cardiovascular risk profile across healthy individuals and heart failure patients
topic Metaproteome
disease risk
short-chain fatty acids
machine learning
url https://www.tandfonline.com/doi/10.1080/19490976.2024.2441356
work_keys_str_mv AT chaoranyang faecalmetaproteomicsanalysisrevealsahighcardiovascularriskprofileacrosshealthyindividualsandheartfailurepatients
AT leticiacamargotavares faecalmetaproteomicsanalysisrevealsahighcardiovascularriskprofileacrosshealthyindividualsandheartfailurepatients
AT hanchunglee faecalmetaproteomicsanalysisrevealsahighcardiovascularriskprofileacrosshealthyindividualsandheartfailurepatients
AT joelrsteele faecalmetaproteomicsanalysisrevealsahighcardiovascularriskprofileacrosshealthyindividualsandheartfailurepatients
AT rosilenevribeiro faecalmetaproteomicsanalysisrevealsahighcardiovascularriskprofileacrosshealthyindividualsandheartfailurepatients
AT annalbeale faecalmetaproteomicsanalysisrevealsahighcardiovascularriskprofileacrosshealthyindividualsandheartfailurepatients
AT stephanieyiallourou faecalmetaproteomicsanalysisrevealsahighcardiovascularriskprofileacrosshealthyindividualsandheartfailurepatients
AT melindajcarrington faecalmetaproteomicsanalysisrevealsahighcardiovascularriskprofileacrosshealthyindividualsandheartfailurepatients
AT davidmkaye faecalmetaproteomicsanalysisrevealsahighcardiovascularriskprofileacrosshealthyindividualsandheartfailurepatients
AT geoffreyahead faecalmetaproteomicsanalysisrevealsahighcardiovascularriskprofileacrosshealthyindividualsandheartfailurepatients
AT ralfbschittenhelm faecalmetaproteomicsanalysisrevealsahighcardiovascularriskprofileacrosshealthyindividualsandheartfailurepatients
AT francinezmarques faecalmetaproteomicsanalysisrevealsahighcardiovascularriskprofileacrosshealthyindividualsandheartfailurepatients