The unique role of anosognosia in the clinical progression of Alzheimer’s disease: a disorder-network perspective

Abstract Alzheimer’s disease (AD) encompasses a long continuum from a preclinical phase, characterized by neuropathological alterations albeit normal cognition, to a symptomatic phase, marked by its clinical manifestations. Yet, the neural mechanisms responsible for cognitive decline in AD patients...

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Main Authors: Katia Andrade, Valentina Pacella
Format: Article
Language:English
Published: Nature Portfolio 2024-10-01
Series:Communications Biology
Online Access:https://doi.org/10.1038/s42003-024-07076-7
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author Katia Andrade
Valentina Pacella
author_facet Katia Andrade
Valentina Pacella
author_sort Katia Andrade
collection DOAJ
description Abstract Alzheimer’s disease (AD) encompasses a long continuum from a preclinical phase, characterized by neuropathological alterations albeit normal cognition, to a symptomatic phase, marked by its clinical manifestations. Yet, the neural mechanisms responsible for cognitive decline in AD patients remain poorly understood. Here, we posit that anosognosia, emerging from an error-monitoring failure due to early amyloid-β deposits in the posterior cingulate cortex, plays a causal role in the clinical progression of AD by preventing patients from being aware of their deficits and implementing strategies to cope with their difficulties, thus fostering a vicious circle of cognitive decline.
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spelling doaj-art-efb7b56923c4412582ceb0dfd22981372025-08-20T02:11:54ZengNature PortfolioCommunications Biology2399-36422024-10-017111010.1038/s42003-024-07076-7The unique role of anosognosia in the clinical progression of Alzheimer’s disease: a disorder-network perspectiveKatia Andrade0Valentina Pacella1Institute of Memory and Alzheimer’s Disease (IM2A), Department of Neurology, Assistance Publique-Hôpitaux de Paris (AP-HP), Sorbonne University, Pitié-Salpêtrière HospitalIUSS Cognitive Neuroscience (ICON) Center, Scuola Universitaria Superiore IUSSAbstract Alzheimer’s disease (AD) encompasses a long continuum from a preclinical phase, characterized by neuropathological alterations albeit normal cognition, to a symptomatic phase, marked by its clinical manifestations. Yet, the neural mechanisms responsible for cognitive decline in AD patients remain poorly understood. Here, we posit that anosognosia, emerging from an error-monitoring failure due to early amyloid-β deposits in the posterior cingulate cortex, plays a causal role in the clinical progression of AD by preventing patients from being aware of their deficits and implementing strategies to cope with their difficulties, thus fostering a vicious circle of cognitive decline.https://doi.org/10.1038/s42003-024-07076-7
spellingShingle Katia Andrade
Valentina Pacella
The unique role of anosognosia in the clinical progression of Alzheimer’s disease: a disorder-network perspective
Communications Biology
title The unique role of anosognosia in the clinical progression of Alzheimer’s disease: a disorder-network perspective
title_full The unique role of anosognosia in the clinical progression of Alzheimer’s disease: a disorder-network perspective
title_fullStr The unique role of anosognosia in the clinical progression of Alzheimer’s disease: a disorder-network perspective
title_full_unstemmed The unique role of anosognosia in the clinical progression of Alzheimer’s disease: a disorder-network perspective
title_short The unique role of anosognosia in the clinical progression of Alzheimer’s disease: a disorder-network perspective
title_sort unique role of anosognosia in the clinical progression of alzheimer s disease a disorder network perspective
url https://doi.org/10.1038/s42003-024-07076-7
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