Mechanism of Baclofen Inhibiting the Proliferation and Metastasis of GBM by Regulating YAP

This study explores the effect of baclofen on the malignant phenotype of glioblastoma (GBM) and the growth of xenograft tumors and investigates the related mechanisms, aiming to reveal the effect of baclofen on the occurrence and development of GBM. The development of new therapeutic drugs for GBM l...

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Main Authors: Lin Zhu, Juan Lu, Zhijun Bao, Shiwen Guo
Format: Article
Language:English
Published: Wiley 2021-01-01
Series:Journal of Chemistry
Online Access:http://dx.doi.org/10.1155/2021/2753571
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author Lin Zhu
Juan Lu
Zhijun Bao
Shiwen Guo
author_facet Lin Zhu
Juan Lu
Zhijun Bao
Shiwen Guo
author_sort Lin Zhu
collection DOAJ
description This study explores the effect of baclofen on the malignant phenotype of glioblastoma (GBM) and the growth of xenograft tumors and investigates the related mechanisms, aiming to reveal the effect of baclofen on the occurrence and development of GBM. The development of new therapeutic drugs for GBM lays a theoretical and experimental foundation. Research results show that baclofen could inhibit GBM cell proliferation and migration and promote GBM cell apoptosis; baclofen dose- and time-dependently could induce GBM cell YAP phosphorylation. YAP participated in the effect of baclofen on GBM cell proliferation and migration inhibition. Baclofen induced YAP phosphorylation in GBM cells through the GABABR2-Gs-Lats1/2 signaling pathway. Baclofen could inhibit the expression of survivin and Bcl2. Baclofen inhibits subcutaneous tumors by inducing YAP phosphorylation in vivo.
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institution DOAJ
issn 2090-9071
language English
publishDate 2021-01-01
publisher Wiley
record_format Article
series Journal of Chemistry
spelling doaj-art-f09d0ca3e6d649fcbb36dc94d986d8952025-08-20T03:21:26ZengWileyJournal of Chemistry2090-90712021-01-01202110.1155/2021/2753571Mechanism of Baclofen Inhibiting the Proliferation and Metastasis of GBM by Regulating YAPLin Zhu0Juan Lu1Zhijun Bao2Shiwen Guo3Department of NeurosurgerySchool of Foreign StudiesDepartment of NeurosurgeryDepartment of NeurosurgeryThis study explores the effect of baclofen on the malignant phenotype of glioblastoma (GBM) and the growth of xenograft tumors and investigates the related mechanisms, aiming to reveal the effect of baclofen on the occurrence and development of GBM. The development of new therapeutic drugs for GBM lays a theoretical and experimental foundation. Research results show that baclofen could inhibit GBM cell proliferation and migration and promote GBM cell apoptosis; baclofen dose- and time-dependently could induce GBM cell YAP phosphorylation. YAP participated in the effect of baclofen on GBM cell proliferation and migration inhibition. Baclofen induced YAP phosphorylation in GBM cells through the GABABR2-Gs-Lats1/2 signaling pathway. Baclofen could inhibit the expression of survivin and Bcl2. Baclofen inhibits subcutaneous tumors by inducing YAP phosphorylation in vivo.http://dx.doi.org/10.1155/2021/2753571
spellingShingle Lin Zhu
Juan Lu
Zhijun Bao
Shiwen Guo
Mechanism of Baclofen Inhibiting the Proliferation and Metastasis of GBM by Regulating YAP
Journal of Chemistry
title Mechanism of Baclofen Inhibiting the Proliferation and Metastasis of GBM by Regulating YAP
title_full Mechanism of Baclofen Inhibiting the Proliferation and Metastasis of GBM by Regulating YAP
title_fullStr Mechanism of Baclofen Inhibiting the Proliferation and Metastasis of GBM by Regulating YAP
title_full_unstemmed Mechanism of Baclofen Inhibiting the Proliferation and Metastasis of GBM by Regulating YAP
title_short Mechanism of Baclofen Inhibiting the Proliferation and Metastasis of GBM by Regulating YAP
title_sort mechanism of baclofen inhibiting the proliferation and metastasis of gbm by regulating yap
url http://dx.doi.org/10.1155/2021/2753571
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AT juanlu mechanismofbaclofeninhibitingtheproliferationandmetastasisofgbmbyregulatingyap
AT zhijunbao mechanismofbaclofeninhibitingtheproliferationandmetastasisofgbmbyregulatingyap
AT shiwenguo mechanismofbaclofeninhibitingtheproliferationandmetastasisofgbmbyregulatingyap